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Human Complement 4 ELISA Kit für Sandwich ELISA - ABIN2683588
Sahu, Lambris: Structure and biology of complement protein C3, a connecting link between innate and acquired immunity. in Immunological reviews 2001
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Human Complement 4 ELISA Kit für Competition ELISA - ABIN2683861
Yang, Chung, Zhou, Lhotta, Hebert, Birmingham, Rovin, Yu: The intricate role of complement component C4 in human systemic lupus erythematosus. in Current directions in autoimmunity 2004
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Human Complement 4 ELISA Kit für Sandwich ELISA - ABIN365889
Hou, Qi, Liao, Zhang, Fang, Zhou, Liu, Bai, Zhang, Kijlstra, Yang: Copy number variations of complement component C4 are associated with Behçet's disease but not with ankylosing spondylitis associated with acute anterior uveitis. in Arthritis and rheumatism 2013
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Human Complement 4 ELISA Kit für Sandwich ELISA - ABIN612679
Koehler, Swain, Sanderson, Krishnan, Watt, Charlton: Growth hormone, dehydroepiandrosterone and adiponectin levels in non-alcoholic steatohepatitis: an endocrine signature for advanced fibrosis in obese patients. in Liver international : official journal of the International Association for the Study of the Liver 2012
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Human Complement 4 ELISA Kit für Sandwich ELISA - ABIN414468
Ma, Cui, Liao, Zhao: Complement activation contributes to the injury and outcome of kidney in human anti-glomerular basement membrane disease. in Journal of clinical immunology 2013
Human Complement 4 ELISA Kit für Competition ELISA - ABIN612676
Roos, Mollenhauer, Démant, Rittner: A molecular basis for the two locus model of human complement component C4. in Nature 1982
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An elevated number of C4 genes was observed in Alzheimer's disease (AD) patients as compared with healthy controls. The presence of high C4A (zeige C4B ELISA Kits) and C4B (zeige C4B ELISA Kits) copy numbers in AD patients could explain the increased C4 protein expression observed in AD patients, thus highlighting a possible role for C4A (zeige C4B ELISA Kits) and C4B (zeige C4B ELISA Kits) copy number variations in the risk of developing AD.
This study shows that the C4c/C4 ratio seems to be a better diagnostic measure than total antigenic C4 alone. Our findings underline that screening with total antigenic C4 implies a risk of overlooking C1-INH-HAE patients.
In comparison with C4-intact patients, C4-deficient patients had a different clinical/serologic lupus-like phenotype and lacked the lupus interferon (zeige IFNA ELISA Kits) signature.
C4 copy number variations and deficiency of C4A (zeige C4B ELISA Kits) both play an important role in the risk and manifestations of systemic lupus erythematosus in East Asian and European populations
Complement C4A deficiency (gene copy number <=1) was identified as a risk factor in a case-control study of juvenile dermatomyositis, particularly when subjects concurrently carried the HLA-DRB1 (zeige HLA-DRB1 ELISA Kits)*0301 allele.
Solution Structures of Complement C2 and Its C4 Complexes Propose Pathway-specific Mechanisms for Control and Activation of the Complement Proconvertases.
Coronary atherosclerosis is distinguished by serum C4 complement up-regulation and ceruloplasmin (zeige CP ELISA Kits) down-regulation.
find strong statistical significance for association of increased copy number of C4A (zeige C4B ELISA Kits) (OR 0.81 (0.73; 0.89);P = 4.4 x 10(-5)), with the effect most pronounced in individuals over 78 years (OR 0.67 (0.55; 0.81)) and females
genetic polymorphism is associated with acute graft versus host disease in unrelated hematopoietic stem cell transplantation
C4A (zeige C4B ELISA Kits) and C4B (zeige C4B ELISA Kits) gene copy numbers are stronger risk factors for juvenile-onset than for adult-onset systemic lupus erythematosus.
The C4A (zeige C4B ELISA Kits)-complete transcript is the main transcript under normal physiological conditions, while C4A (zeige C4B ELISA Kits)-AS is augmented when mastitis develops.
This gene encodes the acidic form of complement factor 4, part of the classical activation pathway. The protein is expressed as a single chain precursor which is proteolytically cleaved into a trimer of alpha, beta, and gamma chains prior to secretion. The trimer provides a surface for interaction between the antigen-antibody complex and other complement components. The alpha chain may be cleaved to release C4 anaphylatoxin, a mediator of local inflammation. Deficiency of this protein is associated with systemic lupus erythematosus and type I diabetes mellitus. This gene localizes to the major histocompatibility complex (MHC) class III region on chromosome 6. Varying haplotypes of this gene cluster exist, such that individuals may have 1, 2, or 3 copies of this gene. Two transcript variants encoding different isoforms have been found for this gene.
C3 and PZP-like alpha-2-macroglobulin domain-containing protein 2
, C4A anaphylatoxin
, Rodgers form of C4
, acidic C4
, acidic complement C4
, complement C4-A
, complement C4
, complement component 4 (within H-2S)
, complement component 4, gene 1
, complement component 4B (Chido blood group)
, complement component 4B (Childo blood group)
, MHC-linked complement C4
, sex-limited protein
, complement component 4