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This is one of two genes encoding similar enzymes that catalyze the conversion of arachinodate to prostaglandin. Zusätzlich bieten wir Ihnen PTGS1 Kits (71) und PTGS1 Proteine (10) und viele weitere Produktgruppen zu diesem Protein an.
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Human Polyclonal PTGS1 Primary Antibody für IF (p), IHC (p) - ABIN670446
Li, Wang, Jiang, Xu, Zhang, Liu, Zhai: Combined effects of cyclooxygenase-1 and cyclooxygenase-2 selective inhibitors on ovarian carcinoma in vivo. in International journal of molecular sciences 2011
Show all 2 Pubmed References
Human Polyclonal PTGS1 Primary Antibody für IF, IHC (p) - ABIN1882120
Helmersson, Arnlöv, Axelsson, Basu: A polymorphism in the cyclooxygenase 1 gene is associated with decreased inflammatory prostaglandin F2alpha formation and lower risk of cardiovascular disease. in Prostaglandins, leukotrienes, and essential fatty acids 2009
This study showed that COX-1 and COX-2 (zeige PTGS2 Antikörper) in genital carcinomas in the horse is poor; microsomal PGES (zeige PTGES Antikörper)-1 is more prominently expressed.
COX-1 and COX-2 (zeige PTGS2 Antikörper) genes were constitutively expressed in baseline samples. Low-flow ischemia resulted in significant upregulation of COX-2 (zeige PTGS2 Antikörper) gene expression at each subsequent time point, compared with baseline values.
In this study, both COX-1 and COX-2 (zeige PTGS2 Antikörper) were expressed in the colon before induced ischemia; ischemic injury increased expression of COX-2 (zeige PTGS2 Antikörper).
Immunoreactivity for COX-1 and COX-2 (zeige PTGS2 Antikörper) is high in equine corneal SCC (zeige CYP11A1 Antikörper), possibly indicating that COX (zeige CPOX Antikörper) plays a role in oncogenesis or progression of this tumor type at this site.
These results suggest that licochalcones inhibit collagen-induced platelet aggregation accompanied by inhibition of COX-1 (zeige COX1 Antikörper) activity.
Data (including data from studies in knockout mice) suggest that delayed parturition in Cox-1 knockout mice is result of impaired luteolysis and cervical dilation, despite the presence of strong uterine contractions.
Data suggest that Il4 (zeige IL4 Antikörper) (usually released from helper T-cells) induces Cox1 (zeige COX1 Antikörper) in macrophages at post-transcriptional level; activation of Ampk (zeige PRKAA1 Antikörper) (catalytic subunit Prkaa1 (zeige PRKAA1 Antikörper)) by metformin blocks Il4 (zeige IL4 Antikörper)-dependent induction of Cox1 (zeige COX1 Antikörper) and blocks macrophage polarization/activation. (Il4 (zeige IL4 Antikörper) = interleukin-4 (zeige IL4 Antikörper); Cox1 (zeige COX1 Antikörper) = cyclooxygenase 1; Ampk (zeige PRKAA1 Antikörper) = AMP-activated protein kinase (zeige PRKAA2 Antikörper))
Specific inhibition of PGE2 synthesis by targeting mPGES-1 (zeige PTGES Antikörper) may weaken host defense against bacterial infections.
Dimethyl ester of bilirubin exhibits anti-inflammatory activity through inhibition of secretory phospholipase A2 (zeige YWHAZ Antikörper), lipoxygenase and cyclooxygenase.
role of cyclooxygenase-1 and -2 in endothelium-dependent contraction of atherosclerotic mouse abdominal aortas
Suggest the expression of COX-1 and COX-2 (zeige COX2 Antikörper) in the urothelium protects bladder damage from radiation.
COX-1 inhibitor SC-560 has a protective effect on the thromboxane A2-mediated decrease in renal function in response to endotoxin.
Expression of COX-1 is essential for the protection of liver against chemical-induced hepatotoxicity and required for hepatic homeostatic maintenance.
Data suggest that multitarget FAAH (zeige FAAH Antikörper)/Cox (zeige CPOX Antikörper) blockade may provide a transformative approach to inflammatory bowel disease (IBD) and other pathologies in which fatty acid amide hydrolase (zeige FAAH Antikörper)/cyclooxygenases (FAAH (zeige FAAH Antikörper), Cox-1, and Cox-2 (zeige COX2 Antikörper)) are overactive.
In arteries from non-insulin (zeige INS Antikörper)-dependent diabetic mice, COX-1 remains a major contributor to the endothelial PGI2 (zeige PTGIR Antikörper) synthesis that evokes vasoconstrictor activity under the pathological condition.
there was no expression of COX-1, either mRNA or protein, on any day of the estrous cycle
Results suggested that in Chinese Han patients with stroke taking aspirin for secondary prevention, PTGS1 polymorphisms may increase the risk of poor functional outcomes and this effect may be modulated by the smoking status. PTGS1 gene-smoking interaction might in part reflect the heterogeneity in the prognosis of patients treated with aspirin.
analysis of co-existing variants in both F8 and PTGS-1 genes in a three-generation pedigree of hemophilia A; the PTGS-1 variant was associated with specific functional defects in the arachidonic acid pathway and more severe hemorrhage.
Data suggest expression of PTGS1 in colon is significantly correlated with expression of PTGS2 (zeige PTGS2 Antikörper), PTGES1, PTGER2 (zeige PTGER2 Antikörper), and PTGER4 (zeige PTGER4 Antikörper); this study was conducted in individuals at high risk for colon cancer treated with Mediterranean diet versus a Healthy Eating diet for prevention of colon cancer. (PG = prostaglandin; PTGS2 (zeige PTGS2 Antikörper) = PG-endoperoxide synthase 2; PTGES1 = PGE (zeige LIPF Antikörper) synthase protein; PTGER2 (zeige PTGER2 Antikörper) = PGE (zeige LIPF Antikörper) receptor 2; PTGER4 (zeige PTGER4 Antikörper) = PGE (zeige LIPF Antikörper) receptor 4)
Panax quinquefolium saponin attenuated HUVEC apoptosis and improved the dual antiplatelet-mediated reduction of platelet adhesion to injured HUVECs and the underlying mechanisms may be associated with PI3K (zeige PIK3CA Antikörper)/AKT (zeige AKT1 Antikörper) and COX (zeige COX8A Antikörper) pathways.
The interactions of COX (zeige COX8A Antikörper)-1of rs3842787 and cox-2 (zeige COX2 Antikörper) of rs20417 were associated with aspirin resistance of stroke.
We provide the first report that pro-angiogenic genes PECAM1 (zeige PECAM1 Antikörper), PTGS1, FGD5 (zeige FGD5 Antikörper), and MCAM (zeige MCAM Antikörper) may play a vital role in pathological dermal angiogenesis disorders of psoriasis.
a new neutrophil-activating platelet-derived lipid generated by COX-1 (zeige COX1 Antikörper) is presented that can activate or prime human neutrophils, suggesting a role in innate immunity and acute inflammation.
Seminal COX-1 (zeige COX1 Antikörper) is over-expressed in infertile oligoasthenoteratozoospermic (OAT (zeige OAT Antikörper)) men with varicocele (Vx) compared with fertile men with/without and infertile OAT (zeige OAT Antikörper) men without Vx being associated with oxidative stress, Vx grade and Vx laterality.
In Indian peptic ulcer hemorrhage patients, those with Cox-1 (zeige COX1 Antikörper) A842G polymorphisms tended to have less gastric ulcers among those with the A842G/C50T polymorphism.
Brain death increases the expression of COX-1 (zeige COX1 Antikörper) and COX-2 (zeige PTGS2 Antikörper) mRNA in the renal medulla
Endometrial prostaglandin-endoperoxide synthase 1 (PTGS1) mRNA expression increased 2- to 3-fold after Day 10 of the estrous cycle and pregnancy, whereas PTGS2 (zeige PTGS2 Antikörper) mRNA expression increased 76-fold between Days 5 and 15 of the estrous cycle and pregnancy.
This is one of two genes encoding similar enzymes that catalyze the conversion of arachinodate to prostaglandin. The encoded protein regulates angiogenesis in endothelial cells, and is inhibited by nonsteroidal anti-inflammatory drugs such as aspirin. The protein may promote cell proliferation during tumor progression. Alternative splicing results in multiple transcript variants.
, prostaglandin G/H synthase 1
, prostaglandin G/H synthase and cyclooxygenase
, prostaglandin-endoperoxide synthase 1 (prostaglandin G/H synthase and cyclooxygenase)
, PGH synthase 1
, PHS 1
, prostaglandin H2 synthase 1
, prostaglandin-endoperoxide synthase 1
, cyclooxygenase 1
, cyclooxygenase 3
, prostaglandin endoperoxide synthase