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Nonvoltage-gated, amiloride-sensitive, sodium channels control fluid and electrolyte transport across epithelia in many organs. Zusätzlich bieten wir Ihnen SCNN1B Proteine (7) und SCNN1B Kits (5) und viele weitere Produktgruppen zu diesem Protein an.
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Human Polyclonal SCNN1B Primary Antibody für WB - ABIN1881778
Song, Wei, Zhou, Lazrak, Liu, Londino, Squadrito, Matalon: Inhibition of lung fluid clearance and epithelial Na+ channels by chlorine, hypochlorous acid, and chloramines. in The Journal of biological chemistry 2010
Show all 3 Pubmed References
Human Polyclonal SCNN1B Primary Antibody für IF (p), IHC (p) - ABIN713471
Pasham, Pathare, Fajol, Rexhepaj, Michael, Pakladok, Alesutan, Rotte, Föller, Lang: OSR1-sensitive small intestinal Na+ transport. in American journal of physiology. Gastrointestinal and liver physiology 2012
The Epithelial Sodium Channel Is a Modifier of the Long-Term Nonprogressive Phenotype Associated with F508del CFTR (zeige CFTR Antikörper) Mutations
The derlin-1 (zeige DERL1 Antikörper) pathway therefore may represent a significant early checkpoint in the recognition and degradation of ENaC (zeige SCNN1A Antikörper) in mammalian cells.
hENaC incorporating the Liddle-mutated beta-subunit lacks one or more PKC phosphorylation sites, thereby significantly reducing the inhibitory effect of PKC on Na(+) channel activity, whereas hENaC incorporating Liddle-mutated gamma-subunits remains as susceptible to PKC as wild-type hENaC.
deltabetagamma-ENaC (zeige SCNN1A Antikörper) is inhibited by CFTR (zeige CFTR Antikörper) but activated by cyclic AMP (zeige APRT Antikörper).
Results identify SCNN1B as a tumor-suppressive function that triggers UPR in gastric cancer cells, with implications for its potential clinical applications as a survival biomarker in gastric cancer patients.
These results indicated a significant association between EH and SCNN1B methylation, which was affected by age, gender and antihypertensive therapy.
Three nonsynonymous amino acid variants in SCNN1B in nonwhite Cystic fibrosis (zeige S100A8 Antikörper) patients with non-diagnostic CFTR (zeige CFTR Antikörper) genotypes was
These results do not suggest an important role for the T594M variant of the ENaC (zeige SCNN1A Antikörper) gene contributing to either the development or severity of hypertension in subjects of Indo (zeige IDO1 Antikörper)-Aryan ancestry.
The transcriptional expression of alpha, beta, and gamma subunits of ENaC (zeige SCNN1A Antikörper) was elevated in nasal polyp compared to nasal mucosa
causative mutation for Liddle's syndrome (LS) in this patient was identified to be a novel frameshift mutation. DNA sequencing resulted in exon 13 of SCNN1B gene: SCNN1B NM_000336.2:c.1 724_1730dupGGCCCAC [p.Pro5 75Argfs*17].
Loss of betaENaC function resulted in meibomian gland disease and severe ocular surface damage that phenocopied aspects of human pseudohypoaldosteronism-1 MG disease and was sex dependent.
Nedd4-2 (zeige NEDD4L Antikörper)-mediated ubiquitination of beta-ENaC leading to endocytosis and degradation of apical Na(+) channels is a key feature of hypoxia-induced inhibition of transepithelial alveolar Na(+) transport.
Data indicate that insulin receptor (InsR (zeige INSR Antikörper)) is important for epithelial sodium channel (ENaC (zeige SCNN1A Antikörper)) activity.
Our findings are consistent with the role of betaENaC as a vascular smooth muscle cell mechanosensor and function of evolutionarily related nematode degenerin (zeige ACCN1 Antikörper) proteins.
These data suggest that adenylyl cyclase VI mediates vasopressin (zeige AVP Antikörper)-stimulated ENaC (zeige SCNN1A Antikörper) activity in the kidney.
Cys (zeige DNAJC5 Antikörper) substitutions at sites in the wrist domains of the beta and gamma subunits alter the Na+ self-inhibition response.
ENaC (zeige SCNN1A Antikörper) is not a passive passenger in regulated epithelial vesicle trafficking, but plays a role in establishing and maintaining the pool of vesicles that respond to cAMP stimulation.
These findings demonstrate that myogenic constriction in afferent arterioles is dependent on normal expression of betaENaC
loss of beta-ENaC associated with early signs of renal injury and increased mean arterial pressure
Nonvoltage-gated, amiloride-sensitive, sodium channels control fluid and electrolyte transport across epithelia in many organs. These channels are heteromeric complexes consisting of 3 subunits: alpha, beta, and gamma. This gene encodes the beta subunit, and mutations in this gene have been associated with pseudohypoaldosteronism type 1 (PHA1), and Liddle syndrome.
sodium channel, nonvoltage-gated 1, beta
, sodium channel, nonvoltage-gated 1, beta (Liddle syndrome)
, amiloride-sensitive sodium channel subunit beta
, amiloride-sensitive sodium channel subunit beta 1
, epithelial Na(+) channel subunit beta
, epithelial sodium channel beta-2 subunit
, epithelial sodium channel beta-3 subunit
, nasal epithelial sodium channel beta subunit
, nonvoltage-gated sodium channel 1 subunit beta
, Amiloride-sensitive sodium channel subunit beta
, epithelial amiloride-sensitive sodium channel beta subunit
, epithelial sodium channel ENaC beta subunit
, epithelial sodium channel, beta subunit
, amiloride sensitive epithelial sodium ion channel beta subunit
, epithelium sodium channel beta subunit
, ENaC beta
, amiloride sensitive sodium channel beta1 subunit
, sodium channel, nonvoltage-gated, type I, beta