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anti-Human CXCL5 Antikörper:
anti-Rat (Rattus) CXCL5 Antikörper:
anti-Mouse (Murine) CXCL5 Antikörper:
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Mouse (Murine) Polyclonal CXCL5 Primary Antibody für IF (p), IHC (p) - ABIN741900
Heilmann, Schinke, Bindl, Wehner, Rapp, Haffner-Luntzer, Nemitz, Liedert, Amling, Ignatius: The Wnt serpentine receptor Frizzled-9 regulates new bone formation in fracture healing. in PLoS ONE 2014
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Human Monoclonal CXCL5 Primary Antibody für IHC (p), ELISA - ABIN562826
Zhou, Dai, Zhou, Wang, Yang, Wang, Huang, Fan, Zhou: Overexpression of CXCL5 mediates neutrophil infiltration and indicates poor prognosis for hepatocellular carcinoma. in Hepatology (Baltimore, Md.) 2012
Show all 3 Pubmed References
Human Polyclonal CXCL5 Primary Antibody für ELISA, WB - ABIN2473135
Strieter, Belperio, Phillips, Keane: CXC chemokines in angiogenesis of cancer. in Seminars in cancer biology 2004
Human Monoclonal CXCL5 Primary Antibody für CyTOF, ELISA (Capture) - ABIN4900299
Edwards, Johnson, Johnston: Combination therapy: Synergistic suppression of virus-induced chemokines in airway epithelial cells. in American journal of respiratory cell and molecular biology 2006
Two haplotype blocks, one upstream to the coding region of UGT2A1 (rs146712414, P = 9.1 x 10(-5); odds ratio [OR], 1.34; 95% confidence interval [CI], 1.16-1.56) and one downstream of the genes PF4/PPBP/CXCL5 (rs1595009, P = 1.3 x 10(-4); OR, 1.32; 95% CI, 1.15-1.52), were associated with AgP.
our findings support CXCL5 as a promoter of colorectal cancer metastasis and a predictor of poor clinical outcomes in colorectal cancer patients.
CXCL5 levels were decreased in LSCC patient serum.
a finely tuned balance between the GAG-bound dimer and free soluble monomer regulates CXCL5-mediated receptor signaling and function.
CXCL5 plays a promoting role in glioma in autocrine- and paracrine-dependent manners.
The expression of CXCL5 is up-regulated in osteosarcoma cells.
CXCL5 expression in urine is related to bladder cancer TNM (zeige ODZ1 Antikörper) stage, lymph node metastasis, tumor size, and tumor grade.
ENA 78 plasma levels were correlated with Expanded Disability Status Scale scores in neuromyelitis optica (NMO) patients; elevated secretion of ENA 78 may be a critical step in neutrophil recruitment during the remission of NMO.
CXCL5 expression is enriched in human atherosclerotic coronary artery. The CXCL5 variant might be a genetic risk factor for the susceptibility of CAD (zeige CAD Antikörper) and the CXCL5 promoter -156 G/C C allele might be an independent predictor for CAD (zeige CAD Antikörper).
Study shows that CXCL5 expression is elevated in positive correlation to bladder cancer grade and promotes cell migration and invasion via binding to its receptor CXCR2 (zeige CXCR2 Antikörper).
Parenchymal polymorphonuclear myeloid-derived suppressor cell (PMN (zeige TBCE Antikörper)-MDSC), have a positive correlation with IL1a (zeige IL1A Antikörper), IL8 (zeige IL8 Antikörper), CXCL5, and Mip-1a (zeige CCL3 Antikörper), suggesting they may attract PMN (zeige TBCE Antikörper)-MDSC into the tumor
These data identify suppression of CXCL2 (zeige CXCL2 Antikörper) and CXCL5 chemoattractant expression by 11beta-HSD1 (zeige HSD11B1 Antikörper) as a novel mechanism with potential for regulation of neutrophil recruitment to the injured myocardium, and cardiac fibroblasts as a key site for intracellular glucocorticoid regeneration during acute inflammation following myocardial injury.
IL-17RA (zeige IL17RA Antikörper) regulates CXL-1 and 5 production in the lungs during the adaptive response.
STAT3 (zeige STAT3 Antikörper) is required for maximal OSM (zeige OSM Antikörper)-induced CXCL5 expression.
CXCL5 has a role in neutrophil recruitment in TH17-mediated glomerulonephritis
Since adaptive villus growth occurs despite impaired CXCL5 expression and enhanced angiogenesis, this suggests that the growth of new blood vessels is not needed for resection-induced mucosal surface area expansion following massive SBR (zeige NXF1 Antikörper).
CXCL5 regulates pulmonary responses to infection and plays a central role in conferring clock control of inflammation.
findings demonstrated that CXCL1 (zeige CXCL1 Antikörper) and CXCL5 are increased in circulation with onset of T2D, are produced by islets under stress, and synergistically affect islet function, suggesting that these chemokines participate in pathogenesis of T2D.
TLR2-induced epithelial-derived CXCL5 is critical for polymorphonuclear leukocyte-driven destructive inflammation in pulmonary tuberculosis.
Our data suggest that the differential regulation of the chemokine CXCL5 between osteoblasts and endothelial cells upon FGF2 treatment is involved in Hematopoietic stem cell mobilization from the osteoblast niche or bone marrow to peripheral blood.
The protein encoded by this gene is an inflammatory chemokine that belongs to the CXC chemokine family. This chemokine is produced concomitantly with interleukin-8 (IL8) in response to stimulation with either interleukin-1 (IL1) or tumor necrosis factor-alpha (TNFA). This chemokine is a potent chemotaxin involved in neutrophil activation.
C-X-C motif chemokine 5
, epithelial-derived neutrophil-activating protein 78
, neutrophil-activating peptide ENA-78
, neutrophil-activating protein 78
, small inducible cytokine subfamily B (Cys-X-Cys), member 5 (epithelial-derived neutrophil-activating peptide 78)
, CXC chemokine LIX
, chemokine (C-X-C motif) ligand 6 (granulocyte chemotactic protein 2)
, cytokine LIX
, small-inducible cytokine B5
, C-X-C motif chemokine 6
, chemokine (C-X-C motif) ligand 5
, granulocyte chemotactic protein 2
, inducible cytokine subfamily B (Cys-X-Cys), member 6
, small-inducible cytokine B6
, granulocyte chemotactic protein-2
, small inducible cytokine B subfamily, member 5
, small inducible cytokine subfamily B, member 15