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Mouse (Murine) Monoclonal IL17F Primary Antibody für ICS, Neut - ABIN2689743
Chang, Dong: IL-17F: regulation, signaling and function in inflammation. in Cytokine 2009
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Human Monoclonal IL17F Primary Antibody für CyTOF, FACS - ABIN4900181
Zrioual, Ecochard, Tournadre, Lenief, Cazalis, Miossec: Genome-wide comparison between IL-17A- and IL-17F-induced effects in human rheumatoid arthritis synoviocytes. in Journal of immunology (Baltimore, Md. : 1950) 2009
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Human Polyclonal IL17F Primary Antibody für IHC (p), WB - ABIN530198
Oda, Shimazu, Naoi, Morimoto, Shimomura, Shimoda, Kagara, Maruyama, Kim, Noguchi: Intratumoral regulatory T cells as an independent predictive factor for pathological complete response to neoadjuvant paclitaxel followed by 5-FU/epirubicin/cyclophosphamide in breast cancer patients. in Breast cancer research and treatment 2012
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Mouse (Murine) Monoclonal IL17F Primary Antibody für CyTOF, FACS - ABIN4900112
Yamaguchi, Fujio, Shoda, Okamoto, Tsuno, Takahashi, Yamamoto: IL-17B and IL-17C are associated with TNF-alpha production and contribute to the exacerbation of inflammatory arthritis. in Journal of immunology (Baltimore, Md. : 1950) 2007
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Human Polyclonal IL17F Primary Antibody für Func, ELISA - ABIN2474948
Starnes, Robertson, Sledge, Kelich, Nakshatri, Broxmeyer, Hromas: Cutting edge: IL-17F, a novel cytokine selectively expressed in activated T cells and monocytes, regulates angiogenesis and endothelial cell cytokine production. in Journal of immunology (Baltimore, Md. : 1950) 2001
Mouse (Murine) Polyclonal IL17F Primary Antibody für ELISA, WB - ABIN1043827
Gomez-Rodriguez, Sahu, Handon, Davidson, Anderson, Kirby, August, Schwartzberg: Differential expression of interleukin-17A and -17F is coupled to T cell receptor signaling via inducible T cell kinase. in Immunity 2009
Human Monoclonal IL17F Primary Antibody für IHC, WB - ABIN1043791
Conti, Shen, Nayyar, Stocum, Sun, Lindemann, Ho, Hai, Yu, Jung, Filler, Masso-Welch, Edgerton, Gaffen: Th17 cells and IL-17 receptor signaling are essential for mucosal host defense against oral candidiasis. in The Journal of experimental medicine 2009
expression of IL-17A, E, and F and their receptors in non-small-cell lung cancer; data suggest that IL-17A, E, F and their receptors IL-17RA, RB, RC may be involved in the pathogenesis of non-small cell lung cancer
CCL20 may play an important role in the pathogenesis of MS. Both allelic variation of (rs6749704) within CCL20 gene and (rs763780) within IL-17F gene can be considered risk factor for development of MS in Egyptian patients.
the results suggest that IL17F genetic polymorphism is not associated with the development of rheumatic heart disease in South Indian population
The expression of IL-6 gene and protein was significantly induced by IL-17F. IL-17F activated TAK1 and NF-kappaB in airway smooth muscle cells.
IL-17FA7488G polymorphism was not significantly associated with colorectal cancer risk
a dietary pattern reflecting inflammation was significantly associated with colorectal cancer risk and this association could be modified according to the IL-17F rs763780 genotype and anatomic site.
Malignant T cells activate endothelial cells via IL-17 F.
The AA genotype on 7489A/G single nucleotide polymorphism of IL-17F and the A allele might be associated with a lower risk of acute rejection with better graft survival in kidney transplant recipients.
Findings suggest that IL-17F rs1889570 gene polymorphisms are significantly associated with the susceptibility to severe EV71 infection in Chinese Han children.
P-TEFb is involved in IL-17F-induced IL-8 expression and that steroids diminish it via the inhibition of CDK9 phosphorylation.
The IL-17F (+7488A/G) genotype revealed a significantly increased risk of accelerated silicosis. The IL-17F (+7488 G) allele was associated with an increased risk of accelerated silicosis.
developed ultrasensitive methods for measuring IL-17A and IL-17F in human serum samples and found that serum from psoriasis patients had higher and a broader range of concentrations of both IL-17 proteins compared to healthy volunteers
SNPs of rs3819024 in IL-17A and rs763780 in IL-17F were weakly related to a prognosis of tuberculosis.
IL17F (rs2397084) and IL10 (rs1800871) genes are associated with functional dyspepsia.
a mutation in IL-17F is associated with susceptibility to recurrent aphthous stomatitis
the G allele at rs763780 (IL-17F) was significantly associated with Takayasu Arteritis in Asian Indian population
levels of mRNA IL-17F and IL17F might be useful parameters for the diagnosis of atopic asthma patient
IL-17F rs763780 polymorphisms may be associated with the development of primary immune thrombocytopenia in a Chinese Han population.
In this study, IL-17F was demonstrated to have functions comparable to IL-17A in human keratinocytes.
Serum IL-17F predicted increased knee bone marrow lesion scores in females only among patients with knee osteoarthritis.
Il17f-/- mice resisted chemically induced colitis, but Il17a-/- mice did not, and that Il17f-/- CD45RBhiCD4+ T cells induced milder colitis in lymphocyte-deficient Rag2-/- mice, accompanied by an increase in intestinal regulatory T cells
IL-17A and IL-17F exert distinct biological effects during pulmonary infection; the IL-17F/IL-17RC signaling axis has the potential to significantly worsen pathogen-associated inflammation of the lower respiratory tract.
our results indicate that Th17 cells contribute to the airway vascular remodeling in asthma by mediating EPC chemotaxis, as well as PMVEC tube formation, via IL-17A rather than IL-17F.
Data indicate that IL-17A repressed IL-17F secretion in vitro and in vivo.
The concentrations in plasma of IL-17A, IL-17F, and the IL-17AF heterodimer all were increased greatly in mice after endotoxemia.
IL-17A and IL-17F secreted by CD4+Th17 cells specific to lung self-antigens are critical mediators of autoimmunity leading to the pathogenesis of obliterative airway disease.
The Th17 subset of T-lymphocytes produces IL-17F to stimulate bone healing.
IL-17F is not required for development of T helper cell (Th)2-type allergic airway inflammation.
Together, our findings demonstrate a protective role for IL-17F in colon cancer development, possibly via inhibiting tumor angiogenesis
These results demonstrate that IL-17A/F plays a critical role in Legionella pneumophila pneumonia, probably through induction of proinflammatory cytokines and accumulation of neutrophils at the infection site.
CNS2 is sufficient and necessary for Il17 and optimal Il17f gene transcription in Th17 cells
IL-17F was produced in peritoneal macrophages after TLR4 activation, peaking after 12 h. This effect was completely dependent on the presence of MyD88. The copresence of the complement activation product C5a amplified IL-17F production.
This work demonstrates the important roles of both IL-17 signaling and neutrophils in clearing this pathogen and surviving pulmonary Bacillus anthracis infection.
these results suggest the proinflammatory and essential role of IL-17F to develop spontaneous intestinal tumorigenesis in Apc(Min/+) mice in the presence of IL-17A.
Data show that Mel-18 and Ezh2 positively regulate the expression of Il17a and Il17f.
Increased resistance to infection in a mouse model of tularemia was associated with a specific increase in IL-17A/F
IL-17A and IL-17F induce the production of chemokines MCP-1 and MIP-2 via MAPK pathways (p38 MAPK and ERK1/2), as well as mRNA transcription and protein translation and have synergistic effects with TNF-alpha and IL-1beta in cultured mesangial cells.
Intranasal administration of adenovirus expressing IL-17F results in bronchoalveolar lavage neutrophilia and inflammatory gene expression in the lung.
a role for IL-17F in the induction of neutrophilia in the lungs and in the exacerbation of Ag-induced pulmonary inflammation.
THi cell differentiation is associated with epigenetic changes in the IL-17-IL-17F locus, which suggests novel mechanisms in T cell functional regulation.
The protein encoded by this gene is a cytokine that shares sequence similarity with IL17. This cytokine is expressed by activated T cells, and has been shown to stimulate the production of several other cytokines, including IL6, IL8, and CSF2/GM_CSF. This cytokine is also found to inhibit the angiogenesis of endothelial cells and induce endothelial cells to produce IL2, TGFB1/TGFB, and monocyte chemoattractant protein-1.
, cytokine ML-1
, mutant IL-17F
, interleukin 17F