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ZMIZ1 encodes a member of the PIAS (protein inhibitor of activated STAT) family of proteins. Zusätzlich bieten wir Ihnen und viele weitere Produktgruppen zu diesem Protein an.
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Human Polyclonal ZMIZ1 Primary Antibody für IHC (p), WB - ABIN390174
Rakowski, Garagiola, Li, Decker, Caruso, Jones, Kuick, Cierpicki, Maillard, Chiang: Convergence of the ZMIZ1 and NOTCH1 pathways at C-MYC in acute T lymphoblastic leukemias. in Cancer research 2013
Show all 2 Pubmed References
we have identified a molecular phenotype of MS defined by expression of the MS risk gene ZMIZ1 in blood, and by other genes, especially transcription factors. ZMIZ1 expression is affected by, and interacts with, the environmental risk factors EBV and Vitamin D.
rs1250569 (ZMIZ1) and rs10114470 (TL1A) are two novel loci that indicate susceptibility to Inflammatory Bowel Disease in Han-Chinese patients.
This case represents the first constitutional balanced translocation disrupting and fusing both MIZ-type containing and proline-rich 12 and provides clues for the potential function and effects of these in the central nervous system.
At the ZMIZ1 locus, we show that perturbation of ZMIZ1 expression in human islets and beta-cells influences exocytosis and insulin (zeige INS Antikörper) secretion, highlighting a novel role for ZMIZ1 in the maintenance of glucose homeostasis.
Targeting the NOTCH1 (zeige NOTCH1 Antikörper)-ZMIZ1 interaction might combat leukemic growth.
the expression of SENP8 (zeige SENP8 Antikörper), SAE1 (zeige SAE1 Antikörper), PIAS1 (zeige PIAS1 Antikörper), PIAS2 (zeige PIAS2 Antikörper) and ZMIZ1 is deregulated in the majority of PTC (zeige F9 Antikörper) tissues, likely contributing to the PTC (zeige F9 Antikörper) phenotype.
ZMIZ1 is a susceptibility gene for vitiligo (zeige MITF Antikörper) in Chinese population.
ZMIZ1 is overexpressed in a significant percentage of human breast, ovarian, and colon cancers in addition to human squamous cell carcinomas, suggesting that ZMIZ1 may play a broader role in epithelial cancers.
ZMIZ1 and activated NOTCH1 ar (zeige NOTCH1 Antikörper)e coexpressed in a subset of human T-ALL patients and cell lines.
First line of evidence demonstrates a physiological (zeige SMAD3 Antikörper) role for endogenous Zimp10 in regulating Smad3/4-mediated transcription.
Ectopic expression of Zmiz1 induces cutaneous squamous cell malignancies in a mouse model of cancer.
we show that activated NOTCH1 (zeige NOTCH1 Antikörper) and ZMIZ1 collaborate to induce T-ALL in mice.
Zimp10 is found in somatic cells, outside the testis cords and ceases to be expressed in the adult testis.
A physiological role for Zimp10 in regulating Smad3 (zeige SMAD3 Antikörper)/4-mediated transcription is demonstrated in mouse embryonic fibroblasts.
provides evidence to demonstrate a crucial role for Zimp10 in vasculogenesis
This gene encodes a member of the PIAS (protein inhibitor of activated STAT) family of proteins. The encoded protein regulates the activity of various transcription factors, including the androgen receptor, Smad3/4, and p53. The encoded protein may also play a role in sumoylation. A translocation between this locus on chromosome 10 and the protein tyrosine kinase ABL1 locus on chromosome 9 has been associated with acute lymphoblastic leukemia.
zinc finger, MIZ-type containing 1
, zinc finger MIZ domain-containing protein 1-like
, retinoic acid induced 17-like
, FLJ00092 protein
, retinoic acid induced 17
, zinc finger MIZ domain-containing protein 1
, zinc finger-containing, Miz1, PIAS-like protein on chromosome 10
, PIAS-like protein Zimp10
, retinoic acid-induced protein 17
, PIAS-like protein hZimp10