CARD17 Antikörper (AA 1-100) (FITC)

Produktnummer ABIN886432

Dieses Kaninchen Polyklonal-Antikörper erkennt spezifisch CARD17 in WB, IF (cc) und IF (p). Er zeigt eine Reaktivität gegenüber Human.

Kurzübersicht für CARD17 Antikörper (AA 1-100) (FITC) (ABIN886432)

Target: CARD17 (Caspase Recruitment Domain Family, Member 17 (CARD17))

Reaktivität: Human

Wirt: Kaninchen

Klonalität: Polyklonal

Konjugat: Dieser CARD17 Antikörper ist konjugiert mit FITC

Applikation: Western Blotting (WB), Immunofluorescence (Cultured Cells) (IF (cc)), Immunofluorescence (Paraffin-embedded Sections) (IF (p))

Produktdetails anti-CARD17 Antikörper (FITC)

Bindungsspezifität: AA 1-100

Homologie: Human

Aufreinigung: Purified by Protein A.

Immunogen: KLH conjugated synthetic peptide derived from human CARD17/INCA

Isotyp: IgG

Anwendungsinformationen

Applikationshinweise: IF(IHC-P) 1:50-200
IF(IHC-F) 1:50-200
IF(ICC) 1:50-200

Beschränkungen: Nur für Forschungszwecke einsetzbar

Handhabung

Format: Liquid

Konzentration: 1 μg/μL

Buffer: Aqueous buffered solution containing 0.01M TBS ( pH 7.4) with 1 % BSA, 0.03 % Proclin300 and 50 % Glycerol.

Konservierungsmittel: ProClin

Vorsichtsmaßnahmen: This product contains ProClin: a POISONOUS AND HAZARDOUS SUBSTANCE, which should be handled by trained staff only.

Lagerung: -20 °C

Informationen zur Lagerung: Store at -20°C. Aliquot into multiple vials to avoid repeated freeze-thaw cycles.

Haltbarkeit: 12 months

Details zu CARD17

Target: CARD17 (Caspase Recruitment Domain Family, Member 17 (CARD17))

Andere Bezeichnung: CARD17

Hintergrund:

Synonyms: CAR17_HUMAN, CARD 17, CARD17, Caspase 1 inhibitor, Caspase recruitment domain containing protein 17, Caspase recruitment domain-containing protein 17, Caspase-1 inhibitor INCA, Caspase1 inhibitor, Inhibitory caspase recruitment domain CARD protein, Inhibitory caspase recruitment domain protein.

Background: Regulator of procaspase-1/CASP1 activation implicated in the regulation of the proteolytic maturation of pro-IL-1beta/IL1B and its release during inflammation. Inhibits the release of IL1B in response to LPS in monocytes. However, unlike CASP1, do not induce NF-kappa-B activation.