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Human PGF Protein expressed in Escherichia coli (E. coli) - ABIN413725
Ding, Huang, Song, Gao, Yuan, Wang, Cai, Fu, Luo: NFAT1 mediates placental growth factor-induced myelomonocytic cell recruitment via the induction of TNF-alpha. in Journal of immunology (Baltimore, Md. : 1950) 2010
Rat (Rattus) PGF Protein expressed in Escherichia coli (E. coli) - ABIN2126310
Zhang, Zhao, Yuan, Wu, Jiang, Luo, Zhao: Knockdown of placental growth factor (PLGF) mitigates hyperoxia-induced acute lung injury in neonatal rats: Suppressive effects on NFκB signaling pathway. in International immunopharmacology 2016
In urban Mozambican women with symptoms and/or signs suggestive of preeclampsia, low maternal plasma PlGF concentrations are associated with increased risks of adverse pregnancy outcomes, especially early delivery and stillbirth.
An sFlt-1:PlGF ratio above 655 is not predictive of impaired perinatal outcomes, and insufficiently reliable for predicting outcomes in cases with clinical signs of preeclampsia.
The maternal sFlt-1 to PlGF ratio in women with hypertensive disorders in pregnancy carries prognostic value for the development of preeclampsia.
Data suggest that PGF and PAPPA (zeige PAPPA Proteine) serve as serum biomarkers for early diagnosis of gestational hypertension. These studies were conducted at antenatal clinics in Ghana using blood from women between 8 and 13 weeks gestation. (PGF = placental growth factor; PAPPA (zeige PAPPA Proteine) = pregnancy-associated plasma protein-A (zeige PAPPA Proteine))
Lower umbilical cord PlGF levels are associated with lower birth weight, deviating fetal growth patterns, and a higher odds of fetal growth retardation.
Data suggest that circulating PGF levels fall by nearly one quarter during term labor (but not during elective caesarean section).
The cross-talk between tumor-associated macrophages and NSCLC cells via PLGF/Flt-1 (zeige FLT1 Proteine) and TGFbeta (zeige TGFB1 Proteine) receptor signaling may promote the growth and vascularization of NSCLC.
PlGF level showed an inversely proportional effect on the foetal weight.
these data suggest that PlGF may increase non-small cell lung cancer metastasis through SRp40 (zeige SRSF5 Proteine)-mediated mRNA splicing of VEGF (zeige VEGFA Proteine).
The present study investigated the interplay of VEGF (zeige VEGFA Proteine)-A165a isoform, the anti-angiogenic VEGF (zeige VEGFA Proteine)-A165b, placental growth factor (PIGF) and their receptors, VEGFR1 (zeige FLT1 Proteine) and VEGFR2 (zeige KDR Proteine), on junctional occupancy of VE-cadherin (zeige CDH5 Proteine) and macromolecular leakage in human endothelial monolayers and the perfused placental microvascular bed.
Data indicate that VEGF-B (zeige VEGFB Proteine) is a high-affinity VEGFR-1 (zeige FLT1 Proteine) ligand that, unlike PIGF (zeige PIGF Proteine), cannot efficiently induce signaling downstream of VEGFR-1 (zeige FLT1 Proteine).
the lack of PIGF (zeige PIGF Proteine) may have contributed to the delayed angiogenic response and the prothrombotic phenotype observed in the PIGF (zeige PIGF Proteine)(-/-)animals.
Overexpression of VEGF (zeige VEGFA Proteine) but not PIGF (zeige PIGF Proteine) exacerbated the lipopolysaccharide-mediated toxic effects, supporting a pathophysiological role for VEGF (zeige VEGFA Proteine) in mediating the sepsis phenotype.
Findings indicate that placental growth factor (PlGF) plays an important role in liver fibrosis-associated angiogenesis and that blockage of PlGF could be an effective strategy for chronic liver disease.
Data demonstrate that PlGF is expressed in vivo and that its expression is restricted to the areas of the tumors where the highest levels of hypoxia occur. Also, hypoxia activates PlGF expression in cultured lymphatic endothelial cells.
Study provides mechanistic and clinical evidence that decreased PLGF levels in the placenta after in utero alcohol exposure are associated to brain angiogenesis defects. Measurement of PLGF levels at birth in the placenta or the fetal blood may serve as a predictive marker for subsequent neurodevelopmental outcomes of exposed fetuses.
PGF (zeige PTGFR Proteine) deficiency is associated with impaired cerebral vascular development in mice.
PlGF inhibition attenuates PERK (zeige EIF2AK3 Proteine) activation, likely by tempering hypoxia in HCC (zeige FAM126A Proteine) via vessel normalisation. The UPR is able to regulate PlGF expression, suggesting the existence of a feedback mechanism for hypoxia-mediated UPR
Placenta growth factor augments airway hyperresponsiveness via leukotrienes and IL-13 (zeige IL13 Proteine).
Data indicate that placental growth factor (PlGF) is needed in the spleen to allow the activation of T cells and blood pressure raising.
Study reports polymorphisms in the bovine PGF gene significantly associated with the maternal effect on stillbirth and calving ease in animals under selection.
placenta growth factor expression is regulated by both VEGF (zeige VEGFA Proteine) and hyperglycaemia via VEGFR-2 (zeige KDR Proteine)
This gene encodes a growth factor found in placenta which is homologous to vascular endothelial growth factor. Alternatively spliced transcripts encoding different isoforms have been found for this gene.
placenta growth factor
, placental growth factor, vascular endothelial growth factor-related protein
, placenta growth factor-like