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Tumor suppressor that is required to sustain G2/M checkpoint after DNA damage.
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FATS status is a biomarker in breast cancer to identify individuals likely to benefit from radiotherapy
Data identified a genetic variant in the FATS gene (905C > T, 262D/N) that is associated with susceptibility to breast cancer in a parity-dependent manner.
FATS is an E2-independent ubiquitin ligase that stabilizes p53 (zeige TP53 Antikörper) and promotes its activation in response to DNA damage.
FATS is involved in the carcinogenesis and development of breast cancer and could be a potential biomarker and prognostic factor for breast cancer therapy.
data suggest centrosomal functions for C10orf90 and KIAA1731 (zeige KIAA1731 Antikörper) and new centriole-related functions for ALMS1 (zeige ALMS1 Antikörper)
Tumor suppressor that is required to sustain G2/M checkpoint after DNA damage. Mediates CDKN1A/p21 protein stability in a ubiquitin-independent manner (By similarity). May have a role in the assembly of primary cilia.
centrosomal protein C10orf90
, fragile-site associated tumor suppressor homolog