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Human Polyclonal SLC1A3 Primary Antibody für ICC, FACS - ABIN151381
Kobayashi, Millhorn: Hypoxia regulates glutamate metabolism and membrane transport in rat PC12 cells. in Journal of neurochemistry 2001
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Human Monoclonal SLC1A3 Primary Antibody für ELISA, WB - ABIN520226
Yamada, Urano-Tashiro, Tanaka, Akiyama, Tashiro: Involvement of crosstalk between Oct4 and Meis1a in neural cell fate decision. in PLoS ONE 2013
Human Polyclonal SLC1A3 Primary Antibody für WB - ABIN520225
Xiao, Li, Jones-Brando, Yolken: Abnormalities of neurotransmitter and neuropeptide systems in human neuroepithelioma cells infected by three Toxoplasma strains. in Journal of neural transmission (Vienna, Austria : 1996) 2013
Human Polyclonal SLC1A3 Primary Antibody für IF (p), IHC (p) - ABIN730783
Xiao, Xiao, Meng, Du, Hu, Gao, Yu, Wang, Ban: Effect of subarachnoid nerve block anesthesia on glutamate transporter GLAST and GLT-1 expressions in rabbits. in Asian Pacific journal of tropical medicine 2015
Rat (Rattus) Polyclonal SLC1A3 Primary Antibody für ELISA, ICC - ABIN346976
Rothstein, Dykes-Hoberg, Pardo, Bristol, Jin, Kuncl, Kanai, Hediger, Wang, Schielke, Welty: Knockout of glutamate transporters reveals a major role for astroglial transport in excitotoxicity and clearance of glutamate. in Neuron 1996
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Eaat1 expression is promoted by Fringe-mediated neuron-glial communication during development and suggest that Eaat1 plays an essential role in regulating central nervous system neural circuits that control locomotion in Drosophila
dEAAT1 is present at the adult, but surprisingly not at embryonic and larval NMJ, suggesting a physiological maturation of the junction during metamorphosis
This is the first study to link SLC1A3 and EPHB2 (zeige EPHB2 Antikörper) to clinically relevant vertebral osteoporosis phenotypes.
crystal structures of a thermostabilized human SLC1 (zeige MCHR1 Antikörper) transporter, the excitatory amino acid transporter 1 (EAAT1), with and without allosteric and competitive inhibitors bound
This study demonstrated that the cytopathology and episodic paralysis in our Drosophila EA6 model stem from a gain-of-function chloride channelopathy of glial cells..
Starvation of Muller cells increased the glutamate (zeige GRIN1 Antikörper) uptake capacity as well as the expression of the most abundant glutamate transporter (zeige SLC1A1 Antikörper), EAAT1.
A heterozygous SLC1A3 c.1177G4A mutation has been detected in a patient with late-onset episodic ataxia. Same heterozygous mutation was identified in one clinically affected family member and two asymptomatic members.
that Abeta1-42 oligomers could cause disturbances in insulin (zeige INS Antikörper)/Akt (zeige AKT1 Antikörper)/EAAT (zeige SLC1A2 Antikörper) signaling in astrocytes
We consider an association between SLC1A3 and the behavioural problems which can also be considered a contributing factor to behavioural problems in larger duplications overlapping the 5p13 microduplication syndrome region.
In combination with other nearby residues, Arg-388 coordinates anion channel gating and forms part of the missing structural link between the anion conducting and substrate transport states in EAAT1.
SPAK (zeige STK39 Antikörper) and OSR1 (zeige OXSR1 Antikörper) are powerful negative regulators of the excitatory glutamate (zeige GRIN1 Antikörper) transporters EAAT1 and EAAT2 (zeige SLC1A2 Antikörper).
There was no association between pyramidal cell EAAT1 splice variant expression and schizophrenia.
Loss of EAAT4 (zeige SLC1A6 Antikörper) accounts for the initial hyper-excitability of Purkinje cells lacking b-III spectrin and that loss of GLAST appears to work synergistically to worsen motor deficits. When levels of both EAAT4 (zeige SLC1A6 Antikörper) and GLAST are compromised in b-III(-/-) mice, the proximal dendrites of Purkinje cells within the posterior cerebellum are the most vulnerable to degeneration.
The results of this study concluded that EAAT (zeige SLC1A2 Antikörper) anion channels play an important and unexpected role in adjusting glial intracellular anion concentration during maturation and in response to cerebellar activity.
SLC1A3 maintains a constant import of acidic amino acids independently of nutritional status in adipocytes.
glutamate/aspartate transporter (GLAST) was elevated in 8- to 10- and/or 20- to 22-month GHR (zeige GHR Antikörper)-KO mice when comparing genotypes
This study findings demonstrate that Ascl1 (zeige ASCL1 Antikörper)(CreERT2) and Glast(CreERT2) mouse lines enable simple and reliable labeling of adult-born GC lineages within restricted time windows.
arundic acid treatment prevented RGC death by upregulating GLAST in heterozygous (GLAST(+/-)) mice
Results show that expression of GLAST is decreased in cerebellar astrocytes in a mouse model of SCA1 (zeige ATXN1 Antikörper); decrease occurs in non-cell autonomous manner late in disease and correlates well with the loss of Purkinje neurons
we have demonstrated for the first time that DOR receptor activation induces astrocytic expression of EAAT1 and EAAT2 (zeige SLC1A2 Antikörper)
The result showed significant correlation between changes in GLUT 8 (zeige SLC2A8 Antikörper) and glucose levels with changes in StAR level in the testis and circulating testosterone level in the mice from birth to senescence
EAAT1 activity and sodium potassium-ATPase (zeige DNAH8 Antikörper) activity are co-regulated by a tightly coupled homeostatic relationship in astrocytes.
This gene encodes a member of a member of a high affinity glutamate transporter family. Mutations in this gene are associated with episodic ataxia, Type 6. Alternative splicing results in multiple transcript variants.
, drosophila glutamate transporter-1
, excitatory amino acid transporter 1
, glutamate transporter
, sodium-dependent excitatory amino acid transporter 1
, sodium-dependent glutamate transporter
, excitatory amino acid transporter SLC1A3a
, solute carrier family 1 (glial high affinity glutamate transporter), member 3
, Na+-dependent glutamate/aspartate transporter
, Sodium-dependent glutamate/aspartate transporter 1
, excitatory amino acid transporter 1-like
, glial glutamate transporter
, glutamate/aspartate transporter
, sodium-dependent glutamate/aspartate transporter 1
, solute carrier family 1, member 3
, solute carrier family 1 member 3
, glial high affinity glutamate transporter
, high-affinity neuronal glutamate transporter