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Lmo4 participates in the regulation of lung epithelial cell proliferation but is not essential for tumor progression.
LMO4 appears to form a hub in protein-protein interaction networks, linking numerous pathways within cells.
Clim2 (zeige LDB1 Antikörper), in a complex with LMO4, supports mammary stem cells by directly targeting the Fgfr2 (zeige FGFR2 Antikörper) promoter in basal cells to increase its expression
Existence of a region in the lateral portion of the mammalian cochlea that is competent to make an organ of Corti is demonstrated in the absence of LMO4 function.
Low LMO4 mRNA levels were associated with response to erlotinib in non-small-cell lung cancer.
oncoprotein HBXIP (zeige HBXIP Antikörper) is able to activate the transcriptional coregulatory protein LMO4 through transcription factor Sp1 (zeige SP1 Antikörper) in promotion of proliferation of breast cancer cells.
Data suggest that overexpression of LMO4 may disrupt some of the normal tumour suppressor activities of CtIP (zeige RBBP8 Antikörper), thereby contributing to breast cancer progression.
the association of high levels of LMO4 with aggressive neuroblastomas is dependent on LMO4 regulation of cadherin expression and hence, tumor invasiveness
LMO4 is a direct target of p53 (zeige TP53 Antikörper) and inhibits p53 (zeige TP53 Antikörper)-mediated proliferative inhibition of breast cancer cells through interacting p53 (zeige TP53 Antikörper)
LMO4 is over-expressed in highly invasive rhabdomyosarcoma, alveolar cells
Multiple roles of Lmo4 during retinal development.
These results reveal a novel, LMO4-dependent transcriptional program within the basolateral amygdala that is essential to cue-reward learning.
activation of SLK (zeige SLK Antikörper) by haptotactic signals requires its recruitment to the leading edge by LMO4 in a Src (zeige SRC Antikörper)-dependent manner.
Thus, our study reveals an essential requirement for LMO4 to modulate central insulin (zeige INS Antikörper) signaling.
A novel sensory competent region in the lateral cochlear duct is regulated by LMO4.
By increasing neuronal activity in the paraventricular hypothalamus, food intake was suppressed in LMO4-deficient mice.
LMO4 is a novel modulator of adipogenesis.
The results of this study found that LIM domain only 4 (LMO4) inhibits PTP1B (zeige PTPN1 Antikörper) activity by increasing the oxidized inactive form of PTP1B (zeige PTPN1 Antikörper) in hypothalamus of mice.
results identify Lmo4 as a central developmental control over the diversity of motor cortex projection neuron subpopulations, establishing their area-specific identity and specialized connectivity.
LMO4 modulates the activity of the DEAF NES (zeige NES Antikörper), causing nuclear accumulation of a construct containing the LMO4-interaction region of DEAF1 (zeige DEAF1 Antikörper).
This gene encodes a cysteine-rich protein that contains two LIM domains but lacks a DNA-binding homeodomain. The encoded protein may play a role as a transcriptional regulator or as an oncogene.
LIM domain only 4
, LIM domain only protein 4
, LIM domain transcription factor LMO4
, breast tumor autoantigen
, ethanol induced 4