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anti-Human RHOA Antikörper:
anti-Mouse (Murine) RHOA Antikörper:
anti-Rat (Rattus) RHOA Antikörper:
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Human Monoclonal RHOA Primary Antibody für IF, IHC (p) - ABIN559931
Bu, Erlander, Hitz, Tillakaratne, Kaufman, Wagner-McPherson, Evans, Tobin: Two human glutamate decarboxylases, 65-kDa GAD and 67-kDa GAD, are each encoded by a single gene. in Proceedings of the National Academy of Sciences of the United States of America 1992
Show all 4 Pubmed References
Human Polyclonal RHOA Primary Antibody für FACS, IF (cc) - ABIN738846
Li, Sun, Yang, Hu, Jiang: Tanshinone II a protects against lipopolysaccharides-induced endothelial cell injury via Rho/Rho kinase pathway. in Chinese journal of integrative medicine 2014
Human Polyclonal RHOA Primary Antibody für IF (p), IHC (p) - ABIN758242
Zhang, He, Lu, Bian, Yang, Fu, Wu, Li: HSF1 Relieves Amyloid-?-Induced Cardiomyocytes Apoptosis. in Cell biochemistry and biophysics 2015
Data show that p120-catenin (zeige CTNND1 Antikörper) interacts with kinesin family member 23 (MKLP1 (zeige KIF23 Antikörper)) to regulate focused rhoA GTP-binding protein (zeige HCAR3 Antikörper) (RhoA) activity during cytokinesis.
Data show that RhoA is upregulated in invasive glioblastoma tissues, and its activation participates in Wnt5a (zeige WNT5A Antikörper)-induced glioblastoma cell invasion.
PCGEM1 may be an inducer in epithelial ovarian cancer tumorigenesis and progression by upregulating RhoA and the subsequent expression of YAP (zeige YAP1 Antikörper), P70S6K (zeige RPS6KB1 Antikörper), MMP2 (zeige MMP2 Antikörper), and Bcl-xL (zeige BCL2L1 Antikörper).
This study identified Piezo2 (zeige FAM38B Antikörper) mechanosensitive cationic channel as a transducer of environmental physical cues into mechanobiological responses.
High RHOA expression is associated with gastric cancer invasion and metastasis.
Results show that RHOA expression level is under the regulation of NORAD to promote EMT (zeige ITK Antikörper) in pancreatic neoplasm.
Data show that phosphorylation of ribosomal protein S6 kinase 2 (RSK2 (zeige RPS6KA3 Antikörper)) at threonine 577 is essential for leukemia-associated RhoGEF (LARG (zeige ARHGEF12 Antikörper))-dependent Rho GTPase (zeige RACGAP1 Antikörper) activation.
RhoA activated the downstream Wnt/betacatenin signaling pathway and inhibited the expression of apoptotic factors.
We identify HGF (zeige HGF Antikörper), acting through the c-Met receptor, as the key polarity-inducing morphogen (zeige SHH Antikörper), which acts to activate b1-integrin-dependent adhesion. HGF (zeige HGF Antikörper) and ECM (zeige MMRN1 Antikörper)-derived integrin signals co-operate via a c-Src (zeige SRC Antikörper)-dependent inhibition of the RhoA-ROCK1 (zeige ROCK1 Antikörper) signalling pathway via p190A (zeige GRLF1 Antikörper) RhoGAP (zeige ARHGAP1 Antikörper)
Extracellular Hsp90alpha mediates house dust mite-induced human bronchial epithelial barrier dysfunction by activating RhoA/MLC signaling, suggesting that eHsp90alpha is a potential therapeutic target for treatment of asthma.
Non-visual arrestins regulate the focal adhesion formation via small GTPases RhoA and Rac1 independently of G-protein-coupled receptors.
RHOA loss reduces YAP (zeige YAP1 Antikörper) signaling of the Hippo pathway and affects YAP (zeige YAP1 Antikörper) effector epiregulin (EREG (zeige EREG Antikörper)) expression in the crypts. Expression of an active YAP (zeige YAP1 Antikörper) (S112A) mutant rescues ntestinal stem cells (ISCs (zeige NFS1 Antikörper)) marker expression, ISC regeneration, and ISC-associated Wnt (zeige WNT2 Antikörper) signaling, but not defective epithelial polarity, in RhoA knockout mice, implicating YAP (zeige YAP1 Antikörper) in RHOA-regulated ISC function.
GTP (zeige AK3 Antikörper)-RhoA and ROCK1 (zeige ROCK1 Antikörper) expression levels were markedly increased in a time-dependent manner in the ears and lungs of mice treated with penicillin.
Tyr42 phosphorylation of RhoA GTPase promotes tumorigenesis through NF-kappaB (zeige NFKB1 Antikörper).
RhoA modulates the choices of molar cuspal shape by coordinating adhesion junctions, actin distribution, and fibronectin (zeige FN1 Antikörper) localization to drive inner dental epithelium invagination.
Kctd13 deletion reduces synaptic transmission, which correlates with increased levels of RhoA, a KCTD13/CUL3 ubiquitin ligase substrate
Crmp4(-/-) OBs exhibited enhanced activation of RhoA/focal adhesion kinase (FAK) signaling that led to cytoskeletal changes with increased cell spreading.
Codepletion of the actomyosin regulator RhoA and Afadin (zeige MLLT4 Antikörper) results in defects in the central lumens and arrests lumen remodeling
Hydrogen peroxide oxidizes RhoA at Cys16 and Cys20, and activates RhoA via Vav2 (zeige VAV2 Antikörper).
These results reveal a novel signaling network, the Sema4D (zeige SEMA4D Antikörper)-RhoA-Akt (zeige AKT1 Antikörper) signal cascade, that coordinates cellular function and morphology and highlights the importance of specific spatiotemporally restricted components of a signaling pathway in the regulation of ameloblast differentiation.
Mgc's GAP activity down-regulates the active populations of RhoA and Rac1 at localized regions of epithelial cells and is necessary for successful cytokinesis and cell-cell junction structure
Data show that shortly after anaphase onset oocytes and embryonic cells exhibit cortical waves of Rho activity and F-actin polymerization.
CASZ1 (zeige CASZ1 Antikörper)/Egfl7 (zeige EGFL7 Antikörper)/RhoA pathway is necessary for promoting endothelial cell behaviors associated with proper vascular assembly.
RhoA can be considered a component of the intracellular pattern formation system.
Kazrin (zeige KAZ Antikörper) interacts with ARVCF (zeige ARVCF Antikörper)-catenin, spectrin and p190B (zeige ARHGAP5 Antikörper) RhoGAP (zeige ARHGAP1 Antikörper), and modulates RhoA activity.
Morphogenesis of the primitive gut tube is generated by Rho/ROCK/myosin II-mediated endoderm rearrangements.
RhoA and membrane fluidity mediates the spatially polarized Src (zeige SRC Antikörper)/FAK (zeige PTK2 Antikörper) activation in response to shear stress.
the Lbc (zeige AKAP13 Antikörper)/alpha-catulin (zeige CTNNAL1 Antikörper) axis participates in 5-HT (zeige DDC Antikörper)-induced PASMC mitogenesis and RhoA/ROCK signaling, and may be an interventional target in diseases involving vascular smooth muscle remodeling.
The RhoA/ROCK signaling pathway is an important negative regulator of vascular calcification.
Vascular endothelial-cadherin signals through RhoA and actin cytoskeletal and affects cell-matrix adhesion
Thrombospondin has a role in inducing RhoA inactivation through FAK (zeige PTK2 Antikörper)-dependent signaling to stimulate focal adhesion disassembly
KCl directly increased Rho and ROCK activities in a concentration-dependent fashion that paralleled closely the effect of KCl on lung smooth muscle tone and [Ca(2 (zeige CA2 Antikörper)+)](i), as well as the voltage-dependent Ca(2 (zeige CA2 Antikörper)+) currents
the Rho-ROCK signal pathway contributes to VEGF (zeige VEGFA Antikörper)-induced hyperpermeability. Myosin light-chain phosphorylation and actin stress fiber formation occur concomitantly with the increase in permeability upon VEGF (zeige VEGFA Antikörper) stimulation.
Formation of polygonal actin network in endothelial cells is a novel rhoA associated response to hypertonic stress.
Cadherins, RhoA and Rac1, have important roles in mechanotransduction and that endothelial and smooth muscle cells use different mechanisms to respond to stretch.
Results indicate that hypergravity induces ATP release and actin reorganization via RhoA activation and FAK (zeige PTK2 Antikörper) phosphorylation, thereby activating cell proliferation and migration in bovine aortic endothelial cells.
Pseudorabies virus US3 expression led to RhoA phosphorylation at serine 188 to induce actin rearrangements.
Data indicate that TNF-alpha (zeige TNF Antikörper) stimulates Rac (zeige AKT1 Antikörper), ADAM17/TACE (zeige ADAM17 Antikörper), and RhoA through the guanine nucleotide exchange factor (zeige ARHGEF12 Antikörper) (GEF)-H1 (zeige ARHGEF2 Antikörper).
Contractile pulmonary arterial myocytes exhibit marked Rho-dependent actin polymerization in hypoxia, with increased active RhoA and LIMK (zeige LIMK1 Antikörper) phosphorylation.
Results suggest that Rac1 and RhoA are regulated by TGFbeta1 (zeige TGFB1 Antikörper) in the process of endothelial tube formation in collagen I gels.
The concentration of RhoA mRNA and activated RhoA enzyme were greater in urothelium than in detrusor. Rho kinase (zeige ROCK1 Antikörper) inhibitor Y-27632 showed a stronger inhibitory effect in detrusor with intact urothelium.
Thrombin (zeige F2 Antikörper) stimulates swine smooth muscle cell differentiation from peripheral blood mononuclear cells via protease-activated receptor-1 (zeige F2R Antikörper), RhoA, and myocardin (zeige MYOCD Antikörper).
Activating Rho could be beneficial to suppress Kras mutant-induced liver malignancies.
Ras homolog, or Rho, proteins interact with protein kinases and may serve as targets for activated GTPase. They play a critical role in muscle differentiation. The protein encoded by this gene binds GTP and is a member of the small GTPase superfamily. It is involved in endosome dynamics and reorganization of the actin cytoskeleton, and it may coordinate membrane transport with the function of the cytoskeleton.
Aplysia ras-related homolog 12
, oncogene RHO H12
, ras homolog gene family, member A
, rho cDNA clone 12
, small GTP binding protein RhoA
, transforming protein RhoA
, Rho-related protein HP1
, ras homolog D
, ras homolog gene family, member D
, rho-related GTP-binding protein RhoD
, Ras family member A
, Rho family GTPase
, aplysia ras-related homolog A
, aplysia ras-related homolog A1
, aplysia ras-related homolog A2
, ras homolog A1
, ras homolog A2
, ras homolog gene family, member A1
, ras homolog gene family, member A2
, plysia ras-related homolog A2
, rho1 GTP-binding protein
, RhoA GTPase
, Rho A