Methamphetamine Antikörper

Produktnummer ABIN723186

This anti-Methamphetamine antibody (ABIN723186) is a Mouse Monoclonal antibody detecting Methamphetamine in ELISA. Suitable for .

Kurzübersicht für Methamphetamine Antikörper (ABIN723186)

Target: Methamphetamine (M-Amp)

Reaktivität: Bitte anfragen

Wirt: Maus

Klonalität: Monoklonal

Konjugat: Dieser Methamphetamine Antikörper ist unkonjugiert

Applikation: ELISA

Klon: 4D2

Produktdetails anti-Methamphetamine Antikörper

Kreuzreaktivität (Details): Methamphetamine

Aufreinigung: Purified by Protein G.

Immunogen: KLH conjugated Methamphetamine

Isotyp: IgG

Anwendungsinformationen

Beschränkungen: Nur für Forschungszwecke einsetzbar

Handhabung

Format: Liquid

Konzentration: 1 μg/μL

Buffer: 0.01M TBS( pH 7.4) with 1 % BSA, 0.02 % Proclin300 and 50 % Glycerol.

Konservierungsmittel: Sodium azide

Vorsichtsmaßnahmen: This product contains sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.

Lagerung: 4 °C,-20 °C

Informationen zur Lagerung: Shipped at 4°C. Store at -20°C for one year. Avoid repeated freeze/thaw cycles.

Haltbarkeit: 12 months

Details zu Methamphetamine

Target: Methamphetamine (M-Amp)

Andere Bezeichnung: Methamphetamine

Substanzklasse: Chemical

Hintergrund:

Synonyms: d-Desoxyephedrine hydrochloride, d-N, -Dimethylphenethylamine hydrochloride, Methylamphetamine hydrochloride, METH.

Background: Methamphetamine (METH) is closely related chemically to amphetamine (AMPH). METH is a potent central nervous system stimulant with additional peripheral sympathomimetic effects. METH and AMPH have been used clinically in the treatment of obesity, minimal brain dysfunction, narcolepsy, depression and to counter fatigue. They are also subjected to widespread abuse. METH is an indirect agonists. It causes the release of newly synthesized norepinephrine and dopamine and it blocks the re uptake of these transmitters from the synapse. This can lead to an increase in the concentration of catecholamines in the synapse as well as an overall increase in catecholaminergic activity in the brain. The mechanism of METH induced neurotoxicity for all monoaminergic cell types may lie primarily with the dopaminergic system in the striatum. It may also lie with the interaction between METH induced release of dopamine and its ability to inhibit monoamine oxidase.