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anti-Human TWIST2 Antikörper:
anti-Mouse (Murine) TWIST2 Antikörper:
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Human Monoclonal TWIST2 Primary Antibody für IHC (p), ELISA - ABIN566756
Murakami, Ohkuma, Nakamura: Molecular mechanism of transforming growth factor-beta-mediated inhibition of growth arrest and differentiation in a myoblast cell line. in Development, growth & differentiation 2008
Show all 8 Pubmed References
Human Polyclonal TWIST2 Primary Antibody für IF, WB - ABIN530431
Wade, Brinas, Welfare, Wicking, Farlie: Twist2 contributes to termination of limb bud outgrowth and patterning through direct regulation of Grem1. in Developmental biology 2012
epithelial membrane protein 3 (zeige EMP3 Antikörper) is a downstream effector of TWIST1 (zeige TWIST1 Antikörper)/2 in inducing epithelial-to-mesenchymal transition in gastric cancer. Epithelial membrane protein 3 (zeige EMP3 Antikörper) upregulation might be associated with gastric cancer metastasis and is a potential indicator of unfavorable overall survival and progression-free survival in gastric cancer patients
Findings indicated that HIF-2alpha (zeige EPAS1 Antikörper) promotes epithelial-mesenchymal transition in pancreatic cancer by regulating Twist2 binding to the promoter of E-cadherin (zeige CDH1 Antikörper).
A homozygous missense mutation in the TWIST2 gene was described in 3 siblings affected by Setleis syndrome. An alteration of bHLH domain, and loss of transcription factor's function was predicted due to protein substitution.
TWIST2 regulates epithelial-mesenchymal transition by depriving the epithelial cell phenotype of E-cadherin (zeige CDH1 Antikörper) and endowing the mesenchymal cell phenotype with Vimentin (zeige VIM Antikörper), which may be involved in the progression and prognosis of ovarian cancer.
MACC1 (zeige MACC1 Antikörper) promotes vasculogenic mimicry in gastric cancer by regulating the HGF/c-Met-TWIST1 (zeige TWIST1 Antikörper)/2 signaling pathway.
In patients with no duplication/triplication of the 1p36.22p36.21 region and no mutations in TWIST2, there are mutation(s) in one of the 30 genes in this region or mutations in other as yet unidentified genes at different locations
Data suggest that TWIST1 (zeige TWIST1 Antikörper) and to a lesser degree TWIST2 expressed within the tumor stroma could contribute to the epithelial-mesenchymal transition (EMT (zeige ITK Antikörper))-like tumor budding phenotype in colorectal cancers.
A substituted lysine at TWIST2 residue 75 results in Ablepharon Macrostomia Syndrome, whereas a glutamine (zeige GFPT1 Antikörper) or alanine yields Barber-Say Syndrome.
After chronic NOD2 (zeige NOD2 Antikörper) stimulation, Twist1 (zeige TWIST1 Antikörper) and Twist2 coordinate the regulation of both transcriptional activators and repressors, thereby mediating optimal cytokine down-regulation.
Results show that Twist2 expression was gradually increased during the progression from normal cervical squamous epithelium to cervical intraepithelial neoplasia (CIN (zeige PDXP Antikörper)) and cervical squamous cell carcinoma.
Twist2 partially retains histone deacetylase 7 (HDAC7 (zeige HDAC7 Antikörper)) in the nucleus and recruits it to the Nur77 (zeige NR4A1 Antikörper) promoter region to repress Nur77 (zeige NR4A1 Antikörper) in positively selected thymocytes.
The Twist2-Cre, Osterix (zeige SP7 Antikörper)-Cre and osteocalcin (zeige BGLAP Antikörper)-Cre lines to generate conditional beta1 integrin deletions, were used to investigate the role of beta1 integrins on skeletal phenotype.
Twist-2 was involved in endotoxin tolerance through inhibiting NF-kappaB (zeige NFKB1 Antikörper) transactivation and cytokines transcriptional activities.
Twist2 functions as a tumor suppressor in osteosarcoma cells.
we identify Twist2 as a key regulator of survival, invasion and anchorage-independent growth in the aneuploid cells
Wnt (zeige WNT2 Antikörper) signaling/beta-catenin (zeige CTNNB1 Antikörper) is absolutely required and sufficient for Dermo1 expression and dermal cell identity in the cranium.
Twist2 is required for normal corneal keratocyte proliferation and eyelid morphogenesis in the mouse.
TWIST2 recessive mutations cause an focal facial dermal dysplasias and dominant TWIST1 (zeige TWIST1 Antikörper) mutations cause Saethre-Chotzen craniocynostosis suggests that they function independently in skin and bone development.
Using the N-terminal domain of ADD1/SREBP1c (zeige SREBF1 Antikörper) as bait, we identified Twist2 (also known as Dermo-1), a basic helix-loop-helix (bHLH) protein, as a novel ADD1/SREBP1c (zeige SREBF1 Antikörper) interacting protein
Twist1 (zeige TWIST1 Antikörper) and Twist2 factors are cross-regulated and inhibit bone specific gene expression and other helix-loop-helix proteins
copy number variant in the 5'-flanking region affects melanocyte development and pigmentation in belted cattle
Basic helix-loop-helix (bHLH) transcription factors have been implicated in cell lineage determination and differentiation. The protein encoded by this gene is a bHLH transcription factor and shares similarity with another bHLH transcription factor, Twist. It is thought that during osteoblast development this protein may inhibit osteoblast maturation and maintain cells in a preosteoblast phenotype. Two transcript variants encoding the same protein have been found for this gene.
twist homolog 2 (Drosophila)
, twist-related protein 2
, class A basic helix-loop-helix protein 39
, dermis-expressed protein 1
, twist homolog 2
, twist-related bHLH protein Dermo1
, dermis expressed 1
, dermo-1 protein