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anti-Human COX2 Antikörper:
anti-Mouse (Murine) COX2 Antikörper:
anti-Rat (Rattus) COX2 Antikörper:
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Human Monoclonal COX2 Primary Antibody für WB - ABIN283213
Cheng, Whitehead, Hachinski, Cechetto: Effects of pyrrolidine dithiocarbamate on beta-amyloid (25-35)-induced inflammatory responses and memory deficits in the rat. in Neurobiology of disease 2006
activation of ERK1/2 signaling was required for hCG-induced up-regulation of SPRY2 expression. Further, SPRY2 knockdown attenuated the AREG-induced COX-2 expression and PGE2 production by inhibiting AREG-activated ERK1/2 signaling.
COX-2 was elevated in glioma tissues and its expression was negatively correlated with the levels of miR (zeige MLXIP Antikörper)-128. These findings may establish miR (zeige MLXIP Antikörper)-128 as a new potential target for the treatment of patients with gliomas.
Post-transcriptional regulation of COX-2 mRNAs translation by SGs (zeige FBN1 Antikörper) indicates a role in IL-1beta (zeige IL1B Antikörper)-mediated catabolic response that could be therapeutically targeted in Osteoarthritis.
Results show that in influenza A viruses (IAV)-infected cells, COX-2 expression is regulated. While the protein is induced at early time of infection, via recognition of IAV vRNA by RIG-I (zeige DDX58 Antikörper), COX-2 expression is reduced again during on-going replication due to destabilization of its mRNA by IAV-induced TTP (zeige ADAMTS13 Antikörper).
Our results highlight the role of COX-2 in constitutive IDO1 (zeige IDO1 Antikörper) expression by human tumors and substantiate the use of COX-2 inhibitors to improve the efficacy of cancer immunotherapy, by reducing constitutive IDO1 (zeige IDO1 Antikörper) expression, which contributes to the lack of T-cell infiltration in "cold" tumors, which fail to respond to immunotherapy
Chinese population with GG genotype of COX-2 gene polymorphism rs689466 have higher risk to develope post-traumatic osteomyelitis.
Extracellular histones disarrange vasoactive mediators release through a COX1 (zeige COX1 Antikörper)-COX2-eNOS (zeige NOS3 Antikörper) interaction in human endothelial cells.
4-Hydroxy-2-nonenal is a natural inducer of COX-2 in atherosclerosis. (Review)
RhoA (zeige RHOA Antikörper) and COX-2 were upregulated in early gastric cancer tissues, which facilitated the proliferation and migration of gastric cancer cells.
LXR (zeige NR1H3 Antikörper) gene expression was significantly increased in obese children with obstructive sleep apnea-hypopnea syndrome (OSAHS). The severity of OSAHS was positively correlated with COX-2 levels.
These data indicate the functional role of the MIF (zeige MIF Antikörper)-COX (zeige CPOX Antikörper)-p53 (zeige TP53 Antikörper) axis in inflammation and cancer at the genomic and proteomic levels in COX-2-ablated cells.
PTGS2 (zeige PTGS2 Antikörper) deletion changes the natural distribution of ANXA2 (zeige ANXA2 Antikörper) in monocytes/macrophages, increasing TF expression and activity predisposing to venous thrombosis.
Study suggest that amyloid beta-protein increase the expression of TRPC6 (zeige TRPC6 Antikörper) via NF-kappaB (zeige NFKB1 Antikörper) in BV-2 microglia and promotes the production of COX-2, which induces hippocampus neuron damage.
Data show that patients with high cyclooxygenase-2 (COX2 (zeige PTGS2 Antikörper)) gene expression who received celecoxib had a significantly higherpathological complete response (pCR) rate compared with patients with low COX2 gene expression.
Topical application of glycolic acid suppresses the UVB induced IL-6 (zeige IL6 Antikörper), IL-8 (zeige IL8 Antikörper), MCP-1 (zeige CPT1B Antikörper) and COX-2 inflammation by modulating NF-kappaB (zeige NFKB1 Antikörper) signaling pathway in mouse skin.
COX-2/mPGES-1 (zeige PTGES Antikörper)/PGE2 cascade activation mediates uric acid-induced glomerular mesangial cell proliferation.
Cobalt protoporphyrin induces COX-2 expression through activating P2X7 (zeige P2RX7 Antikörper) receptors and ASK1 (zeige MAP3K5 Antikörper)/MAP kinases as well as PIAS1 (zeige PIAS1 Antikörper) degradation signaling pathways.
sUV activated the transcription factors nuclear factor-kappaB and activator protein-1 (zeige JUN Antikörper) which, in turn, induces COX-2 expression.
Results indicate that Cox-2 promotes Col10a1 (zeige COL10A1 Antikörper) expression and chondrocyte hypertrophy in vitro.
These data reveal important structure-function and signaling differences between the two FFA4 isoforms, and for the first time link FFA4 to modulation of ROS (zeige ROS1 Antikörper) in macrophages.
B1 receptors are coupled to COX2 (zeige PTGS2 Antikörper) in causing endothelium-independent contractions in endotoxin-treated pig coronary arteries
Expression of COX-2 (zeige PTGS2 Antikörper) was strongest in gilts with acute endometritis, but did not differ between those with chronic endometritis and normal endometrium.
Data indicate that the proton-pumping pathway of heart cytochrome c (zeige CYCS Antikörper) oxidase includes a hydrogen-bond network and a water channel (zeige AQP4 Antikörper) located in tandem between the positive and negative side of the mitochondrial membrane.
Study suggests that His503 is involved in the proton supply to the D-path as a proton acceptor in cytochrome c (zeige CYCS Antikörper) oxidase.
Data suggest that the axial Met residue moderately increased the redox potential of the Cu(A) site in cytochrome c (zeige CYCS Antikörper) oxidase.
Studies indicate that heart cytochrome c (zeige CYCS Antikörper) oxidase subunits I, II and III are encoded by the mitochondrial genome.
extensive pre- and intrapartal rise of COX (zeige COX7A1 Antikörper)-II expression in bovine placentomes with a 70-100-fold increase of COX (zeige COX7A1 Antikörper)-II-mRNA levels
Pretreatment of cells with selective COX-2 blocker etodolac markedly inhibited ICl.
Safflower injection may attenuate lung ischemia/reperfusion injury through inhibiting cyclooxygenase-2 (zeige PTGS2 Antikörper) expression.
The RNA interference targeting COX-2 can effectively inhibit the expression of COX-2 and MMP-2 (zeige MMP2 Antikörper) in IL-1alpha stimulated rabbit corneal stromal cells in vitro.
Prostaglandin-endoperoxide synthase (PTGS), also known as cyclooxygenase, is the key enzyme in prostaglandin biosynthesis, and acts both as a dioxygenase and as a peroxidase. There are two isozymes of PTGS: a constitutive PTGS1 and an inducible PTGS2, which differ in their regulation of expression and tissue distribution. This gene encodes the inducible isozyme. It is regulated by specific stimulatory events, suggesting that it is responsible for the prostanoid biosynthesis involved in inflammation and mitogenesis.
PGH synthase 2
, PHS II
, cyclooxygenase 2b
, prostaglandin G/H synthase 2
, prostaglandin H2 synthase 2
, cyclooxygenase 2
, glucocorticoid-regulated inflammatory cyclooxygenase
, macrophage activation-associated marker protein P71/73
, cytochrome c oxidase subunit II
, cytochrome c oxidase subunit ii
, cytochrome C oxidase subunit II