CXCL12 Antikörper (AA 23-89)
-
- Target Alle CXCL12 Antikörper anzeigen
- CXCL12 (Chemokine (C-X-C Motif) Ligand 12 (CXCL12))
-
Bindungsspezifität
- AA 23-89
-
Reaktivität
- Human
-
Wirt
- Kaninchen
-
Klonalität
- Polyklonal
-
Konjugat
- Dieser CXCL12 Antikörper ist unkonjugiert
-
Applikation
- Western Blotting (WB), Immunohistochemistry (IHC), Immunocytochemistry (ICC)
- Spezifität
- The antibody is a rabbit polyclonal antibody raised against SDF1. It has been selected for its ability to recognize SDF1 in immunohistochemical staining and western blotting.
- Aufreinigung
- Antigen-specific affinity chromatography
- Immunogen
- SDF1 (Pro23-Lys89)
- Isotyp
- IgG
- Top Product
- Discover our top product CXCL12 Primärantikörper
-
-
- Applikationshinweise
-
- Western blotting: 1:50-400 Immunocytochemistry in formalin fixed cells: 1:50-500 Immunohistochemistry in formalin fixed frozen section: 1:50-500 Immunohistochemistry in paraffin section: 1:10-100 Enzyme-linked Immunosorbent Assay: 1:100-1:5000 Optimal working dilutions must be determined by end user.
- Kommentare
-
The thermal stability is described by the loss rate. The loss rate was determined by accelerated thermal degradation test, that is, incubate the protein at 37°C for 48h, and no obvious degradation and precipitation were observed. The loss rate is less than 5% within the expiration date under appropriate storage condition.
- Beschränkungen
- Nur für Forschungszwecke einsetzbar
-
- Format
- Liquid
- Konzentration
- Lot specific
- Buffer
- PBS, pH 7.4, containing 0.02 % Sodium azide, 50 % glycerol.
- Handhabung
- Avoid repeated freeze-thaw cycles.
- Haltbarkeit
- 12 months
-
- Target
- CXCL12 (Chemokine (C-X-C Motif) Ligand 12 (CXCL12))
- Andere Bezeichnung
- Stromal Cell Derived Factor 1 (SDF1) (CXCL12 Produkte)
- Hintergrund
- Alternative Names: CXCL12, PBSF, P-BSF, SCYB12, SDF1-A, SDF1-B, SDF1A, SDF1B, TLSF-A, TLSF-B, TPAR1, Pre-B Cell Growth-Stimulating Factor, Chemokine C-X-C-Motif Ligand 12
- Pathways
- Regulation of Cell Size, CXCR4-mediated Signaling Events, Negative Regulation of intrinsic apoptotic Signaling
-