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MAGE-Like 2 Antikörper (AA 578-847)

Dieser Kaninchen Polyklonal Antikörper detektiert spezifisch MAGE-Like 2 in ELISA, WB, IHC (p) und FACS. Es zeigt Reaktivität gegenüber Proben von Human.
Produktnummer ABIN7875833
644,88 €
Zzgl. Versandkosten 20,00 € und MwSt
100 μg
Lieferung nach: Deutschland
Lieferung in 6 bis 9 Werktagen

Kurzübersicht für MAGE-Like 2 Antikörper (AA 578-847) (ABIN7875833)

Target

Alle MAGE-Like 2 (MAGEL2) Antikörper anzeigen
MAGE-Like 2 (MAGEL2)

Reaktivität

  • 34
  • 2
  • 2
  • 2
  • 2
  • 1
Human

Wirt

  • 34
  • 1
Kaninchen

Klonalität

  • 34
  • 1
Polyklonal

Konjugat

  • 10
  • 2
  • 2
  • 2
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
Dieser MAGE-Like 2 Antikörper ist unkonjugiert

Applikation

  • 13
  • 13
  • 11
  • 8
  • 8
  • 5
  • 3
  • 1
  • 1
ELISA, Western Blotting (WB), Immunohistochemistry (Paraffin-embedded Sections) (IHC (p)), Flow Cytometry (FACS)
  • Bindungsspezifität

    • 15
    • 8
    • 3
    • 1
    • 1
    AA 578-847

    Verwendungszweck

    MAGEL2 Antibody / MAGE-like protein 2

    Aufreinigung

    Antigen affinity purified

    Immunogen

    An E.coli-derived human recombinant protein (amino acids Q578-A847) was used as the immunogen for the MAGEL2 antibody.

    Isotyp

    IgG
  • Applikationshinweise

    Optimal dilution of the MAGEL2 antibody should be determined by the researcher.

    Beschränkungen

    Nur für Forschungszwecke einsetzbar
  • Format

    Lyophilized

    Buffer

    0.5 mg/mL if reconstituted with 0.2 mL sterile DI water

    Lagerung

    4 °C,-20 °C

    Informationen zur Lagerung

    After reconstitution, the MAGEL2 antibody can be stored for up to one month at 4oC. For long-term, aliquot and store at -20oC. Avoid repeated freezing and thawing.
  • Target

    MAGE-Like 2 (MAGEL2)

    Andere Bezeichnung

    MAGEL2

    Hintergrund

    Prader-Willi syndrome (PWS) is caused by the loss of expression of imprinted genes in chromosome 15q11-q13 region. Affected individuals exhibit neonatal hypotonia, developmental delay, and childhood-onset obesity. Necdin (NDN), a gene involved in the terminal differentiation of neurons, localizes to this region of the genome and has been implicated as one of the genes responsible for the etiology of PWS. This gene is structurally similar to NDN, is also localized to the PWS chromosomal region, and is paternally imprinted, suggesting a possible role for it in PWS.

    UniProt

    Q9UJ55
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