Use your antibodies-online credentials, if available.
Keine Produkte auf Ihrer Vergleichsliste.
Ihr Warenkorb ist leer.
Alle Spezies anzeigen
Weitere Synonyme anzeigen
Wählen Sie die Spezies und Applikation aus
anti-Human CCL26 Antikörper:
anti-Mouse (Murine) CCL26 Antikörper:
anti-Rat (Rattus) CCL26 Antikörper:
Sie gelangen zu unserer vorgefilterten Suche.
Mouse (Murine) Polyclonal CCL26 Primary Antibody für IF (p), IHC (p) - ABIN1715139
Tukler Henriksson, Coursey, Corry, De Paiva, Pflugfelder: IL-13 Stimulates Proliferation and Expression of Mucin and Immunomodulatory Genes in Cultured Conjunctival Goblet Cells. in Investigative ophthalmology & visual science 2015
Based on the ADEPT study data set, we report that airway mucosal expression of CCL26 was a robust discriminator of type 2 inflammation from healthy nonatopic subjects. Furthermore, airway mucosal CCL26 expression was best identified by using a panel of clinical biomarkers, including Feno values, bEOS counts, and expression of 2 novel markers, sCCL17 and sCCL26
hypoxia-inducible factors activate the transcription of chemokine ligand 26 in cancer cells to recruit chemokine receptor 1-expressing myeloid-derived suppressor cells to the primary tumor
this study shows that expression of CCL26 is increased in eosinophilic myocarditis patients compared to chronic lymphocytic myocarditis patients
The reduction of circulating levels of MCP-4, eotaxin-3, and MIP-1beta could be one of the mechanisms by which bariatric surgery contributes to the reduction of cardiovascular risk in these patients.
Review: eotaxins (CCL11, CCL24, and CCL26) play key role(s) during symptomatic inflammatory responses raised in response to allergic crisis of allergic asthma and atopic dermatitis
Data show that the interleukins IL-4 and IL-13 are critical factors for the induction of eotaxin-3/CCL26 in the pancreas.
These results show a relation between CCL26 production by IL-13-stimulated bronchial epithelial cells , sputum eosinophil counts, and asthma severity. They also suggest a role for CCL26 in the sustained inflammation observed in patients with severe eosinophilic asthma and reveal CCL26 as a potential target for treating patients with eosinophilic asthma that are refractory
The most potent inhibition of RANTES, eotaxin and eotaxin3 as well as MMP9 activity.
These findings indicate that eotaxin-3 is expressed in Chronic subdural hematoma fluid
these data suggest that CCL26 and CCL24 are likely involved in the pathogenesis of chronic nasal hypereosinophilia, with a complex cooperation and different involvement of the various members of eotaxin family.
CCL11, CCL24, and CCL26 are increased in TB patients; hence, it seems that TB suppresses Th1 and the classic function of macrophages subsequently by inducing the chemokines' expression
these data identify a contributory role for DNA methylation in regulating eotaxin-3 production in human allergic inflammation.
CCL11, CCL24, and CCL26 has a role in the recruitment of extravillous trophoblast into decidual tissue and vessels.
CCL26 is a more effective chemoattractant than CCL11 or CCL24 for eosinophils of asthmatics. All eotaxins induced the eosinophil migration from 0 to 6 h, but CCL26 triggered a second phase of migration between 12 & 18 h.
CCL1, CCL26, and IgE may be associated with pruritus in cutaneous T-cell lymphoma.
elevated expression in the active lesions of ulcerative colitis and Crohn's disease
Oesophageal squamous cells from gastro-oesophageal reflux disease and Eosinophilic oesophagitis patients express similar levels of eotaxin-3 when stimulated by Th2 cytokines, and omeprazole blocks that eotaxin-3 expression.
PPIs, in concentrations achieved in blood with conventional dosing, significantly inhibit IL-4-stimulated eotaxin-3 expression in EoE esophageal cells and block STAT6 binding to the promoter.
Up-regulation of CCL2, CCL26, IL6 and LOXL2 genes in cancer cells are part of the common effects of cancer-associated fibroblasts on hepatocellular carcinoma cells
Phosphodiesterase 4 inhibitors, rolipram and RO-20-1724 have no effect on CCL26 expression in human primary bronchial epithelial cells.
Data indicate that puerarin attenuates airway inflammation via eotaxin-3.
Distinct acidic and basic residues within CCR3 determine both receptor expression and activation by the eotaxins.
This gene is one of two Cys-Cys (CC) cytokine genes clustered on the q arm of chromosome 7. Cytokines are a family of secreted proteins involved in immunoregulatory and inflammatory processes. The CC cytokines are proteins characterized by two adjacent cysteines. The cytokine encoded by this gene displays chemotactic activity for normal peripheral blood eosinophils and basophils. The product of this gene is one of three related chemokines that specifically activate chemokine receptor CCR3. This chemokine may contribute to the eosinophil accumulation in atopic diseases.
chemokine (C-C motif) ligand 26
, C-C motif chemokine 26
, CC chemokine IMAC
, chemokine N1
, macrophage inflammatory protein 4-alpha
, small inducible cytokine A26
, small inducible cytokine subfamily A (Cys-Cys), member 26
, small-inducible cytokine A26
, thymic stroma chemokine-1
, chemokine (C-C motif) ligand 26-like
, eotaxin 3
, small chemokine ligand 26