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The protein encoded by TSG101 belongs to a group of apparently inactive homologs of ubiquitin-conjugating enzymes. Zusätzlich bieten wir Ihnen TSG101 Antikörper (125) und TSG101 Proteine (12) und viele weitere Produktgruppen zu diesem Protein an.
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loss of ept alters nuclear cell cycle control in developing imaginal discs and the DaPKC, presenilin, and rbf1 genes as modifiers of molecular and cellular phenotypes that result from loss of ept
analysis of genetic interactions between the Drosophila tumor suppressor gene ept and the stat92E transcription factor
Results indicate that Vpr overcomes the effects of TSG101 overexpression to support viral production by competing with TSG101 to bind Gag.
our studies reveal that Tsg101 plays a role in the trafficking of macropinocytosed Kaposi's sarcoma-associated herpesvirus in the endothelial cells
Knockdown of LAMP2A, a CMA-related protein, and TSG101, an mA-related protein, significantly but only partially decreased the punctate accumulation of GAPDH (zeige GAPDH ELISA Kits)-HT in AD293 cells and primary cultured rat cortical neurons.
the variant alleles of TSG101 rs2292179 and ATF2 (zeige ATF2 ELISA Kits) rs3845744 were associated with a reduced risk of breast cancer, particularly for subjects with BMI <24 (kg/m(2)) and postmenopausal women, respectively
TSGDelta154-1054 splice variant increases TSG101 oncogenicity by inhibiting its E3-ligase-mediated proteasomal degradation.
Results show that TSG101 bidirectionally modulates cell invasion through regulating MMP-9 (zeige MMP9 ELISA Kits) mRNA expression in different cell types.
TSG101 plays an important role in the development of hepatocellular carcinoma
Expression of tsg101 mRNA and of TSG101 protein were significantly higher in the oxaliplatin resistant cell line than parent HT-29 cels.
PSAP (zeige PSAP ELISA Kits) motif of OFR3 is required for hepatitis E virus exit and interaction with host TSG101.
Stress-internalized EGFR (zeige EGFR ELISA Kits) is retained intracellularly by continued p38 (zeige CRK ELISA Kits) activity in a mechanism involving ubiquitin-independent, ESCRT/ALIX (zeige PDCD6IP ELISA Kits)-dependent incorporation onto intraluminal vesicles (ILVs (zeige IARS ELISA Kits)) of MVBs
Tsg101 was deleted from mature oligodendroglia in vivo. This resulted in severe spongiform encephalopathy, histopathologically similar to that observed in Mgrn1 null mutant mice but with a more rapid onset.
BLOS1 (zeige BLOC1S1 ELISA Kits) interacts with sorting nexin 2 and the ESCRT-I component TSG101 to mediate the sorting of EGFR (zeige EGFR ELISA Kits) into endosomal compartments
pigment-type switching likely requires MGRN1's ubiquitin ligase activity but not its ability to bind TSG101 or NEDD4.
Tsg101 has a role in expression of EGFR (zeige EGFR ELISA Kits) and ErbB2 (zeige ERBB2 ELISA Kits) and preventing induction of autophagy prior to cell death
show that ubiquitin recognition by TSG101 is required for cSMAC formation, T cell receptor (TCR) microcluster signal termination, TCR downregulation.
Negative regulation of cell growth and differentiation by TSG101 through association with p21(Cip1/WAF1 (zeige CDKN1A ELISA Kits)).
Targeted deletion results in cell cycle arrest and p53 (zeige TP53 ELISA Kits)-independent cell death
our results suggest that Tsg101 is required for normal cell function of embryonic and adult tissues but that this gene is not a tumor suppressor for sporadic forms of breast cancer.
the TSG101 interaction with HRS is a crucial step in endocytic down-regulation of mitogenic signaling and this interaction may have a role in linking the functions of early and late endosomes
the Tsg101 protein has only weak oncogenic properties
The protein encoded by this gene belongs to a group of apparently inactive homologs of ubiquitin-conjugating enzymes. The gene product contains a coiled-coil domain that interacts with stathmin, a cytosolic phosphoprotein implicated in tumorigenesis. The protein may play a role in cell growth and differentiation and act as a negative growth regulator. In vitro steady-state expression of this tumor susceptibility gene appears to be important for maintenance of genomic stability and cell cycle regulation. Mutations and alternative splicing in this gene occur in high frequency in breast cancer and suggest that defects occur during breast cancer tumorigenesis and/or progression.
, tumor susceptibility Gene-101
, Tumor Susceptibility Gene homolog family member (tsg-101)
, ESCRT-I complex subunit TSG101
, tumor susceptibility gene 101 protein
, tumor susceptibility protein 101
, tumor susceptibility gene 101
, tumor susceptibility gene 10
, tumor susceptibility protein
, Tumor supressor