anti-Amylase 1, Salivary (AMY1) Antikörper

Mouse (Murine) Amylase 1, Salivary (AMY1) Interaktionspartner

1. our findings suggest that AMY1 copy number differences play a role in childhood-onset obesity but the effect differs between males and females.

2. Data indicate that the amylase (zeige AMY Antikörper) 1 (AMY1) copy number in an individual's genome is generally even and partially correlates with nearby single nucleotide polymorphism (SNPs), which do not associate with body mass index (BMI).

Human Amylase 1, Salivary (AMY1) Interaktionspartner

1. 66 proteins that interact with amylase (zeige AMY Antikörper) in whole saliva (zeige RAG1AP1 Antikörper) were identified.

2. AMY1 (zeige AMY1A Antikörper) copy number was significantly correlated with the variation observed in salivary amylase (zeige AMY Antikörper) production and enzyme activity but did not explain the majority of observed variation between individuals. AMY1 (zeige AMY1A Antikörper)-odd and AMY1 (zeige AMY1A Antikörper)-even haplotypes showed a different relationship between copy number and expression levels, but the difference was not statistically significant.

3. Alcohol dependent patients showed significantly lower stress-related salivary alpha-amylase (zeige AMY1A Antikörper) activity than healthy controls.

4. Results from 2 case-control cohorts of Chinese and Malays, show no previously reported association between AMY1 (zeige AMY1A Antikörper) and obesity or body mass index.

5. It was concluded that the genetic variant determining starch metabolism influences the response to weight-loss dietary intervention. Overweight and obese individuals carrying the AMY1-AMY2 (zeige AMY Antikörper) rs11185098 genotype associated with higher amylase (zeige AMY Antikörper) activity may have greater loss of adiposity during low-calorie diet interventions.

6. higher levels of sAA (zeige SAA1 Antikörper) in EHS participants

7. Serum amylase (zeige AMY Antikörper) levels in the normal range are positively associated with integrated islet beta cell function in patients with early type 2 diabetes.

8. Our findings suggest an effect of the interaction between starch intake and AMY1 (zeige AMY1A Antikörper) copy number on obesity. Individuals with high starch intake but low genetic capacity to digest starch had the lowest BMI, potentially because larger amounts of undigested starch are transported through the gastrointestinal tract, contributing to fewer calories extracted from ingested starch.

9. Data show that carcinoembryonic antigen (CEA (zeige CEACAM5 Antikörper)) modestly differentiated between mucinous and nonmucinous lesions, and amylase (zeige AMY Antikörper) did not distinguish intraductal papillary mucinous neoplasms (IPMNs) from mucinous cystadenomas (MCAs).

10. genetic association studies in a population of men in Republic of Korea: Data suggest that low AMY1A (zeige AMY1A Antikörper) gene copy number is associated with high insulin (zeige INS Antikörper) resistance and thus with genetic predisposition to diabetes type 2 and metabolic syndrome.