anti-Cholecystokinin (CCK) Antikörper

Cow (Bovine) Cholecystokinin (CCK) Interaktionspartner

1. This review explores the role of luminal cholecystokinin on the regulation of exocrine pancreatic secretion in author's studies of newborn calves.

Pig (Porcine) Cholecystokinin (CCK) Interaktionspartner

1. We conclude that only a small fraction of neuronal CCK is nonsulfated. The intracellular distribution of nonsulfated CCK in neurons suggests that they contribute only modestly to the CCK transmitter activity.

2. Cardiac expression of pro-cholecystokinin is cell-specific, which differentiates the expression from that of intestinal endocrine cells and cerebral neurons.

3. This review explores the role of luminal cholecystokinin on the regulation of exocrine pancreatic secretion in author's studies of neonatal pigs.

Human Cholecystokinin (CCK) Interaktionspartner

1. L-trp (zeige TBPL1 Antikörper) is a luminal regulator of CCK release with effects on gastric emptying, an effect that could be mediated by CCK. L-trp's effect on GLP-1 (zeige GCG Antikörper) secretion is only minor. At the doses given, the two amino acids did not affect subjective appetite feelings.

2. The CCK polymorphism have reported significant association of -45C>T polymorphism with the presence of hallucinations.

3. CCK does not appear to play a unique independent role in satiety/satiation.

4. CCK release has been found to be halved in pregnant women with hyperemesis gravidarium, which supports the hypothesis that gastrointestinal motility is increased in pregnant women with hyperemesis gravidarium.

5. CCK in plasma is an independent marker of cardiovascular mortality in elderly female patients.

6. These data offer preliminary evidence supporting an association between the rs1799923 polymorphism in the CCK gene and PTSD

7. Data suggest that endocrine responses differ between jejunal and gastric enteral feeding, with higher peak plasma CCK (cholecystokinin), PYY (peptide YY), and GLP-1/2 (glucagon-like peptides 1/2) concentrations being attained after jejunal feeding.

8. active GLP-1 (zeige GCG Antikörper) produced in the islet stimulates cholecystokinin production and secretion in a paracrine manner via cyclic AMP (zeige APRT Antikörper) and CREB (zeige CREB1 Antikörper).

9. Cardiac expression of pro-cholecystokinin is cell-specific, which differentiates the expression from that of intestinal endocrine cells and cerebral neurons. Plasma Pro-CCK is a prognostic marker in patients with stable heart failure.

10. CCK binding modulates the contractile function of the lower esophageal sphincter through differential binding to the CCK-A receptor (zeige CCKAR Antikörper) on the sling and clasp (zeige CLASRP Antikörper) fibers

Mouse (Murine) Cholecystokinin (CCK) Interaktionspartner

1. In summary, our study is the first to indicate that CCK/CCK-AR (zeige CCKAR Antikörper) pathway is critical and protective against liver I/R injury. The activation of this pathway not only prevents hepatocellular apoptosis, but also reduces inflammatory response by suppressing NF-kappaB (zeige NFKB1 Antikörper).

2. CCK/GLP-1 (zeige GCG Antikörper) play contributory roles in anorexia induction by trichothecenes T-2 toxin, HT-2 toxin, diacetoxyscirpenol and neosolaniol.

3. GPR120 (zeige O3FAR1 Antikörper)-induced incretin glucse-dependent insulinotropic polypeptide secretion is indirectly mediated by cholecystokinin.

4. Medullary interstitial cells respond to body fluid expansion by CCK release for feedback regulation of the late proximal tubular reabsorption.

5. results suggest that normal integration of CCK+ basket cells in cortical networks is key to support spatial coding in the hippocampus.

6. PC7 (zeige PCSK7 Antikörper) has a critical role in normal processing of cholecystokinin in mouse brain

7. These studies reemphasize the beneficial effects imparted by co-administration of obestatin and CCK8 and their potential use towards countering obesity.

8. new information on the cell specific localization of NUCB2/nesfatin-1 (zeige NUCB2 Antikörper) in the intestinal mucosa, and a novel function for nesfatin-1 (zeige NUCB2 Antikörper) in modulating intestinal CCK and PYY expression and secretion

9. Results showed that CCK is important for lipid transport and energy expenditure to control body weight in response to dietary lipid feeding

10. SP1 (zeige SP1 Antikörper) results in a CCK response deficiency that may contribute to the increased meal size and overall hyperphagia in synphillin-1 transgenic mice