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The protein encoded by BTG3 is a member of the BTG/Tob family. Zusätzlich bieten wir Ihnen BTG3 Kits (15) und BTG3 Proteine (5) und viele weitere Produktgruppen zu diesem Protein an.
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Human Polyclonal BTG3 Primary Antibody für ICC, IF - ABIN4285522
Deng, Zhao, Gou, Chen, Mao, Takano, Zheng: Decreased expression of BTG3 was linked to carcinogenesis, aggressiveness, and prognosis of ovarian carcinoma. in Tumour biology 2013
Deficiency of antiproliferative family protein Ana correlates with development of lung adenocarcinoma.
Results suggest that ANA is a suppressor of ectopic bone formation induced by bone morphogenetic proteins, and this inhibitory ANA activity is a part of the negative feedback regulation of BMP function.
Data show that miR (zeige MLXIP Antikörper)-139 can repress the proliferation of hepatocellular carcinoma (HCC (zeige FAM126A Antikörper)) cells via directly inhibiting the expression of ANA protein, human Add B-cell translocation gene 3 protein (BTG3).
Taken together, the results of our study suggest that BTG3 overexpression could inhibit cell proliferation and invasion and promotes cell apoptosis in EOC cell, possibly by regulating the AKT (zeige AKT1 Antikörper)/GSK3beta/beta-catenin (zeige CTNNB1 Antikörper) signaling pathway
BTG3 expression might contribute to CRC (zeige CALR Antikörper) carcinogenesis. BTG3 knockdown might strengthen the aggressive colorectal cancer behavior.
BTG3 is a direct downstream target of miR (zeige MLXIP Antikörper)-106b-5p.
BTG3 overexpression inhibited tumor growth of SW620 cells by suppressing proliferation and inducing apoptosis. It was suggested that down-regulated BTG3 expression might be considered as a marker for colorectal carcinogenesis.
ANA and ASMA (zeige ACTA1 Antikörper) evaluation in patients with liver transplantation and no history of autoimmune disease has no clinical relevance, since it varies in time and is not related to any risk factors or liver injury. Routine autoimmunity evaluation should be avoided.
BTG3 overexpression might reverse the aggressive phenotypes and be employed as a potential target for gene therapy of gastric cancer.
The suppressed migration and invasion abilities found in BTG3-overexpressing esophageal adenocarcinoma cells. Our findings suggested that BTG3 is suppressor in the progression of esophageal adenocarcinoma
BTG3 binds and suppresses AKT (zeige AKT1 Antikörper), a kinase frequently deregulated in cancers.
Down-regulation of BTG3 promotes cell proliferation, migration and invasion in gastric cancer.
The protein encoded by this gene is a member of the BTG/Tob family. This family has structurally related proteins that appear to have antiproliferative properties. This encoded protein might play a role in neurogenesis in the central nervous system. Two transcript variants encoding different isoforms have been found for this gene.
B cell translocation protein 3
, protein BTG3
, B-cell translocation gene 3
, BTG family member 3
, abundant in neuroepithelium area protein
, protein Tob5