Jun B Proto-Oncogene (JUNB) ELISA Kits

Transcription factor involved in regulating gene activity following the primary growth factor response. Zusätzlich bieten wir Ihnen JUNB Antikörper (296) und JUNB Proteine (9) und viele weitere Produktgruppen zu diesem Protein an.

list all ELISA KIts Gen GeneID UniProt
JUNB 3726 P17275
Anti-Ratte JUNB JUNB 24517 P24898
JUNB 16477 P09450
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Top JUNB ELISA Kits auf antikoerper-online.de

Showing 4 out of 12 products:

Katalog Nr. Reaktivität Sensitivität Bereich Bilder Menge Lieferzeit Preis Details
Maus 0.075 ng/mL 0.15 ng/mL - 10 ng/mL 96 Tests 13 bis 16 Tage
$757.89
Details
Human
  96 Tests 26 bis 36 Tage
$899.80
Details
Human
  96 Tests 31 bis 41 Tage
$728.20
Details
Human
  96 Tests 2 bis 3 Tage
$525.00
Details

Weitere ELISA Kits für JUNB Interaktionspartner

Human Jun B Proto-Oncogene (JUNB) Interaktionspartner

  1. JUNB governs a feed-forward network of TGFB signaling that aggravates breast cancer invasion.

  2. MVIH and Jun-B have a synergistic effect on the regulation of angiogenesis and cell proliferation.

  3. IL-6-induced JunB expression contributes to epithelial-mesenchymal transition changes in uveal melanoma cells.

  4. LINC00657 might be involved in regulating esophageal squamous cell carcinoma (ESCC's) response to radiation; and it functioned as an oncogene in ESCC by targeting miR-615-3p and JunB.

  5. It has been shown that the AP-1 family member JunB and retinoic acid receptor alpha (RARa) mediate catalase transcriptional activation and repression, respectively, by controlling chromatin remodeling through a histone deacetylases-dependent mechanism.

  6. JunB neddylation mediated by Itch promotes its ubiquitination-dependent degradation.

  7. a specific role for AP-1/JunB in multiple myeloma cell proliferation, survival and drug resistance.

  8. BATF/JUN-B and BATF/C-JUN complexes play important roles in OA cartilage destruction through regulating anabolic and catabolic gene expression in chondrocytes.

  9. VEGF-induced endothelial migration is mediated primarily by induction of JunB whereas the promotion of endothelial proliferation by VEGF is mediated by JunB-independent AP-1 family members.

  10. Results suggested that JunB could play an important role in promoting cell invasion, migration and distant metastasis in head and neck squamous cell carcinoma via pathways other than epithelial-to-mesenchymal transition.

  11. Highly recurrent mutation of JUNB is associated with nodular lymphocyte predominant Hodgkin lymphoma.

  12. ETS2, HNF4A and JUNB are synergistic master regulators of epithelial-to-mesenchymal transition in cancer.

  13. CARMA1- and MyD88-dependent activation of Jun/ATF-type AP-1 complexes is a hallmark of ABC diffuse large B-cell lymphomas.

  14. PDK1 functions as a tumor promoter in human gallbladder cancer by upregulating JunB, promoting epithelial mesenchymal transformation, and cell migration.

  15. Our findings demonstrate that miRNA-149* may serve as an oncogenic regulator in T-cell acute lymphoblastic leukemia by negatively regulating JunB

  16. The MAPK pathway plays a primary role in the control of JUNB gene expression.

  17. JunB is likely to be a key target of c-Abl in expression of p21 in Adriamycin-induced DDR.

  18. Caveolin 2 disengages repressed Egr-1 and JunB promoters from lamin A/C through disassembly of H3K9me3 in the inner nuclear membrane.

  19. JunB expression was significantly increased while cyclin-D1 expression was significantly down-regulated in pre-eclampsia relative to control placental mesenchymal stromal cells.

  20. findings uncover the oncogenic role of the JUNB/CD30 axis and its potential as therapeutic target in ALK+ ALCL.

Zebrafish Jun B Proto-Oncogene (JUNB) Interaktionspartner

  1. Data demonstrate for the first time an essential role of JunB-CBFbeta signaling for maintaining sarcomere architecture and function.

Xenopus laevis Jun B Proto-Oncogene (JUNB) Interaktionspartner

  1. JunB is a regulator of tail organization possibly through integration of several morphogen signaling pathways.

Cow (Bovine) Jun B Proto-Oncogene (JUNB) Interaktionspartner

  1. The present data indicate that bovine dialyzable leukocyte extract can block the AP-1 DNA-binding activity and expression of several transcriptions factors in breast cancer cells.

Mouse (Murine) Jun B Proto-Oncogene (JUNB) Interaktionspartner

  1. JunB is required for Th17 cell differentiation. CD4+ T cells deficient in the JunB-encoding gene Junb fail to differentiate into Th17 cells. JunB appears to activate Th17 signature genes by forming a heterodimer with BATF, another AP-1 factor essential for Th17 differentiation.

  2. JunB is a critical regulator of IRF4-dependent regulatory T cell effector programs, highlighting important functions for AP-1 in regulatory T cell-mediated immune homeostasis.

  3. JunB has an essential role in IL-23-dependent pathogenicity of Th17 cells

  4. Data show that JunB is a nonredundant regulator of transcriptional programs that support Th17 cell identity and restrain alternative Th1 and Treg cell fates.

  5. the present data demonstrates for the first time that JunB plays an important role in the formation of embryonic vascular networks.

  6. BATF/JUN-B and BATF/C-JUN complexes play important roles in OA cartilage destruction through regulating anabolic and catabolic gene expression in chondrocytes.

  7. JUNB is a significant modulator of both classical and alternative macrophage activation.

  8. Study shows that myeloid deletion of JUNB dampens immune polarization and reshapes disease outcomes during infection with both P. berghei and N. brasiliensis by limiting type 1 and type 2 responses, respectively. Thus, JUNB is an important regulator of myeloid responses to both type 1 and type 2 infections in vivo.

  9. Loss of JunB expression led to increased proliferation and decreased senescence, likely owing to decreased p16(Ink4a) and p21(CIP1) in epithelial cells.

  10. JunB and c-Jun expression in post-mitotic oligodendrocytes is mostly dispensable for the maintainance of white matter tracts throughout adult life, even under demyelinating conditions.

  11. JunB controls epidermal growth, barrier formation, and proinflammatory responses through direct and indirect mechanisms, pinpointing SQSTM1 as a key mediator of JunB suppression of NF-kappaB-dependent inflammation

  12. our results suggest a functional cooperation between NFAT1 and JunB in mediating IL-31 gene expression in CD4(+) T cells

  13. an important role of the A2B receptor-dependent upregulation of JunB in VEGF production and possibly other AP-1-regulated events.

  14. In experimental hepatitis, the absence of JUNB in immune cells decreased IFN-gamma expression and secretion from NK & NKT cells, leading to reduced STAT1 pathway activation.

  15. The results show that the junb gene is organized in a nuclear chromatin loop bringing into close spatial proximity the upstream promoter region and the downstream enhancer and that this configuration permits immediate RNA Pol II release on the junb body on binding of LPS-activated NF-kappaB to the enhancer.

  16. Data indicate that the C-terminal JunB caspase cleavage product functions as a potent inhibitor of AP-1-dependent transcription.

  17. Data suggest that JunB contributes to Id2 repression and provides important input in setting the transcriptional program of the EMT and profibrotic responses to TGF-beta.

  18. Wwp1 induces JunB ubiquitination and degradation.

  19. investigation of role of JunB: growth retardation of JunB-deficient mice correlates with strongly reduced fat mass (and is also associated with high lethality); in white adipose tissue, TNFalpha is up-regulated and perilipin is down-regulated

  20. T cell receptor signaling together with TGF-beta/IL-2 stimulation cooperatively enhance Foxp3 gene expression by maintaining accessible chromatin structure and by actively recruiting key transcription factors JunB and c-Rel.

JUNB Antigen-Profil

Beschreibung des Gens

Transcription factor involved in regulating gene activity following the primary growth factor response. Binds to the DNA sequence 5'-TGATCA-3'.

Genbezeichner und Symbole assoziert mit Jun B Proto-Oncogene (JUNB) ELISA Kits

  • JunB proto-oncogene, AP-1 transcription factor subunit (JUNB) Antikörper
  • JunB proto-oncogene, AP-1 transcription factor subunit (Junb) Antikörper
  • JunB proto-oncogene, AP-1 transcription factor subunit a (junba) Antikörper
  • jun B proto-oncogene L homeolog (junb.L) Antikörper
  • jun B proto-oncogene (junb) Antikörper
  • jun B proto-oncogene S homeolog (junb.S) Antikörper
  • jun B proto-oncogene (Junb) Antikörper
  • JunB proto-oncogene, AP-1 transcription factor subunit b (junbb) Antikörper
  • AP-1 Antikörper
  • JUNB Antikörper
  • junbl Antikörper
  • MGC81322 Antikörper
  • wu:cegs2778 Antikörper
  • wu:fa92h07 Antikörper
  • wu:fa95h09 Antikörper
  • wu:fj44e11 Antikörper
  • wu:fj68b07 Antikörper
  • wu:fq24h03 Antikörper
  • zgc:63933 Antikörper
  • zgc:64066 Antikörper

Bezeichner auf Proteinebene für Jun B Proto-Oncogene (JUNB) ELISA Kits

activator protein 1 , transcription factor jun-B , Jun-B oncogene , jun B proto-oncogene , jun-B proto-oncogene , myD21 , JunB proto-oncogene, AP-1 transcription factor subunit b , jun B proto-oncogene b , jun B proto-oncogene, like

GENE ID SPEZIES
3726 Homo sapiens
24517 Rattus norvegicus
407086 Danio rerio
432129 Xenopus laevis
443117 Ovis aries
468739 Pan troglodytes
514246 Bos taurus
733713 Xenopus (Silurana) tropicalis
779417 Xenopus laevis
16477 Mus musculus
100716068 Cavia porcellus
100146801 Equus caballus
100856456 Canis lupus familiaris
102181201 Capra hircus
336038 Danio rerio
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