Methamphetamine Antikörper

Produktnummer ABIN318601

Dieser Schaf Polyklonal Antikörper detektiert spezifisch Methamphetamine in EIA.

Kurzübersicht für Methamphetamine Antikörper (ABIN318601)

Target: Methamphetamine (M-Amp)

Wirt: Schaf

Klonalität: Polyklonal

Konjugat: Dieser Methamphetamine Antikörper ist unkonjugiert

Applikation: Enzyme Immunoassay (EIA)

Produktdetails anti-Methamphetamine Antikörper

Aufreinigung: Purified IgG prepared by affinity chromatography on Protein G

Immunogen: Para-amino-metamphetamine conjugated to keyhole limpet hemocyanin

Isotyp: IgG

Anwendungsinformationen

Applikationshinweise: ELISA.
Other applications not tested.
Optimal dilutions are dependent on conditions and should be determined by the user.

Beschränkungen: Nur für Forschungszwecke einsetzbar

Handhabung

Format: Liquid

Konzentration: 5.0 mg/mL

Buffer: Phosphate buffered saline containing 0.09 % Sodium Azide (NaN3)

Konservierungsmittel: Sodium azide

Vorsichtsmaßnahmen: This product contains Sodium Azide: a POISONOUS AND HAZARDOUS SUBSTANCE, which should be handled by trained staff only.

Handhabung: Avoid repeated freezing and thawing.

Lagerung: 4 °C/-20 °C

Informationen zur Lagerung: Store the antibody undiluted at 2-8 °C for one month or (in aliquots) at -20 °C for longer.

Details zu Methamphetamine

Target: Methamphetamine (M-Amp)

Andere Bezeichnung: Methamphetamine

Substanzklasse: Chemical

Hintergrund: Methamphetamine (METH) is closely related chemically to amphetamine (AMPH). METH is a potent central nervous system stimulant with additional peripheral sympathomimetic effects. METH and AMPH have been used clinically in the treatment of obesity, minimal brain dysfunction, narcolepsy, depression and to counter fatigue. They are also subjected to widespread abuse. METH is an indirect agonists. It causes the release of newly synthesized norepinephrine and dopamine and it blocks the re uptake of these transmitters from the synapse. This can lead to an increase in the concentration of catecholamines in the synapse as well as an overall increase in catecholaminergic activity in the brain. The mechanism of METH induced neurotoxicity for all monoaminergic cell types may lie primarily with the dopaminergic system in the striatum. It may also lie with the interaction between METH induced release of dopamine and its ability to inhibit monoamine oxidase.