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anti-Human SOX9 Antikörper:
anti-Mouse (Murine) SOX9 Antikörper:
anti-Rat (Rattus) SOX9 Antikörper:
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Human Polyclonal SOX9 Primary Antibody für ICC, IF - ABIN4355424
Vestentoft, Jelnes, Hopkinson, Vainer, Møllgård, Quistorff, Bisgaard: Three-dimensional reconstructions of intrahepatic bile duct tubulogenesis in human liver. in BMC developmental biology 2011
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Human Polyclonal SOX9 Primary Antibody für IF (p), IHC (p) - ABIN754963
Papaioannou, Mirzamohammadi, Lisse, Nishimori, Wein, Kobayashi: MicroRNA-140 provides robustness to the regulation of hypertrophic chondrocyte differentiation by the PTHrP-HDAC4 pathway. in Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research 2014
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Human Monoclonal SOX9 Primary Antibody für IF, IHC (p) - ABIN520382
Hodgin, Borczuk, Nasr, Markowitz, Nair, Martini, Eichinger, Vining, Berthier, Kretzler, DAgati: A molecular profile of focal segmental glomerulosclerosis from formalin-fixed, paraffin-embedded tissue. in The American journal of pathology 2010
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Human Monoclonal SOX9 Primary Antibody für ICC, ELISA - ABIN969568
Dupasquier, Abdel-Samad, Glazer, Bastide, Jay, Joubert, Cavaillès, Blache, Quittau-Prévostel: A new mechanism of SOX9 action to regulate PKCalpha expression in the intestine epithelium. in Journal of cell science 2009
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Cat (Feline) Polyclonal SOX9 Primary Antibody für IHC (p), SimWes - ABIN4355427
Antunes, Tsaryk, Gonçalves, Pereira, Landes, Brochhausen, Ghanaati, Barbosa, Kirkpatrick: Poly(γ-Glutamic Acid) as an Exogenous Promoter of Chondrogenic Differentiation of Human Mesenchymal Stem/Stromal Cells. in Tissue engineering. Part A 2015
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Zebrafish (Danio rerio) Polyclonal SOX9 Primary Antibody für ELISA - ABIN408740
Yan, Willoughby, Liu, Crump, Wilson, Miller, Singer, Kimmel, Westerfield, Postlethwait: A pair of Sox: distinct and overlapping functions of zebrafish sox9 co-orthologs in craniofacial and pectoral fin development. in Development (Cambridge, England) 2005
Human Monoclonal SOX9 Primary Antibody für ELISA, WB - ABIN562960
Wainwright, Svingen, Ng, Wicking, Koopman: Primary cilia function regulates the length of the embryonic trunk axis and urogenital field in mice. in Developmental biology 2014
Human Polyclonal SOX9 Primary Antibody für IF, IHC (p) - ABIN272082
Zhang, Ding, Nie, Li-Ling, Zhang, Zhang, Chen, Li, Ding: Association of hsa-miR‑145 overexpression in human testicular cells with male infertility. in Molecular medicine reports 2015
Cow (Bovine) Polyclonal SOX9 Primary Antibody für FACS, WB - ABIN2780338
Vidal, Ortonne, Schedl: SOX9 expression is a general marker of basal cell carcinoma and adnexal-related neoplasms. in Journal of cutaneous pathology 2008
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Study found a positive relationship between LINC00052 and miR (zeige MLXIP Antikörper)-101-3p, and a negative relationship between miR (zeige MLXIP Antikörper)-101-3p and SOX9 in hepatocellular carcinoma (HCC (zeige FAM126A Antikörper)) tissues. Besides, miR (zeige MLXIP Antikörper)-101-3p was involved in LINC00052 inhibiting HCC (zeige FAM126A Antikörper) cells proliferation and metastasis. At the molecular level, LINC00052 downgulated SOX9 to inhibit HCC (zeige FAM126A Antikörper) cells proliferation and metastasis by interacting with miR (zeige MLXIP Antikörper)-101-3p.
SOX9 expression is related to prognosis in patients with oesophageal squamous cell carcinoma, although it is not an independent prognostic factor.
the findings of this study establish the SOX9/CA9 (zeige CA9 Antikörper)-mediated oncogenic pathway in glioma, the inhibition of which enhances the sensitivity of glioma cells to Temozolomide (TMZ) treatment, and thus highlights the value of developing small molecules or antibodies against the SOX9/CA9 (zeige CA9 Antikörper) pathway, for combination therapy with TMZ, in the more efficient management of glioma
Heterogeneous Expression of Embryonal Development Master Regulator SOX9 in Patients with Pancreatic Cancer
Melatonin inhibits cancer stem cell by down-regulation of SOX9-mediated signaling pathway in osteosarcoma
Our results demonstrate that the linc-ROR-miRNA-SOX9 regulatory network may represent a novel therapeutic target for esophageal squamous cell carcinoma
These results indicate that THRAP3 (zeige THRAP3 Antikörper) negatively regulates SOX9 transcriptional activity as a cofactor of a SOX9 transcriptional complex during chondrogenesis.
MiR (zeige MLXIP Antikörper)-19a could promote cell viability and migration of chondrocyte via positively regulating SOX9 expression through NF-kappaB (zeige NFKB1 Antikörper) signaling pathway.
miR (zeige MLXIP Antikörper)-185 inversely regulates SOX9 expression in non-small cell lung cancer.
Data suggest that the SOX9 transcription factor (SOX9)-fibroblast growth factor receptor 2 (zeige FGFR2 Antikörper) (FGFR2b) feed-forward loop has a lineage dependency role in pancreatic ductal adenocarcinoma (PDAC).
ETV5 (zeige ETV5 Antikörper) regulates ductal morphogenesis with Sox9 and is critical for regeneration from pancreatitis
a novel regulatory relationship between the retinal pigmented epithelium (RPE (zeige RPE Antikörper)) transcription factors Pax6 (zeige PAX6 Antikörper) and Sox9 that controls the timing of RPE (zeige RPE Antikörper) differentiation and the adjacent choroid maturation, is reported.
enhancer 13, a 557-base pair element located 565 kilobases 5' from the transcriptional start site, is essential to initiate mouse testis development; its deletion results in XY females with Sox9 transcript levels equivalent to those in XX gonads.
Sox9 is identified as important factor of VSMC function by modulation the extracellular matrix composition and calcium deposition, which are important processes in plaque development.
these findings widen our current knowledge of SOX9 targets in early chondrogenesis and call for new studies to identify the pioneer and transactivating factors that act upstream of or along with SOX9 to prompt chromatin remodeling and specific gene activation at the onset of chondrogenesis and other processes.
The data support a model of SOX9-GLI (zeige GLI1 Antikörper)-FOXA phasic gene regulatory network in chondrocyte development. Together, SOX9-GLI (zeige GLI1 Antikörper) auto-regulate and cooperate to activate and repress genes in proliferating chondrocytes. Upon hypertrophy, FOXA competes with SOX9, and control toward terminal differentiation passes to FOXA, RUNX, AP1 (zeige JUN Antikörper) and MEF2 (zeige MEF2C Antikörper) factors.
Ctgf (zeige CTGF Antikörper) is the direct target gene of SOX9 in chondrocytes and nucleus pulposus cells.
MOB1 (zeige HOPX Antikörper)-dependent YAP1 (zeige YAP1 Antikörper)/TAZ (zeige TAZ Antikörper)-TEAD complex functions as a transcriptional repressor of SOX9 and thereby negatively regulates chondrogenesis.
Analysis of tissue maturation markers showed an increase in COLL II and a decrease in SOX9 expression in the swine tibial menisci with age.
The expression of the differentiation and stemness genes, FN1 (zeige FN1 Antikörper) and SOX9, is accompanied by cell proliferation.
data presented here demonstrate that the 18 bp indel in the porcine SOX9 5'-UTR (zeige UTS2R Antikörper) is of functional importance and may therefore indeed be a causative variation in SOX9 associated traits
HIF-1alpha (zeige HIF1A Antikörper) activates Sox9 expression and enhances Sox9-mediated transcriptional activity.
Sox9 gene transfer could significantly enhance the repair effect of bone marrow mesenchymal stem cells on the degenerated discs.
The patterns of SRY (zeige SRY Antikörper)/SOX9 expression associated with the process of gonadal morphogenesis in rabbit appear similar to those of other mammals, including humans.
The protein encoded by this gene recognizes the sequence CCTTGAG along with other members of the HMG-box class DNA-binding proteins. It acts during chondrocyte differentiation and, with steroidogenic factor 1, regulates transcription of the anti-Muellerian hormone (AMH) gene. Deficiencies lead to the skeletal malformation syndrome campomelic dysplasia, frequently with sex reversal.
SRY (sex-determining region Y)-box 9 protein
, SRY-related HMG-box, gene 9
, transcription factor SOX-9
, SRY (sex determining region Y)-box 9
, SRY (sex determining region Y)-box 9 (campomelic dysplasia, autosomal sex-reversal)
, SRY-box containing gene 9
, SRY-box containing protein 9
, transcription factor SOX9
, LOW QUALITY PROTEIN: transcription factor SOX-9
, sex determining region Y-box 9
, transcription factor Sox-9-B
, transcription factor Sox9b