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guanylyl cyclase\; functions as receptor for heat-stable enterotoxin responsible for acute diarrhea [RGD, Feb 2006]..
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Human Polyclonal GUCY2C Primary Antibody für IHC, IHC (p) - ABIN4316480
Brenna, Bruland, Furnes, Granlund, Drozdov, Emgård, Brønstad, Kidd, Sandvik, Gustafsson: The guanylate cyclase-C signaling pathway is down-regulated in inflammatory bowel disease. in Scandinavian journal of gastroenterology 2015
High Expressions of guanylyl cyclase C is associated with rectal cancer.
The findings support that the activating mutation in GUCY2C creates an intestinal environment with a major influence on the microbiota, which could contribute to the increased susceptibility to inflammatory bowel disease in patients with Familial GUCY2C diarrhea syndrome.
Results demonstrate that GC-C and its ligands, guanylin (zeige GUCA2A Antikörper) and uroguanylin (zeige GUCA2B Antikörper) are downregulated in ulcerative colitis (UC), and this downregulation is more significant with aggravation of the clinical condition. Therefore, the GC-C signaling pathway may be implicated in the progression of UC.
the expression of GCC is maintained throughout the process of tumor progression and formation of metastatic disease.
To investigate gut (zeige GUSB Antikörper) motility and hormones before and after a meal in Familial GUCY2C diarrhea syndrome patients and compare with healthy controls
Mutations in GUCY2C indicate a role for this receptor in the pathogenesis of sporadic Congenital secretory diarrhea.
these data support Ad5 (zeige PSEN2 Antikörper)-GUCY2C-PADRE as a safe and effective vaccination strategy in preclinical models and position Ad5 (zeige PSEN2 Antikörper)-GUCY2C-PADRE for Phase I clinical testing in colorectal cancer patients.
Familial GUCY2C diarrhoea syndrome is caused by an activating mutation in the GUCY2C gene, which causes impaired contractility and fluid stagnation in the small bowel
Findings show how caloric suppression of the guanylin (zeige GUCA2A Antikörper)-GUCY2C signaling axis links obesity to negation of a universal tumor suppressor pathway in colorectal cancer.
Data show that individuals affected with meconium ileus (MI) had either homozygous or compound heterozygous variants in enterotoxin receptor GUCY2C.
Loss of Gucy2c expression is associated with Acute Radiation-Induced GI Syndrome.
The GC-C signaling pathway blunts colonic mucosal inflammation that is initiated by systemic cytokine burst or loss of mucosal immune cell immunosuppression.
GC-C signaling is an essential component of host defense during murine enteric infection by reducing bacterial load and preventing systemic dissemination of attaching/effacing-lesion forming bacterial pathogens such as C. rodentium.
Intestinal cell proliferation and senescence are regulated by receptor guanylyl cyclase C and p21 (zeige D4S234E Antikörper).
GUCY2C opposes systemic genotoxic tumorigenesis by regulating AKT (zeige AKT1 Antikörper)-dependent intestinal barrier integrity
The role for GUCY2C-directed CD8 (zeige CD8A Antikörper)(+) T cells targeting specific epitopes in antitumor efficacy.
Data show that silencing of GUCY2C in mice disrupts satiation, resulting in hyperphagia and subsequent obesity and metabolic syndrome.
a novel role for GC-C signaling in facilitating mucosal wounding and inflammation, and further suggest that this may be mediated, in part, through control of RELMbeta (zeige RETNLB Antikörper) production
guanylyl cyclase\; functions as receptor for heat-stable enterotoxin responsible for acute diarrhea
, STA receptor
, guanylyl cyclase C
, heat-stable enterotoxin receptor
, intestinal guanylate cyclase
, heat stable enterotoxin receptor
, Guanylate cyclase 2C (heat stable enterotoxin receptor)
, guanylate cyclase 2C
, guanylate cyclase 2C (heat stable enterotoxin receptor)
, heat-stable enterotoxin receptor-like