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GFAP encodes one of the major intermediate filament proteins of mature astrocytes. Zusätzlich bieten wir Ihnen GFAP Antikörper (835) und GFAP Proteine (34) und viele weitere Produktgruppen zu diesem Protein an.
Showing 10 out of 76 products:
Rat (Rattus) GFAP ELISA Kit für Sandwich ELISA - ABIN416142
Tskitishvili, Nisolle, Munaut, Pequeux, Gerard, Noel, Foidart: Estetrol attenuates neonatal hypoxic-ischemic brain injury. in Experimental neurology 2014
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Human GFAP ELISA Kit für Sandwich ELISA - ABIN365729
Akdemir, Yardan, Kati, Duran, Alacam, Yavuz, Okuyucu: The role of S100B protein, neuron-specific enolase, and glial fibrillary acidic protein in the evaluation of hypoxic brain injury in acute carbon monoxide poisoning. in Human & experimental toxicology 2014
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Rat (Rattus) GFAP ELISA Kit für Sandwich ELISA - ABIN367441
Moallem, Mohamadpour, Abnous, Sankian, Sadeghnia, Tsatsakis, Shahsavand: Erythropoietin in the treatment of carbon monoxide neurotoxicity in rat. in Food and chemical toxicology : an international journal published for the British Industrial Biological Research Association 2015
Human GFAP ELISA Kit für Sandwich ELISA - ABIN414459
Ekingen, Yilmaz, Yildiz, Atescelik, Goktekin, Gurger, Alatas, Basturk, Ilhan: Utilization of glial fibrillary acidic protein and galectin-3 in the diagnosis of cerebral infarction patients with normal cranial tomography. in Nigerian journal of clinical practice 2017
There was significantly more GFAP immunoreactivity in the prefrontal cortex and hippocampus of aged animals compared to adult or middle-aged animals.
GFAP, along with tau and AmyloidBeta42, were increased in plasma up to 90 days after traumatic brain injury compared with controls.
Results show that the positive rates and expression levels of nestin (zeige NES ELISA Kits), tyrosine hydroxylase (TH (zeige TH ELISA Kits)), GFAP and IL-17 (zeige IL17A ELISA Kits) were significantly decreased while Foxp3 (zeige FOXP3 ELISA Kits) and the ratio of Foxp3 (zeige FOXP3 ELISA Kits)/IL-17 (zeige IL17A ELISA Kits) were statistically elevated in BM of AML (zeige RUNX1 ELISA Kits) patients.
GFAP levels >0.29 ng/ml were seen only in intracerebral hemorrhage, thus confirming the diagnosis of ICH (zeige COL4a2 ELISA Kits) during prehospital care.
These results indicate that autoantibodies against GFAP could serve as a predictive marker for the development of overt autoimmune diabetes.
Higher median plasma GFAP values were documented in intracerebral hemorrhage compared with acute ischemic stroke, stroke mimics, and controls.
GFAP is specifically expressed in the auricular chondrocytes, and assumes a pivotal role in resistance against mechanical stress.
Bevacizumab treatment was also associated with structural protein abnormalities, with decreased GFAP and vimentin (zeige VIM ELISA Kits) content and upregulated GFAP and vimentin (zeige VIM ELISA Kits) mRNA expression.
Tat (zeige TAT ELISA Kits) expression or GFAP expression led to formation of GFAP aggregates and induction of unfolded protein response (UPR) and endoplasmic reticulum (ER) stress in astrocytes.
This study demonstrated that GFAP exhibited distinct temporal profiles over the course of 7 days in patient with traumatic brain injury.
e data indicates that serum GFAP levels may be associated with severity of autism spectrum disorders among Chinese children.
Isolation of an evolutionary conserved novel GFAP isoform, GFAPkappa, produced by alternative splicing and polyadenylation of the 3'-region of the human GFAP pre-mRNA is described.
compared open-skull and thinned-skull imaging methods for two-photon laser microscopy of live astrocytes in neocortex of GFAP-GFP transgenic mice
work reveals that an Alexander disease-causing mutation alters GFAP turnover kinetics in vivo and provides an essential foundation for future studies aimed at preventing or reducing the accumulation of GFAP.
Study provides evidence that transcription of one of the astrocyte-specific genes, Gfap, is cooperatively regulated by co-expressed genes and their regulatory factors.
This study demonstrated the GFAP-ApoE4 mice exhibited motor impairments when compared to GFAP-ApoE3 and wild-type mice.
PINK1 deficiency causes defects in GFAP-positive astrogliogenesis during brain development.
Gnasxl (zeige GNAS ELISA Kits) deficiency does not directly affect glial development in the hypothalamus, since it is expressed in neurons, and Gfap-positive astrocytes and tanycytes appear normal during early postnatal stages.
Induction of glial cytokine expression was sequential, aligned with active sickness behavior, and preceded increased Iba-1 (zeige AIF1 ELISA Kits) or GFAP immunoreactivity after lipopolysaccharide challenge
The distribution of GFAP immunoreactivity implies that enteric glia are widespread in the fish gastrointestinal tract.
Generation of transgenic zebrafish that express green fluorescent protein (GFP) in glial cells driven by the zebrafish glial fibrillary acidic protein (GFAP) regulatory elements.
Cells expressing the two reporters display radial glial morphology, colocalize with the NSC marker Sox2 (zeige SOX2 ELISA Kits), undergo proliferation, and are capable of self-renewal within the matrix of distinct thickness in the telencephalon.
This gene encodes one of the major intermediate filament proteins of mature astrocytes. It is used as a marker to distinguish astrocytes from other glial cells during development. Mutations in this gene cause Alexander disease, a rare disorder of astrocytes in the central nervous system. Alternative splicing results in multiple transcript variants encoding distinct isoforms.
glial fibrillary acidic protein
, glial fibrillary acidic protein alpha
, intermediate filament
, intermediate filament protein
, zrf-1 antigen