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Septin 5 Antikörper

Dieses Kaninchen Polyklonal-Antikörper erkennt spezifisch Septin 5 in IHC und IF. Er zeigt eine Reaktivität gegenüber Human, Ratte und Maus.
Produktnummer ABIN7256500

Kurzübersicht für Septin 5 Antikörper (ABIN7256500)

Target

Alle Septin 5 (SEPT5) Antikörper anzeigen
Septin 5 (SEPT5)

Reaktivität

  • 52
  • 26
  • 8
  • 2
Human, Ratte, Maus

Wirt

  • 43
  • 9
Kaninchen

Klonalität

  • 43
  • 8
  • 1
Polyklonal

Konjugat

  • 29
  • 4
  • 4
  • 2
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
Dieser Septin 5 Antikörper ist unkonjugiert

Applikation

  • 50
  • 25
  • 24
  • 13
  • 13
  • 7
  • 7
  • 6
  • 5
  • 3
  • 3
  • 1
  • 1
  • 1
Immunohistochemistry (IHC), Immunofluorescence (IF)
  • Produktmerkmale

    Polyclonal Antibody

    Aufreinigung

    Affinity purification

    Immunogen

    Recombinant fusion protein of human 44079 (NP_002679.2).

    Isotyp

    IgG
  • Applikationshinweise

    IHC 1:100-1:200 IF 1:50-1:200

    Beschränkungen

    Nur für Forschungszwecke einsetzbar
  • Format

    Liquid

    Konzentration

    1 mg/mL

    Buffer

    PBS with 0.02 % sodium azide, 50 % glycerol, pH 7.3

    Konservierungsmittel

    Sodium azide

    Vorsichtsmaßnahmen

    This product contains Sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.

    Lagerung

    -20 °C

    Informationen zur Lagerung

    Store at -20°C. Avoid freeze / thaw cycles.
  • Target

    Septin 5 (SEPT5)

    Andere Bezeichnung

    SEPT5

    Hintergrund

    This gene is a member of the septin gene family of nucleotide binding proteins, originally described in yeast as cell division cycle regulatory proteins. Septins are highly conserved in yeast, Drosophila, and mouse and appear to regulate cytoskeletal organization. Disruption of septin function disturbs cytokinesis and results in large multinucleate or polyploid cells. This gene is mapped to 22q11, the region frequently deleted in DiGeorge and velocardiofacial syndromes. A translocation involving the MLL gene and this gene has also been reported in patients with acute myeloid leukemia. Alternative splicing results in multiple transcript variants. The presence of a non-consensus polyA signal (AACAAT) in this gene also results in read-through transcription into the downstream neighboring gene (GP1BB, platelet glycoprotein Ib), whereby larger, non-coding transcripts are produced.

    Gen-ID

    5413

    UniProt

    Q99719

    Pathways

    Synaptic Vesicle Exocytosis
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