Influenza A Virus H1N1 Antikörper

Produktnummer ABIN643615

Dieser Ziege not_set Antikörper detektiert spezifisch Influenza A Virus H1N1 in IF, IHC (p) und H. Es zeigt Reaktivität gegenüber Proben von Influenza A Virus H1N1.

Kurzübersicht für Influenza A Virus H1N1 Antikörper (ABIN643615)

Target: Influenza A Virus H1N1

Reaktivität: Influenza A Virus H1N1

Wirt: Ziege

Konjugat: Unkonjugiert

Applikation: Immunofluorescence (IF), Immunohistochemistry (Paraffin-embedded Sections) (IHC (p)), Haemagglutination (H)

Produktdetails

Spezifität: DS-PB-01152 is specific for Influenza A virus H1N1 glycoprotein and does not react with Influenza B, RSV, Para 1-3 or Adenovirus. It does not react with HEp-2 cells but may react with some chicken cellular proteins.

Produktmerkmale: Goat Anti-Influenza A H1N1

Aufreinigung: Purified

Immunogen: Influenza A, strain USSR (H1N1)

Isotyp: IgG

Anwendungsinformationen

Beschränkungen: Nur für Forschungszwecke einsetzbar

Handhabung

Format: Liquid

Konzentration: IgG concentration 1.0 mg/ml

Buffer: Phosphate buffered saline pH 7.2

Konservierungsmittel: Sodium azide

Vorsichtsmaßnahmen: This product contains sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.

Lagerung: 4 °C

Antigendetails

Target: Influenza A Virus H1N1

Andere Bezeichnung: Influenza A H1N1

Substanzklasse: Influenza Virus

Hintergrund: Influenza type A viruses are divided into subtypes based on the antigenic differences of two viral surface proteins, hemagglutinin (H) and neuraminidase (N). On infection of the respiratory tract, the hemagglutinin molecule binds to sialic acid-containing receptors on the epithelial cells resulting in endocytosis. Once the virus has been engulfed, the hemagglutinin allows the viral membrane to fuse with the endosomal membrane. Neuraminidase functions to aid viral release from host cells by cleaving terminal sialic acid residues from carbohydrate moieties on the cell surface. Subtype antigenic variations result from a process known as antigenic drift whereby these surface proteins constantly mutate in order to evade the host immune response. Subtype A (H1N1) was the cause of Spanish flu that killed approximately 500,000 people between 1918-1919.