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anti-Human AGT Antikörper:
anti-Mouse (Murine) AGT Antikörper:
anti-Rat (Rattus) AGT Antikörper:
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Human Polyclonal AGT Primary Antibody für ICC, IF - ABIN447446
Dimitrijevic, Rissler, Luts, Edvinsson: Reduced expression of angiotensin II and angiotensin receptor type 1 and type 2 in resistance arteries from nasal lesions in granulomatosis with polyangiitis (Wegener's granulomatosis). in Scandinavian journal of rheumatology 2011
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Human Monoclonal AGT Primary Antibody für WB - ABIN1882204
Desong Liu, Fang Lu, Songhui Zhai, Liu Wei, Shi Ma, Xiuying Chen, Liqun Dong, Yannan Guo, Jin Wu, Zheng Wang: Renin-angiotensin system gene polymorphisms in children with Henoch-Schönlein purpura in West China. in Journal of the renin-angiotensin-aldosterone system : JRAAS 2010
Show all 3 Pubmed References
Human Monoclonal AGT Primary Antibody für WB - ABIN1882205
Kieć-Wilk, Olszanecka, Mikołajczyk, Kawecka-Jaszcz et al.: [Role of the M235T (c.704c>T) polymorphism of angiotensynogen gene as well as A724A (c.2171G>A) polymorphism of SERCA2a gene in ethiopathogenesis of left ventricular hypertrophy in essential... in Przegla̧d lekarski 2010
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Human Monoclonal AGT Primary Antibody für ELISA, WB - ABIN968948
Xu, Carretero, Lin, Cavasin, Shesely, Yang, Reudelhuber, Yang: Role of cardiac overexpression of ANG II in the regulation of cardiac function and remodeling postmyocardial infarction. in American journal of physiology. Heart and circulatory physiology 2007
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Human Monoclonal AGT Primary Antibody für ELISA, WB - ABIN559812
Jain, Li, Patil, Kumar: HNF-1alpha plays an important role in IL-6-induced expression of the human angiotensinogen gene. in American journal of physiology. Cell physiology 2007
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Mouse (Murine) Polyclonal AGT Primary Antibody für WB - ABIN4886440
Lan, Yue, Han, Shi, Yang, Pu, Yao, Kang: Genome-wide identification of TCF7L2/TCF4 target miRNAs reveals a role for miR-21 in Wnt-driven epithelial cancer. in International journal of oncology 2011
Human Polyclonal AGT Primary Antibody für IHC, IHC (p) - ABIN4352882
Bachmann, Burté, Pramana, Conte, Brown, Orimadegun, Ajetunmobi, Afolabi, Akinkunmi, Omokhodion, Akinbami, Shokunbi, Kampf, Pawitan, Uhlén, Sodeinde, Schwenk, Wahlgren, Fernandez-Reyes, Nilsson: Affinity proteomics reveals elevated muscle proteins in plasma of children with cerebral malaria. in PLoS pathogens 2014
Unfavorable genotype of polymorphic variants of candidate gene participating in endothelial dysfunction AGT (zeige AGXT Antikörper) (Thrl74Met and Met23SThr) was associated with changes in levels of their active substances in individuals exposed to mercury.
These findings reveal the critical role of hypoxia in producing local angiotensin II by a lactate-chymase (zeige CMA1 Antikörper)-dependent mechanism and highlight the importance of local angiotensin II in regulating radioresistance of hypoxic tumor cells.
The reduced urinary AGT (zeige AGXT Antikörper)/creatinine in Autralian Indigenous pregnant women may reflect subclinical renal dysfunction which limits the ability of the kidney to maintain sodium balance and could indicate an increased risk of pregnancy complications and/or future renal disease.
ACE2 (zeige ACE2 Antikörper) and other enzymes can form ANG-(1 (zeige ANGPT1 Antikörper)-7) directly or indirectly from either the decapeptide ANG I or from ANG II. [review]
The ACE (zeige ACE Antikörper) and AGT (zeige AGXT Antikörper) gene polymorphisms are not associated with the progress of diabetes developing into retinopathy in Chinese patients with type 2 diabetes.
AGT (zeige AGXT Antikörper) M235T and T174M variants contribute to an increased risk of developing preeclampsia (PE), and for M235T to PE severity.
Data, including data using network analysis, suggest that angiotensinogen (AGT), mitogen-activated protein kinase-14 (MAPK14 (zeige MAPK14 Antikörper)), and prothrombin (zeige F2 Antikörper) (F2) in placental villous tissues are core factors in early embryonic development; these studies involved proteomics and bioinformatics analysis of altered protein expression in placental villous tissue from early recurrent miscarriage patients in comparison to control tissues.
The AGT (zeige AGXT Antikörper) (M235T) gene polymorphism do not seem to have a significant effect on the development of clinical properties or cardiovascular comordities of acromegalic patients.
Angiotensin II has a role in increasing glomerular permeability by beta-arrestin mediated nephrin (zeige NPHS1 Antikörper) endocytosis
After donor nephrectomy, increasing uAGT (zeige DPAGT1 Antikörper) levels can be the result of activation of the intrarenal renin (zeige REN Antikörper)-angiotensin system affecting the compensatory changes in the remaining kidney.
ANG II is up-regulated in serum and heart tissues of mice with EAM and that ANG II significantly drives monocyte/macrophage infiltration through the C-C chemokine receptor 2/5 (CCR2/5) axis.
results established that A20 (zeige TNFAIP3 Antikörper) is involved in the renoprotective effect by calcitriol via negatively modulating the NF-kappaB (zeige NFKB1 Antikörper) pathway and necroptotic pathway in AngII-induced renal injury.
NLRP3 (zeige NLRP3 Antikörper) gene deletion attenuates Ang II-induced NLRP3 (zeige NLRP3 Antikörper) inflammasome activation, phenotypic transformation from a contractile phenotype to a synthetic phenotype and proliferation in primary mice Vascular Smooth Muscle Cells.
adipocyte-derived Agt (zeige AGXT Antikörper) has essentially no contribution to the plasma concentration and no impact on blood pressure compared to liver-derived Agt (zeige AGXT Antikörper).
Lung ischemia-reperfusion injury causes a dysregulation of circulating Ang 2 (zeige ANGPT2 Antikörper) levels and plasma PREP (zeige PREP Antikörper) activity, although no direct link between both phenomena could be shown.
Inhibition of TLR4 (zeige TLR4 Antikörper) ameliorates AngII-impaired cavernosal relaxation, decreases TNF-alpha (zeige TNF Antikörper) levels, and restores Nitric Oxide bioavailability, demonstrating that TLR4 (zeige TLR4 Antikörper) partly mediates AngII-induced cavernosal dysfunction.
Our study is the first to show the important role of IL-6 (zeige IL6 Antikörper) in regulating cardiac pathogenesis via inflammation and apoptosis during AngII-induced hypertension. We also provide a novel link between IL-6 (zeige IL6 Antikörper)/STAT3 (zeige STAT3 Antikörper) and EndoG (zeige ENDOG Antikörper)/MEF2A (zeige MEF2A Antikörper) pathway that affects cardiac hypertrophy during AngII stimulation.
this study demonstrated that Ang II could increase TRPC6 (zeige TRPC6 Antikörper) induced Ca(2 (zeige CA2 Antikörper)+) influx and enhance autophagy through increasing reactive oxygen species levels in podocytes, and autophagy could protect Ang II-treated podocytes.
These results implied that AngII could effectively induce EpiCs to differentiate into vascular smooth muscle-like cells through the AT1 receptor (zeige AGTRAP Antikörper).
Results suggest the involvement of angiotensin II (Ang II), through its angiotensin type-1 receptor (AT1R (zeige AGTRAP Antikörper)) in the inflammation induced by Aah (zeige ASPH Antikörper) venom, in the heart and the aorta.
In conclusion, endothelial vWF (zeige VWF Antikörper) knockdown prevented angiotensin II-induced ET-1 (zeige EDN1 Antikörper) upregulation through attenuation of NOX-mediated O2- production.
Data suggest that intra-adrenal metabolism of Ang II to Ang III is required for zona glomerulosa cell-mediated relaxation of adrenal arterioles but not for aldosterone secretion.
NADPH oxidase (zeige NOX1 Antikörper) plays an important role in proMMP-2 expression and activation and MMP-2 (zeige MMP2 Antikörper) mediated SMC (zeige DYM Antikörper) proliferation occurs through the involvement of Spm (zeige NPC1 Antikörper)-Cer (zeige CBLN1 Antikörper)-S1P (zeige MBTPS1 Antikörper) signaling axis under ANG II stimulation of PASMCs
The metabolism of angiotensin II (Ang II) to angiotensin III (Ang III) and its role in the vasorelaxation response in adrenal arteries are reported.
The study identified the serine phosphorylation (p-Ser (zeige SIGLEC1 Antikörper)) sites induced by PKC-Beta (zeige PRKCB Antikörper) activation or AGT, which inhibits insulin (zeige INS Antikörper)-induced p-Tyr (zeige TYR Antikörper) sites on IRS2 (zeige IRS2 Antikörper) and its signals in endothelial cells.
Data suggest up-regulation of AGT in granulosa cells and of Ang II in follicular fluid during preovulatory period; Ang II appears to amplify stimulatory effects of luteinizing hormone on secretion of progesterone/prostaglandins by granulosa cells.
Data suggest that angiotensin II promotes uptake/accumulation of iron (non-transferrin (zeige Tf Antikörper) bound iron) into vascular endothelial cells; such iron accumulation appears to depend on activation of angiotensin type 1 receptor and promotes oxidative stress.
a critical role for H(2)O(2) in angiotensin-II signaling to the endothelial cytoskeleton in a novel pathway that is critically dependent on MARCKS, Rac1, and c-Abl.
The objective of this study was to characterize the profiles of Ang-(1-7), MAS receptor, ACE(2), NEP and PEP during the ovulatory process in cattle.
Fetal adrenal cells in primary culture respond to angiotensin-II by increasing aldosterone production and aldosterone synthase (zeige CYP11B2 Antikörper) [P450c18/CYP11B2 (zeige CYP11B2 Antikörper)] activity.
ANG II inhibits bTREK-1 K(+) channels by a Ca(2+)-dependent mechanism that does not require the depletion of membrane-associated PIP(2).
The protein encoded by this gene, pre-angiotensinogen or angiotensinogen precursor, is expressed in the liver and is cleaved by the enzyme renin in response to lowered blood pressure. The resulting product, angiotensin I, is then cleaved by angiotensin converting enzyme (ACE) to generate the physiologically active enzyme angiotensin II. The protein is involved in maintaining blood pressure and in the pathogenesis of essential hypertension and preeclampsia. Mutations in this gene are associated with susceptibility to essential hypertension, and can cause renal tubular dysgenesis, a severe disorder of renal tubular development. Defects in this gene have also been associated with non-familial structural atrial fibrillation, and inflammatory bowel disease.
alpha-1 antiproteinase, antitrypsin
, angiotensin I
, angiotensin II
, serine (or cysteine) proteinase inhibitor
, serpin A8
, angiotensin ll
, angiotensinogen (PAT)
, zC8A9.1 (angiotensinogen )
, Serpin A8