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The protein encoded by FUT6 is a Golgi stack membrane protein that is involved in the creation of sialyl-Lewis X, an E-selectin ligand. Zusätzlich bieten wir Ihnen Fucosyltransferase 6 (Alpha (1,3) Fucosyltransferase) Kits (9) und Fucosyltransferase 6 (Alpha (1,3) Fucosyltransferase) Proteine (4) und viele weitere Produktgruppen zu diesem Protein an.
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characterisation of FUT4 and FUT6 alpha-(1 --> 2)-fucosyltransferases
FUT6 contributed to fucosylation of root arabinogalactan-proteins.
AtFUT4 and AtFUT6 are the first enzymes to be characterized for AGP glycosylation
meta-analysis identified new variants, rs3760775 (P = 1.2 x 10-23) and rs78060698 (P = 8.3 x 10-17) in FUT6 to be associated with circulating B12 (zeige NDUFB3 Antikörper) concentrations.
ur results strongly showed that the low expression of FUT6 regulated by miR (zeige MLXIP Antikörper)-106b contributed to cell migration, invasion, and proliferation in human breast cancer.
There were significant differences between the type 2 diabetes mellitus patients and controls in the risk allele distributions of rs3792267 (CAPN10 (zeige CAPN10 Antikörper)) (P = 0.002), rs1501299 (APM1 (zeige ZBTB7C Antikörper)) (P = 0.017), and rs3760776 (FUT6) (P = 0.031).
The T allele of rs3760776 might repress the transcription of the FUT6 gene.
In normal colon samples a significant relationship between sLe(x) expression and the ratio between FUT6/B4GALNT2 (zeige B4GALNT2 Antikörper) activities exists, demonstrating for the first time a role for B4GALNT2 (zeige B4GALNT2 Antikörper) in sLe(x) inhibition in vivo
Results suggest that FUT4 (zeige FUT4 Antikörper)-, FUT6- or FUT8 (zeige FUT8 Antikörper)-mediated MDR in human HCC (zeige FAM126A Antikörper) is associated with the activation of the PI3K (zeige PIK3CA Antikörper)/Akt (zeige AKT1 Antikörper) pathway and the expression of MRP1 (zeige MDM4 Antikörper).
these results suggest that FUT6 plays an important role in hepatocellular carcinoma growth by regulating the PI3K (zeige PIK3CA Antikörper)/Akt (zeige AKT1 Antikörper) signaling pathway.
The biosynthesis of the selectin-ligand sialyl Lewis x (zeige FUT4 Antikörper) in colorectal cancer tissues is regulated by fucosyltransferase VI and can be inhibited by an RNA interference-based approach.
GALNT14 (zeige GALNT14 Antikörper) and FUT3 (zeige FUT3 Antikörper)/6 H-scores were significantly higher in non-small cell lung cancer cell lines sensitive to dulanermin and drozitumab versus resistant cell lines
endo-beta-galactosidase (zeige GLB1 Antikörper)-sensitive and mAb FH6-reactive carbohydrate chains are generated under the control of expression levels of FUT6 and involved in the adhesion of colon carcinoma cells to liver sections.
The protein encoded by this gene is a Golgi stack membrane protein that is involved in the creation of sialyl-Lewis X, an E-selectin ligand. Mutations in this gene are a cause of fucosyltransferase-6 deficiency. Two transcript variants encoding the same protein have been found for this gene.
, fucosyltransferase 5
, fucosyltransferase 6 (alpha (1,3) fucosyltransferase)
, fucosyltransferase VI
, galactoside 3-L-fucosyltransferase