Use your antibodies-online credentials, if available.
Keine Produkte auf Ihrer Vergleichsliste.
Ihr Warenkorb ist leer.
Alle Spezies anzeigen
Weitere Synonyme anzeigen
Wählen Sie die Spezies und Applikation aus
anti-Human S100A9 Antikörper:
anti-Rat (Rattus) S100A9 Antikörper:
anti-Mouse (Murine) S100A9 Antikörper:
Sie gelangen zu unserer vorgefilterten Suche.
Human Polyclonal S100A9 Primary Antibody für ICC, IF - ABIN409938
Sumi, Shimizu, Himeno: Involvement of Nrf2 activation in the upregulation of S100A9 by exposure to inorganic arsenite. in International journal of molecular medicine 2012
Show all 6 Pubmed References
Human Monoclonal S100A9 Primary Antibody für EIA, FACS - ABIN111889
Brandtzaeg, Jones, Flavell, Fagerhol: Mac 387 antibody and detection of formalin resistant myelomonocytic L1 antigen. in Journal of clinical pathology 1989
Show all 11 Pubmed References
Human Monoclonal S100A9 Primary Antibody für EIA, FACS - ABIN111888
Delabie, de Wolf-Peeters, van den Oord, Desmet: Differential expression of the calcium-binding proteins MRP8 and MRP14 in granulomatous conditions: an immunohistochemical study. in Clinical and experimental immunology 1990
Show all 9 Pubmed References
Human Monoclonal S100A9 Primary Antibody für EIA, FACS - ABIN238498
Mortezai, Harder, Schnabel, Moors, Gauly, Schlüter, Wagener, Buck: Tandem affinity depletion: a combination of affinity fractionation and immunoaffinity depletion allows the detection of low-abundance components in the complex proteomes of body fluids. in Journal of proteome research 2010
Show all 2 Pubmed References
Human Monoclonal S100A9 Primary Antibody für ELISA, WB - ABIN948434
Li, Riau, Setiawan, Mehta, Ti, Tong, Tan, Beuerman: S100A expression in normal corneal-limbal epithelial cells and ocular surface squamous cell carcinoma tissue. in Molecular vision 2011
Show all 2 Pubmed References
Human Polyclonal S100A9 Primary Antibody für WB - ABIN519967
Bianchi, Niemiec, Siler, Urban, Reichenbach: Restoration of anti-Aspergillus defense by neutrophil extracellular traps in human chronic granulomatous disease after gene therapy is calprotectin-dependent. in The Journal of allergy and clinical immunology 2011
Show all 2 Pubmed References
Human Polyclonal S100A9 Primary Antibody für IHC (p), IHC - ABIN449656
McCormick, Rahimi, Bobryshev, Gaus, Zreiqat, Cai, Lord, Geczy: S100A8 and S100A9 in human arterial wall. Implications for atherogenesis. in The Journal of biological chemistry 2005
Show all 2 Pubmed References
Human Monoclonal S100A9 Primary Antibody für IA, FACS - ABIN2192037
Robinson, Tessier, Poulsom, Hogg: The S100 family heterodimer, MRP-8/14, binds with high affinity to heparin and heparan sulfate glycosaminoglycans on endothelial cells. in The Journal of biological chemistry 2002
Human Monoclonal S100A9 Primary Antibody für ELISA, WB - ABIN519968
Feng, Lee, Chang, Chan, Kuo, Lin, Chung, Kuo, Yu, Lin, Wang, Chou, Huang, Kuo: CD14(+)S100A9(+) monocytic myeloid-derived suppressor cells and their clinical relevance in non-small cell lung cancer. in American journal of respiratory and critical care medicine 2012
An immunohistochemical investigation revealed that patients with both UC and CD had higher neutrophil and monocyte/macrophage calprotectin (zeige S100A8 Antikörper)-positive cell expression levels, compared with those in the normal controls. Fecal calprotectin (zeige S100A8 Antikörper) was considered a reliable marker for disease activity, and the assessment of fecal calprotectin (zeige S100A8 Antikörper) via POCT showed potential as a rapid and simple measurement in clinical settings.
the levels of S100A8 (zeige S100A8 Antikörper) and S100A9 in the sera, and calprotectin (zeige S100A8 Antikörper) levels in the sera and urines of bladder cancer patients, were determined.
MRP-8 (zeige S100A8 Antikörper)/14 concentration increases in coronary artery blood in association with thrombus formation in acute coronary syndrome.
ANGPTL6 (zeige ANGPTL6 Antikörper), S100A8 (zeige S100A8 Antikörper) and S100A9 are overexpressed in skin of psoriasis patients versus zero expression in controls.
To examine the efficacy of faecal calprotectin (zeige S100A8 Antikörper) (FC) and C-reactive protein (CRP (zeige CRP Antikörper)) as predictors of small bowel capsule endoscopy SBCE findings in suspected and known Crohn's disease.
S100A9, mainly produced by myeloid cells in the BM, is involved in the pathogenesis of MM. S100A9 attracted MM cells to the BM where it provided a survival niche by inducing the growth factors IL6 (zeige IL6 Antikörper) and IL10 (zeige IL10 Antikörper), as well as promoting angiogenesis.
Study identified S100A9 protein to be up-regulated protein in hepatocellular carcinoma (HCC (zeige FAM126A Antikörper)) and demonstrated its potentiality as a candidate for HCC (zeige FAM126A Antikörper) diagnosis.
In pediatric inflammatory bowel disease treatment groups, a significant inverse association was found between the quality of life and the levels of fecal calprotectin (zeige S100A8 Antikörper).
Fecal calprotectin (zeige S100A8 Antikörper) (FC) was elevated in two-thirds of stool specimens of patients with cystic fibrosis (zeige S100A8 Antikörper) (CF). Increased FC was more common in patients with pancreatic insufficiency. Whether increased FC reflects intestinal inflammation in patients with CF remains to be determined.
In healthy children, 95th Percentile values ranged from 1519 mg/kg at 0 months to 54.4 mg/kg at 144 months for fecal calprotectin (zeige S100A8 Antikörper) (fCP) and from 9.9 mg/kg at 0 months to 0.2 mg/kg at 144 months for fecal eosinophil-derived neurotoxin (zeige RNASE2 Antikörper) (fEDN). There was a significant association between age and fCP concentrations and age and fEDN concentrations and a significant association between fEDN and fCP.
Data suggest that up-regulation of S100A8 (zeige S100A8 Antikörper) and S100A9 is a key component of early endometrial response to uterine involution in the post-partum period and to prevent chronic endometritis/uterine inflammation; up-regulation can be influenced by diet.
Study verified porcine calprotectin (S100A8 (zeige S100A8 Antikörper)/A9) expression at the protein level in multiple Haemophilus parasuis infected tissues and explored their molecular characterization.
this study shows that S100A9 inhibits B lymphopoiesis by acting on myeloid cells and promoting the release of inflammatory molecules, including IL-1beta (zeige IL1B Antikörper)
Chronic Pseudomonas aeruginosa biofilm infection reduces murine S100A8 (zeige S100A8 Antikörper)/ S100A9 expression and delays species-specific burn wound closure.
data suggest that S100A8 (zeige S100A8 Antikörper)/A9 acts directly on BV-2 microglial cells via binding to TLR4 (zeige TLR4 Antikörper) and RAGE (zeige AGER Antikörper) on the membrane and then stimulates the secretion of proinflammatory cytokines through ERK (zeige EPHB2 Antikörper) and JNK (zeige MAPK8 Antikörper)-mediated NF-kappaB (zeige NFKB1 Antikörper) activity in BV-2 microglial cells.
Like S100A8 (zeige S100A8 Antikörper), S100A9 and S100A8 (zeige S100A8 Antikörper)/A9 reduced neutrophil influx in acute lung injury provoked by lipopolysaccharide (LPS (zeige TLR4 Antikörper)) challenge.
findings reveal unexpected gene expression differences between WT and KO mice at a young age (in the absence of physiological stress), and address the hypothesis that Mrp8 (zeige S100A8 Antikörper) and Mrp14 accumulation promotes age-related inflammation
S100A8 (zeige S100A8 Antikörper) and S100A9 aggravate Coxsackievirus B3-induced myocarditis and might serve as therapeutic targets in inflammatory cardiomyopathies.
An excessive expansion of monocytic-myeloid-derived suppressor cells in S100A9-/- neonates compared to wild-type neonates. This strong baseline expansion was associated with hyperinflammatory responses during endotoxemia and fatal septic courses
Data support a role of S100A9 to initiate and amplify the neutrophilic inflammation in asthma, possibly via inducing IL-1beta (zeige IL1B Antikörper), IL-17 (zeige IL17A Antikörper) and IFN-gamma (zeige IFNG Antikörper).
The findings show that reduced PTP1B (zeige PTPN1 Antikörper) responses contribute to disease symptoms in part by enhancing S100A9 expression during viral-associated chronic obstructive pulmonary disease exacerbations.
Neutrophil-derived S100A8 (zeige S100A8 Antikörper)/A9 promotes thrombocytosis in diabetic mice
S100A9 knockdown almost completely abrogated the effects of IRF7 (zeige IRF7 Antikörper) deletion on granulocytic myeloid-derived suppressor cells development and tumor metastasis.
Calprotectin (zeige S100A8 Antikörper) expression in laminar epidermis occurs after extravasation of leukocytes indicating that leukocyte emigration may be an initiating factor in laminar epithelial stress and inflammation in black walnut extract-induced laminitis.
The protein encoded by this gene is a member of the S100 family of proteins containing 2 EF-hand calcium-binding motifs. S100 proteins are localized in the cytoplasm and/or nucleus of a wide range of cells, and involved in the regulation of a number of cellular processes such as cell cycle progression and differentiation. S100 genes include at least 13 members which are located as a cluster on chromosome 1q21. This protein may function in the inhibition of casein kinase and altered expression of this protein is associated with the disease cystic fibrosis.
, S100 calcium-binding protein A9 (calgranulin B)
, calgranulin B
, calprotectin L1H subunit
, leukocyte L1 complex heavy chain
, migration inhibitory factor-related protein 14
, protein S100-A9
, S100 calcium binding protein A9 (calgranulin B)
, intracellular calcium-binding protein (MRP14)
, myeloid-related protein 14
, protein MRP-126
, S100 calcium binding protein A9
, S100 calcium-binding protein A9
, neutrophil cytosolic 23 kDa protein
, RNA-binding region containing protein 2-like