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Anti-SmD1 antibodies are associated with renal disorder, seizures, and pulmonary arterial hypertension in Chinese patients with active systemic lupus erythematosus.
Data suggest that SNRPA1, SNRPD1, and PNN are key players in the regulation of pluripotency-specific spliceosome assembly and the acquisition and maintenance of pluripotency.
Data show that the elevation of cerebrospinal fluid anti-Sm antibodies through transudation from systemic circulation due to damaged blood-brain barrier (BBB) plays a critical role in the pathogenesis of acute confusional state (ACS).
The interaction of SMN with the spliceosomal SmD1 (also known as SNRPD1), severely decreases SMN-coilin interaction and prevents Cajal body assembly.
Overexpression of SNRPD1 is associated with highly aggressive breast cancers.
data suggest that the anti-Sm D response arises as a consequence of antigenic cross-reactivity by anti-EBNA-1 antibodies, supporting a possible association between EBV infection and SLE
SmD1 83-119 peptide may play a dominant role in the activation of helper and regulatory T cells that influence autoantibody generation and murine lupus.
This gene encodes a small nuclear ribonucleoprotein that belongs to the SNRNP core protein family. The protein may act as a charged protein scaffold to promote SNRNP assembly or strengthen SNRNP-SNRNP interactions through nonspecific electrostatic contacts with RNA.
, small nuclear ribonucleoprotein D1 polypeptide 16kDa pseudogene
, small nuclear ribonucleoprotein Sm D1
, snRNP core protein D1
, small nuclear ribonucleoprotein D2 polypeptide 16.5kDa
, sm-D autoantigen