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anti-Human Cathepsin S Antikörper:
anti-Mouse (Murine) Cathepsin S Antikörper:
anti-Rat (Rattus) Cathepsin S Antikörper:
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Human Polyclonal Cathepsin S Primary Antibody für IP, IHC - ABIN223091
Zaehringer, Sapoval, Pattynama, Rabbia, Vignali, Maleux, Boyer, Szczerbo-Trojanowska, Jaschke, Hafsahl, Downes, Beregi, Veeger, Stoll, Talen: Sirolimus-eluting versus bare-metal low-profile stent for renal artery treatment (GREAT Trial): angiographic follow-up after 6 months and clinical outcome up to 2 years. in Journal of endovascular therapy : an official journal of the International Society of Endovascular Specialists 2007
Show all 4 Pubmed References
Human Polyclonal Cathepsin S Primary Antibody für ICC, IF - ABIN4288275
Ma, Visser, Roelofsen, de Vries, Diepstra, van Imhoff, van der Wal, Luinge, Alvarez-Llamas, Vos, Poppema, Vonk, van den Berg: Proteomics analysis of Hodgkin lymphoma: identification of new players involved in the cross-talk between HRS cells and infiltrating lymphocytes. in Blood 2008
Show all 2 Pubmed References
Human Polyclonal Cathepsin S Primary Antibody für IHC, IHC (fro) - ABIN4288274
Otsuki, Sawada, Yodoya, Shinohara, Kato, Ohashi, Zhang, Imanaka-Yoshida, Shimpo, Maruyama, Komada, Mitani: Potential contribution of phenotypically modulated smooth muscle cells and related inflammation in the development of experimental obstructive pulmonary vasculopathy in rats. in PLoS ONE 2015
The present data indicating that Cat-S activity increases with CKD progression suggest that Cat-S might be a therapeutic target to prevent cardiovascular complications in CKD.
High expression of CTSS was independently associated with lymph node metastasis (OR, 2.015; 95% CI, 1.225-3.315; P=0.006). Therefore, CTSS may serve as a predictive risk marker for the progression and prognosis of papillary thyroid cancer .
This study therefore considerably improves our understanding of the molecular mechanism responsible for cathepsin S inhibition and facilitates the identification of potential novel selective inhibitors of cathepsin S.
cathepsin S is increased in periodontal cells and tissues under inflammatory and infectious conditions, suggesting a critical role of this autophagy-associated molecule in the pathogenesis of periodontitis.
Elevated cathepsin S activity was associated with collagen I degradation and thus might be involved in the progression of abdominal aortic aneurysms.
Cathepsin S was identified as the major IL-36gamma-activating protease expressed in epithelial cells.
Our results indicate that autophagy is essential for decreasing CTSS activity to inhibit tumor metastasis by hispolon treatment in cervical cancer; this finding provides a new perspective on molecular regulation.
These results revealed that CTSS can regulate EGFR (zeige EGFR Antikörper) signalling by facilitating EGF (zeige EGF Antikörper)-mediated EGFR (zeige EGFR Antikörper) degradation.
This study suggested serum Cat S may be a potential biomarker for the diagnosis and prognosis of gastric cancer
Overall, these results indicate that exploitation of the cathepsin S activity in MPS tissues can be utilized to substantially lower non-target accumulation, suggesting this is a promising approach for the development of diagnostic and radiotherapeutic nanomedicine platforms.
the roles of CTSB (zeige CTSB Antikörper)/S and SIRT1 (zeige SIRT1 Antikörper) in the regulation of hepatic inflammation using primary parenchymal and non-parenchymal hepatic cell types and cell lines.
Cathepsin S activity controls injury-related vascular remodeling via TLR2/p38MAPK (zeige MAPK14 Antikörper)/PI3K/Akt (zeige AKT1 Antikörper)/p-HDAC6 (zeige HDAC6 Antikörper) signaling pathway.
Loss of Rab3D (zeige RAB3D Antikörper) from secretory vesicles, leading to disproportionate Rab27 (zeige RAB27A Antikörper)-to-Rab3D (zeige RAB3D Antikörper) activity, may contribute to the enhanced release of cathepsin S in tears of patients with Sjogren's syndrome.
Ctss induction during muscular dystrophy is a pathologic event that partially underlies disease pathogenesis, and its inhibition might serve as a new therapeutic strategy in Duchenne muscular dystrophy (zeige DMD Antikörper).
Demonstrate the utility of intracellular caspase 1 (zeige CASP1 Antikörper) and extracellular CTSS proteolytic activities as surrogate biomarkers of lysosomal rupture and acute inflammation.
Fluorogen substrate, Mca (zeige RSPH1 Antikörper)-GRWPPMGLPWE-Lys (zeige LYZ Antikörper)(Dnp)-DArg-NH2 can detect CTSS activities in mouse antigen presenting cells.
Data show that cathepsins S (CatS) regulates CCL2 (zeige CCL2 Antikörper) chemokine (zeige CCL1 Antikörper) expression by modulation of CD74 (zeige CD74 Antikörper) antigen processing.
Cathepsin S activates MrgprC11 (zeige MRGPRX1 Antikörper) and evokes receptor-dependent scratching in mice.
cathepsin S deficiency alters the balance between adipocyte and osteoblast differentiation, increases bone turnover, and changes bone microarchitecture. Therefore, bone and fat metabolisms should be monitored when using cathepsin S inhibitors clinically
The cathepsin S deserves further evaluation as therapeutic targets to develop disease modifying drugs to treat Alzheimer's disease.
These results, together with those previously reported for other genes of this family, suggest that cathepsin genes play a role in defining economically important traits in pigs.
The protein encoded by this gene, a member of the peptidase C1 family, is a lysosomal cysteine proteinase that may participate in the degradation of antigenic proteins to peptides for presentation on MHC class II molecules. The encoded protein can function as an elastase over a broad pH range in alveolar macrophages. Alternatively spliced transcript variants encoding distinct isoforms have been found for this gene.
, cathepsin S
, cathepsin S, gene 1
, Cathepsin S