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ENPP2 Antikörper

ENPP2 Reaktivität: Human WB, ELISA Wirt: Ziege Polyclonal unconjugated
Produktnummer ABIN7581209
  • Target Alle ENPP2 Antikörper anzeigen
    ENPP2 (Ectonucleotide Pyrophosphatase/phosphodiesterase 2 (ENPP2))
    Reaktivität
    • 57
    • 44
    • 13
    • 4
    • 2
    • 2
    • 2
    • 2
    • 1
    • 1
    • 1
    Human
    Wirt
    • 71
    • 3
    • 1
    Ziege
    Klonalität
    • 73
    • 2
    Polyklonal
    Konjugat
    • 38
    • 4
    • 4
    • 3
    • 2
    • 2
    • 2
    • 2
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    Dieser ENPP2 Antikörper ist unkonjugiert
    Applikation
    • 38
    • 24
    • 14
    • 14
    • 12
    • 9
    • 7
    • 6
    • 4
    • 4
    • 3
    • 1
    • 1
    Western Blotting (WB), ELISA
    Verwendungszweck
    Goat polycolonal antibody targeting human Autotaxin (ATX)
    Spezifität
    Anti-ATX antibody recognizes endogenous human ATX and is predicted to recognize rat, mouse, horse, and feline ATX based on amino acid conservation.
    Aufreinigung
    affinity purified
    Immunogen
    ATX peptide
    Isotyp
    IgG
    Top Product
    Discover our top product ENPP2 Primärantikörper
  • Applikationshinweise
    WB – 1:1000, ELISA – 1:1000 Other in vitro and cellular applications are possible using this antibody, but have not been verified.
    Beschränkungen
    Nur für Forschungszwecke einsetzbar
  • Format
    Liquid
    Buffer
    PBS, pH 7.4
    Lagerung
    4 °C,-20 °C
    Informationen zur Lagerung
    Antibody is stable for up to 1 year at -20°C. Antibody is stable at 4°C for up to 60 days. Avoid repeated freeze/thaw cycles.
  • Target
    ENPP2 (Ectonucleotide Pyrophosphatase/phosphodiesterase 2 (ENPP2))
    Andere Bezeichnung
    Autotaxin (ENPP2 Produkte)
    Hintergrund
    ATX, also known as ENPP2, cleaves choline from lysophosphatidylcholine forming lysophosphatidic acid (LPA), a potent mitogen that has been implicated in the pathophysiology of ovarian cancer. ATX has been demonstrated to increase cell motility, neovascularization, proliferation, and aggressiveness of tumors and is upregulated in numerous cancer lineages (non-small cell lung , glioma, mammary carcinoma, renal cell carcinoma, hepatocellular carcinoma). In addition, dysregulation of the ATX/LPA pathway is central to the pathophysiology of idiopathic pulmonary fibrosis, rheumatoid arthritis, and other inflammatory diseases.
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