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anti-Human ARNTL Antikörper:
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Human Polyclonal ARNTL Primary Antibody für ChIP, ICC - ABIN151688
Hayashida, Kuramoto, Koyanagi, Oishi, Fujiki, Matsunaga, Ikeda, Ohdo, Shimeno, Soeda: Proxisome proliferator-activated receptor-α mediates high-fat, diet-enhanced daily oscillation of plasminogen activator inhibitor-1 activity in mice. in Chronobiology international 2010
Show all 11 Pubmed References
Human Monoclonal ARNTL Primary Antibody für IHC, ELISA - ABIN968967
Motzkus, Loumi, Cadenas, Vinson, Forssmann, Maronde: Activation of human period-1 by PKA or CLOCK/BMAL1 is conferred by separate signal transduction pathways. in Chronobiology international 2007
Show all 3 Pubmed References
Human Monoclonal ARNTL Primary Antibody für IHC, ELISA - ABIN965595
Lee, Lee, Lee, Park, Kang, Chung, Lee, Kim: Dual modification of BMAL1 by SUMO2/3 and ubiquitin promotes circadian activation of the CLOCK/BMAL1 complex. in Molecular and cellular biology 2008
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Human Monoclonal ARNTL Primary Antibody für FACS, IF - ABIN2716387
Trejo-Muñoz, Navarrete, Montúfar-Chaveznava, Caldelas: Temporal modulation of the canonical clockwork in the suprachiasmatic nucleus and olfactory bulb by the mammary pheromone 2MB2 in pre-visual rabbits. in Neuroscience 2014
Polyclonal ARNTL Primary Antibody für ChIP, IP - ABIN540781
Oishi, Sakamoto, Okada, Nagase, Ishida: Antiphase circadian expression between BMAL1 and period homologue mRNA in the suprachiasmatic nucleus and peripheral tissues of rats. in Biochemical and biophysical research communications 1999
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Human Polyclonal ARNTL Primary Antibody für ELISA, WB - ABIN449912
Fuller, Lu, Saper: Differential rescue of light- and food-entrainable circadian rhythms. in Science (New York, N.Y.) 2008
ARNTL rs7107287 was associated with a cyclothymic temperament, depressive and stress symptoms
NR1D1 (zeige NR1D1 Antikörper) and BMAL1 mRNA and protein levels were significantly reduced in OA compared to normal cartilage. In cultured human chondrocytes, a clear circadian rhythmicity was observed for NR1D1 (zeige NR1D1 Antikörper) and BMAL1.
These results suggest that the circadian clock system can be recovered through BMAL1 expression induced by aza-dC within a day.
Determined a novel role of TNF-alpha (zeige TNF Antikörper) in inducing Bmal1 via dual calcium dependent pathways; Roralpha was up-regulated in the presence of Ca(2 (zeige CA2 Antikörper)+) influx and Rev-erbalpha (zeige NR1D1 Antikörper) was down-regulated in the absence of that.
results of this study suggest that genetic variability in the ARNTL and CLOCK genes might be associated with risk for multiple sclerosis
PI3K (zeige PIK3CA Antikörper)-PTEN upregulated-mTORC1 and mTORC2 (zeige CRTC2 Antikörper) complex plays a critical role in controlling BMAL1, establishing a connection between PI3K (zeige PIK3CA Antikörper) signaling and the regulation of circadian rhythm, ultimately resulting in deregulated BMAL1 in tumor cells with disrupted PI3K (zeige PIK3CA Antikörper) signaling
M. tuberculosis infection caused enhanced MMP-1 (zeige MMP1 Antikörper), -9, and miR (zeige MLXIP Antikörper)-223 expression, with inhibited BMAL1 expression. MiR (zeige MLXIP Antikörper)-223 modulated BMAL1 expression via the direct binding of BMAL1 3'-UTR (zeige UTS2R Antikörper).
research describes an association between changes in the methylation of the BMAL1 gene with the intervention and the effects of a weight loss intervention on blood lipids levels
Study found rhythmic methylation of BMAL1 was altered in Alzheimer's disease brains and fibroblasts and correlated with transcription cycles. Results indicate that cycles of DNA methylation (zeige HELLS Antikörper) contribute to the regulation of BMAL1 rhythms in the brain.
TFEB (zeige TFEB Antikörper) regulates PER3 expression via glucose-dependent effects on CLOCK/BMAL1
the second half of the photolyase homology region (PHR) of CRY (zeige CRY2 Antikörper) is important for repression through facilitating interaction with BMAL1
Results indicated that pBMAL1 cDNA had a coding region of 1,878 bp and shared 94.36, 89.85 and 89.79% identity with human, mouse and rat BMAL1. pBMAL1 expression levels in different tissues were studied.
The expression of the clock gene Bmal1 in myeloid cells regulates the innate and adaptive immune responses that mediate autoimmune diseases.
Insulin (zeige INS Antikörper) reduces Bmal1 transcriptional activity by affecting its intracellular localization in mouse liver, which requires Akt (zeige AKT1 Antikörper)-dependent phosphorylation of the Ser42 residue.
Bmal1 modulates lipoprotein production and biliary cholesterol excretion by regulating the expression of Mtp (zeige LAPTM4A Antikörper) and Abcg5 (zeige ABCG5 Antikörper)/Abcg8 (zeige ABCG8 Antikörper) via Shp (zeige LAMC1 Antikörper) and Gata4 (zeige GATA4 Antikörper).
these results suggest a role for bmal1 in the development of asthmatic airway disease via the regulation of lung antiviral responses to common viral triggers of asthma
The daily rhythm of BMAL1 mRNA was completely abolished in the EGR1 (zeige EGR1 Antikörper)-deficient mice.
Bmal1 and Dio2 (zeige DIO2 Antikörper) are required to maintain cone photoreceptor functional integrity.
Islet cell maturation was also characterized by induction in the expression of circadian transcription factor BMAL1, deletion of which altered postnatal development of glucose-stimulated insulin (zeige INS Antikörper) secretion and the associated transcriptional network.
PML (zeige PML Antikörper) mediates the binding of PER2 (zeige PER2 Antikörper) to BMAL1 in the BMAL1/CLOCK heterodimer and is an important component in the organization of a functional clock complex in the nucleus.
The results demonstrate that Bmal1 expression in skeletal muscle is both necessary and sufficient to regulate total sleep amount and reveal that critical components of normal sleep regulation occur in muscle.
The findings indicate that BMAL1 deregulation is a feature of the mTOR (zeige FRAP1 Antikörper)-activated state and suggest a molecular mechanism for mitigating circadian phenotypes in a neurodevelopmental disorder.
The protein encoded by this gene is a basic helix-loop-helix protein that forms a heterodimer with CLOCK. This complex binds an E-box upstream of the PER1 gene, activating this gene and possibly other circadian rhythym-associated genes. Three transcript variants encoding two different isoforms have been found for this gene.
ARNT-like protein 1, brain and muscle
, PAS domain-containing protein 3
, aryl hydrocarbon receptor nuclear translocator-like protein 1
, bHLH-PAS protein JAP3
, basic-helix-loop-helix-PAS orphan MOP3
, basic-helix-loop-helix-PAS protein MOP3
, brain and muscle ARNT-like 1
, class E basic helix-loop-helix protein 5
, member of PAS protein 3
, member of PAS superfamily 3
, brain and muscle ARNT-like protein 1
, bHLH-PAS transcription factor BMAL1
, clock protein
, aryl hydrocarbon receptor nuclear translocator-like
, Brain and muscle ARNT-like 1
, aryl hydrocarbon receptor nuclear translocator-like protein 1-like
, brain and muscle Arnt-like protein 1
, bHLH-PAS transcription factor