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Data indicate a functional circuit linking PTEN and LXRs, and highlights LXRs as metabolic gatekeepers that are able to constrain prostate cancer (PCa (zeige ENPP1 ELISA Kits)) progression.
PANDER strongly impacts hepatic lipid metabolism across metabolic states and may induce a selective hepatic insulin resistant phenotype via the LXR pathway.
Data identify LXR as an important factor in early-pregnancy lipogenesis that is also necessary to protect against abnormalities in fetoplacental lipid homeostasis.
Wild-type mice and transgenic mice deficient for both alpha and beta Liver X Receptor isoforms were fed a control or an oleate enriched diet; results suggests that dietary oleic acid reduces cholesterolemia while promoting LXR-dependent hepatic lipogenesis without detrimental effects to the liver.
Ativation of LXRbeta increases AQP2 (zeige AQP2 ELISA Kits) protein levels in the renal collecting ducts via a posttranscriptional mechanism top regulate water electrolyte balance.
EEPD1 is a novel LXR (zeige NR1H3 ELISA Kits)-regulated gene in macrophages and that it promotes cellular cholesterol efflux by controlling cellular levels and activity of ABCA1 (zeige ABCA1 ELISA Kits).
Saponin-enriched sea (zeige Slc25a1 ELISA Kits) cucumber extracts up-regulation hepatic LCR-beta signaling, preventing obesity in mice fed a high-fat diet.
This study showed that although a certain redundancy exists in the functions of liver X receptor alpha (zeige NR1H3 ELISA Kits) and beta, some physiological processes are more under the influence of only one of them.
these data identify a new mechanism of LXR (zeige NR1H3 ELISA Kits) regulation that involves TIPARP (zeige Tiparp ELISA Kits), ADP-ribosylation and MACROD1 (zeige MACROD1 ELISA Kits).
Intestinal activation of LXR (zeige NR1H3 ELISA Kits) reduces the production of chylomicrons by a mechanism dependent on the apical localization of SR-B1 (zeige SCARB1 ELISA Kits).
ULK1 (zeige ULK1 ELISA Kits) has a role in RPS6KB1 (zeige RPS6KB1 ELISA Kits)-NCOR1 (zeige NCOR1 ELISA Kits) repression of NR1H/LXR (zeige NR1H3 ELISA Kits)-mediated Scd1 (zeige SCD ELISA Kits) transcription and augments lipotoxicity in hepatic cells
These data suggest that high doses of insulin (zeige INS ELISA Kits) downregulate apoA-I (zeige APOA1 ELISA Kits) gene expression in HepG2 cells through redistribution of FOXO1 (zeige FOXO1 ELISA Kits)/LXRbeta complex, FOXA2 (zeige FOXA2 ELISA Kits), and LXRalpha (zeige NR1H3 ELISA Kits) on hepatic enhancer of apoA-I (zeige APOA1 ELISA Kits) gene.
Expression of some LXR-dependent genes of cholesterol trafficking is related to breast tumor characteristics, but not time to recurrence.
a positive association of placental PPARgamma (zeige PPARG ELISA Kits) mRNA levels and placental DHA levels with baby weight
GW3965 significantly increases the expression of liver X nuclear receptor beta (LXRbeta) mRNA, while the liver X nuclear receptor alpha (LXRalpha (zeige NR1H3 ELISA Kits)( mRNA expression did not change a lot, and sensitizes gefitinib by inhibiting NF-kappa B (NF-kappaB (zeige NFKB1 ELISA Kits)) activation.
The effects of LXR (zeige NR1H3 ELISA Kits) agonist on interleukin-8 (IL-8 (zeige IL8 ELISA Kits)) secretion and nuclear factor-kappa B (NF-kappaB (zeige NFKB1 ELISA Kits)) activation in human umbilical vein endothelial cells (HUVECs), is reported.
Interactions among SNPs in nucleotide excision repair (NER) genes.
we demonstrate that joint deletion of two short conserved motifs that bind UNR (zeige P2RY4 ELISA Kits) and DDX6 (zeige DDX6 ELISA Kits) relieves repression of 4E-T (zeige EIF4ENIF1 ELISA Kits)-bound mRNA, in part reliant on the 4E-T (zeige EIF4ENIF1 ELISA Kits)-DDX6 (zeige DDX6 ELISA Kits)-CNOT1 (zeige CNOT1 ELISA Kits) axis.
These results suggested that 25-HC promoted ADC cell migration and invasion in an LXR-dependent manner in the monoculture system but that in the coculture system, the 25-HC-induced IL-1beta secretion enhanced the effect of 25-HC in an LXR-independent manner.
Protein level of LXRbeta protein was markedly reduced in focal cortical dysplasia.
The liver X receptors, LXRA (NR1H3\; MIM 602423) and LXRB, form a subfamily of the nuclear receptor superfamily and are key regulators of macrophage function, controlling transcriptional programs involved in lipid homeostasis and inflammation. The inducible LXRA is highly expressed in liver, adrenal gland, intestine, adipose tissue, macrophages, lung, and kidney, whereas LXRB is ubiquitously expressed. Ligand-activated LXRs form obligate heterodimers with retinoid X receptors (RXRs\; see MIM 180245) and regulate expression of target genes containing LXR response elements (summary by Korf et al., 2009
nuclear receptor subfamily 1, group H, member 2
, LXR beta
, liver X receptor beta
, oxysterols receptor LXR-beta
, retinoid X receptor-interacting protein No.15
, ubiquitously-expressed nuclear receptor 2
, nuclear orphan receptor LXR-beta
, orphan nuclear receptor OR-1
, ubiquitous receptor
, ubiquitously-expressed nuclear receptor
, LX receptor beta
, liver X nuclear receptor beta
, nuclear receptor NER
, steroid hormone-nuclear receptor NER
, nuclear receptor subfamily 1 group H member 2