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CXCL9 Antikörper

Dieses Armenischer Hamster Monoklonal-Antikörper erkennt spezifisch CXCL9 in FACS. Er zeigt eine Reaktivität gegenüber Maus.
Produktnummer ABIN2664906

Kurzübersicht für CXCL9 Antikörper (ABIN2664906)

Target

Alle CXCL9 Antikörper anzeigen
CXCL9 (gamma-Interferon-Induced Monokine (CXCL9))

Reaktivität

  • 94
  • 27
  • 21
  • 16
  • 3
  • 2
  • 1
Maus

Wirt

  • 66
  • 38
  • 2
  • 2
  • 1
  • 1
Armenischer Hamster

Klonalität

  • 71
  • 39
Monoklonal

Konjugat

  • 59
  • 12
  • 9
  • 4
  • 4
  • 4
  • 2
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
Dieser CXCL9 Antikörper ist unkonjugiert

Applikation

  • 84
  • 43
  • 40
  • 27
  • 22
  • 13
  • 13
  • 9
  • 8
  • 7
  • 3
  • 3
  • 3
  • 3
  • 3
  • 2
  • 2
  • 1
Flow Cytometry (FACS)

Klon

MIG-2F5-5
  • Aufreinigung

    The antibody was purified by affinity chromatography.

    Isotyp

    IgG, IgG kappa
  • Applikationshinweise

    Optimal working dilution should be determined by the investigator.

    Beschränkungen

    Nur für Forschungszwecke einsetzbar
  • Konzentration

    0.5 mg/mL

    Buffer

    Phosphate-buffered solution, pH 7.2, containing 0.09 % sodium azide.

    Konservierungsmittel

    Sodium azide

    Vorsichtsmaßnahmen

    This product contains Sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.

    Lagerung

    4 °C

    Informationen zur Lagerung

    The antibody solution should be stored undiluted between 2°C and 8°C.
  • Target

    CXCL9 (gamma-Interferon-Induced Monokine (CXCL9))

    Andere Bezeichnung

    CXCL9

    Hintergrund

    MIG, also known as mig-1, CXCL9, is a member of the alpha subfamily of inflammatory chemokine. It is inducible in macrophages, hepatocytes, and endothelial cells by IFN-γ, but not by TNF-α or bacterial lipopolysacchrides (LPS). Mig functions as a chemotactic factor for resting memory and activated T cells, both CD4+ and CD8+, and natural killer cells. Furthermore, it was reported that Mig induced both calcium signals and chemotaxis in activated B cells and that B cell activation induced expression of mouse CXCR3. MIG and CXCR3 may be important not only to recruit T cells to peripheral inflammatory sites, but also in some cases to maximize interactions among activated T cells, B cells, and dendritic cells within lymphoid organs to provide optimal humoral responses to pathogens.
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