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Both XBcl9 and XPygo2 are required to induce supernumerary axis and dorsal gene activation in Xenopus embryos.
Transcriptional cofactors Bcl9, Bcl9l (zeige BCL9L ELISA Kits) and Pygo1/2 act independently of beta-catenin (zeige CTNNB1 ELISA Kits) to ensure proper enamel formation.
ARX positively regulates Wnt (zeige WNT2 ELISA Kits)/ beta-catenin (zeige CTNNB1 ELISA Kits) signaling and the C-terminal domain of ARX interacts with the armadillo (zeige PKP1 ELISA Kits) repeats in beta-catenin (zeige CTNNB1 ELISA Kits) to promote Wnt (zeige WNT2 ELISA Kits)/beta-catenin (zeige CTNNB1 ELISA Kits) signaling. In addition, we found BCL9 and P300 (zeige NOTCH1 ELISA Kits) also interact with ARX to modulate Wnt (zeige WNT2 ELISA Kits)/beta-catenin (zeige CTNNB1 ELISA Kits) signaling.
Study demonstrates that the Golgi resident protein GM130 (zeige GOLGA2 ELISA Kits) activates the spindle assembly factor TPX2 (zeige DAZL ELISA Kits) to nucleate microtubules around the Golgi and further captures them to couple mitotic membranes to the spindle.
Pax6 (zeige PAX6 ELISA Kits), the master regulator of eye development, directly activates Bcl9/9l transcription.
These results suggest a critical role of BCL9/9-2 in the Wnt (zeige WNT2 ELISA Kits)-mediated regulation of adult, as opposed to embryonic, myogenic progenitors.
MEF2D (zeige MEF2D ELISA Kits)-BCL9-positive patients had B-cell precursor immunophenotype and were characterized as being older in age, being resistant to chemotherapy, having very early relapse, and having leukemic blasts that mimic morphologically mature B-cell leukemia with markedly high expression of HDAC9 (zeige HDAC9 ELISA Kits).
it was demonstrated that miR218 modulated a novel molecular target and the present study provided novel insights into potential mechanisms of RCC (zeige XRCC1 ELISA Kits) oncogenesis.
findings indicate that BCL9 most likely does not harbor a common genetic variant that can increase the risk for schizophrenia in the Japanese population
BCL9/9L-beta-catenin (zeige CTNNB1 ELISA Kits) Signaling is Associated With Poor Outcome in Colorectal Cancer
BCL9 is a molecular driver of DCIS invasive progression.
PCDH10 (zeige PCDH10 ELISA Kits) antagonized MM cell proliferation via the downregulation of Wnt (zeige WNT2 ELISA Kits)/beta-catenin (zeige CTNNB1 ELISA Kits)/BCL-9 signaling, whereas PCDH10 (zeige PCDH10 ELISA Kits) repressed the expression of AKT (zeige AKT1 ELISA Kits) to promote the expression of GSK3beta (zeige GSK3b ELISA Kits) and then to restrain the activation of beta-catenin (zeige CTNNB1 ELISA Kits)
By beta-catenin's association with LEF1 (zeige LEF1 ELISA Kits) and BCL9-2/B9L (zeige BCL9L ELISA Kits).
MiR (zeige MLXIP ELISA Kits)-30-5p downregulation occurs as a result of interaction between multiple myeloma cells and bone marrow stromal cells, which in turn enhances expression of BCL9.
we detected five SNPs in the first two genes/loci - BCL9 and C9orf5 - strongly associated with negative symptoms of schizophrenia
Inhibition of the BCL9-beta-catenin (zeige CTNNB1 ELISA Kits) interaction and selectively suppresses oncogenic Wnt (zeige WNT2 ELISA Kits) transcription.
BCL9 is associated with B-cell acute lymphoblastic leukemia. It may be a target of translocation in B-cell malignancies with abnormalities of 1q21. Its function is unknown. The overexpression of BCL9 may be of pathogenic significance in B-cell malignancies.
B-cell CLL/lymphoma 9
, B-cell lymphoma 9
, B-cell CLL/lymphoma 9 protein-like
, b-cell CLL/lymphoma 9 protein-like
, B-cell CLL/lymphoma 9 protein
, B-cell lymphoma 9 protein
, nuclear co-factor of beta-catenin signalling
, protein legless homolog