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a significant increase in levels of adhesion molecules (VCAM-1) and selectins (E-selectin) in parallel with increased severity of diabetic retinopathy, with a significant difference of inflammatory markers between stages of retinopathy, is reported.
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Disease-modifying treatments are not responsible for the higher VAP-1 levels in patients with cerebral microbleeds (CMBs) in multiple sclerosis; instead, disease-modifying treatments could mask even higher VAP-1 levels in patients with CMBs
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Urinary angiostatin, CXC chemokine ligand 4 (CXCL4) and vascular cell adhesion molecule-1 (VCAM-1) were significantly higher in patients with active renal disease than in patients with active non-renal disease.
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Increased sEng and sVCAM-1 and decreased free VEGF plasma levels were found in women with severe preeclampsia, compared with normotensive pregnant group.
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VCAM-1 displays significantly higher expression on high-risk (symptomatic) vs low-risk (asymptomatic) carotid plaques. Ultrasound contrast agents bearing ligands for VCAM-1 can sustain high-shear attachment and may be useful for identifying patients in whom more aggressive treatment is warranted.
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Knockdown of YAP/TAZ using siRNA significantly reduced the expression of the leukocyte adhesion molecule VCAM1 induced by TNF-alpha.
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High VCAM1 expression is associated with Cardiovascular Disease.
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not a specific screening marker for pulmonary arterial hypertension in systemic sclerosis
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We observed an increase of VCAM-1 urine levels in diabetic nephropathy-basal patients compared to diabetic controls and an increase of urinary neprilysin in DN-treated patients with persistent albuminuria
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Urinary angiostatin and VCAM-1 are predictive of specific histological changes in concurrent lupus nephritis renal biopsies.
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In summary, it could be concluded that the expression of gene VCAM-1 was down regulated by taurocholic acid through miR-590-3p and that gene VCAM-1 could participate in the regulation of cell growth, cell proliferation and cell apoptosis
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VCAM-1 expression is upregulated by CD34+/CD133+-stem cells derived from septic patients
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Circulating soluble vascular cell adhesion molecule (VCAM-1) level was a useful diagnostic parameter for understanding the difference between coronary artery disease (CAD) and cardiac syndrome X (CSX).
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findings indicate a novel role for JAK2-V617F in activation of beta1 integrins and enhanced adhesion of granulocytes and 32D myeloid progenitors to VCAM1-coated surfaces
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High VCAM1 expression is associated with Cardiovascular Disease Risk.
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VCAM-1 levels are elevated in serum of cystic fibrosis patients.
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VCAM-1 levels are able to accurately predict significant fibrosis in non-alcoholic fatty liver disease patients
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These results revealed the effect of CD106 and NF-kappaB in bone marrow failure of Acquired aplastic anemia.
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Overexpression of LINC00341 suppresses the expression of vascular cell adhesion molecule 1 (VCAM1) and the adhesion of monocytes induced by atheroprone flow and tumor necrosis factor-alpha.
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Increased expression of PECAM-1, ICAM-3, and VCAM-1 in colonic biopsies from patients with inflammatory bowel disease (IBD) in clinical remission is associated with subsequent flares; this suggests that increases in the expression of these proteins may be early events that lead to flares in patients with IBD
