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anti-Human TRAF5 Antikörper:
anti-Mouse (Murine) TRAF5 Antikörper:
anti-Rat (Rattus) TRAF5 Antikörper:
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Gerbil Polyclonal TRAF5 Primary Antibody für IHC (p), IP - ABIN537431
Zapata, Krajewska, Krajewski, Kitada, Welsh, Monks, McCloskey, Gordon, Kipps, Gascoyne, Shabaik, Reed: TNFR-associated factor family protein expression in normal tissues and lymphoid malignancies. in Journal of immunology (Baltimore, Md. : 1950) 2000
High TRAF5 expression is associated with colorectal cancer.
Study presents the characterization of the peptide binding preferences of TRAFs 2, 3, and 5 using deep mutational scanning. The three TRAF (zeige TRAF1 Antikörper) proteins demonstrated different preferences for binding to members of the CD40 (zeige CD40 Antikörper) library, and three peptides from that library individually showed striking differences in affinity for the three TRAFs.
These findings indicate that miR (zeige MLXIP Antikörper)-26b might influence TRAF5-MAPK (zeige MAPK1 Antikörper) signaling pathways to facilitate the malignant progression of melanoma cells.
findings reveal a direct link between TRAF5-mediated ubiquitination and RORgammat protein regulation
RIG-I (zeige DDX58 Antikörper)-like receptor mediated antiviral innate immune responses in the lower respiratory tract involves TRAF3 (zeige TRAF3 Antikörper) and TRAF5 signaling.
This study provides evidence that TRAF5 and TRAF3IP2 (zeige TRAF3IP2 Antikörper) genes are involved in the development of Behcet's disease (BD) and Vogt-Koyanagi-Harada (VKH) syndrome.
Data indicate that TNF receptor-associated factor 5 (TRAF5) gene is involved in the development of acute anterior uveitis (AAU) and pediatric uveitis.
Data indicate that in monocyte- derived macrophages (MDMs) acutely infected with HIV-1 and treated with HCV rCore and HIV-1 rNef, the HIV-1 replication depends on an upstream signal mediated through TRAF2 (zeige TRAF2 Antikörper), TRAF5 and TRAF6 (zeige TRAF6 Antikörper).
TRAF3 (zeige TRAF3 Antikörper) and TRAF5 are overexpressed in inflammatory bowel disease
Numbl (zeige NUMBL Antikörper) interacted with tumor necrosis factor (zeige TNF Antikörper) receptor-associated factor 5, which signals upstream and is required for the activation of NF-kappaB (zeige NFKB1 Antikörper), and committed it to proteasomal degradation by promoting K48-linked polyubiquitination of TRAF5
TRAF5 is crucially involved in OSM (zeige OSM Antikörper)-mediated lung inflammation probably by inducing lung stromal/fibroblast cell activation.
our data indicate that Berberine exerts protective effects against chronic renal failurepartly through the TRAF5-mediated activation of the NF-kappaB (zeige NFKB1 Antikörper) signaling pathway in mouse podocytes
this study shows that TRAF2 (zeige TRAF2 Antikörper) and TRAF5 work as important regulators of the IL-6R signaling needed for Th17 development
The results indicated that TRAF5 deficiency significantly aggravated DSS (zeige PMP22 Antikörper)-induced colitis, most likely by regulating Th cell-mediated inflammation.
In conclusion, our results suggest that as an intrinsic cardioprotective factor, TRAF5 plays a crucial role in the development of cardiac hypertrophy through the negative regulation of the MEK-ERK1/2 pathway
TRAF5 negatively regulates TLR signaling in B lymphocytes.
These results provide the first demonstration that TRAF5 is a critical mediator of brain ischemia/reperfusion in an experimental stroke model.
In vitro and cellular experiments showed that forms of TRAF5 with mutation of the corresponding residues to those of TRAF3 (zeige TRAF3 Antikörper) had TRAF3 (zeige TRAF3 Antikörper)-like antiviral activity.
Treatment with IL-17 (zeige IL17A Antikörper) prolongs the half-life of chemokine (zeige CCL1 Antikörper) CXCL1 (zeige CXCL1 Antikörper) mRNA via the adaptor TRAF5 and the splicing-regulatory factor SF2 (ASF (zeige SRSF1 Antikörper)).
TNF receptor-associated factor 5 (TRAF5) deficiency accelerates atherogenesis in mice by increasing inflammatory cell recruitment and foam cell formation.
The scaffold protein encoded by this gene is a member of the tumor necrosis factor receptor-associated factor (TRAF) protein family and contains a meprin and TRAF homology (MATH) domain, a RING-type zinc finger, and two TRAF-type zinc fingers. TRAF proteins are associated with, and mediate signal transduction from members of the TNF receptor superfamily. This protein is one of the components of a multiple protein complex which binds to tumor necrosis factor (TNF) receptor cytoplasmic domains and mediates TNF-induced activation. Alternate transcriptional splice variants have been characterized.
TNF receptor-associated factor 5
, RING finger protein 84