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The ninjurin protein is upregulated after nerve injury both in dorsal root ganglion neurons and in Schwann cells (Araki and Milbrandt, 1996 [PubMed 8780658]). Zusätzlich bieten wir Ihnen Ninjurin 1 Antikörper (44) und Ninjurin 1 Kits (15) und viele weitere Produktgruppen zu diesem Protein an.
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Data show that Ninj1 assembles into a homomeric protein complex and that N-glycosylation is a prerequisite for Ninj1 homomer assembly.
Ninj1 suppresses migration, invasion and metastasis of lung cancer via inhibition of the IL-6 signaling pathway in vitro and in vivo.
High ninjurin 1 expression is associated with Prostate Cancer.
Ninj1 expression was detected in both HSMCs and HUVECs, and was higher in the former. Ninj1 expression was enhanced by hypoxia in HUVECs.
Ninj1 modulates TLR4 signaling, which itself plays a major role in systemic inflammatory response syndrome and sepsis.
The NINJ1 gene polymorphism plays an important role in nerve degeneration in leprosy patients.
Data show an important cell-specific role for Ninjurin-1 in the transmigration of inflammatory APCs across the BBB.
Ninjurin1 is able to induce the senescence program. Ninjurin1 may be involved in the regulation of cellular senescence in the liver during carcinogenesis.
Our data suggests that Ninjurin 1 asp110ala SNP could be a valuable genetic marker of nerve damage in leprosy.
The ninjurin protein is upregulated after nerve injury both in dorsal root ganglion neurons and in Schwann cells (Araki and Milbrandt, 1996
, nerve injury-induced protein 1
, nerve injury-induced protein-1