Tenascin XB (TNXB) ELISA Kits

TNXB encodes a member of the tenascin family of extracellular matrix glycoproteins. Zusätzlich bieten wir Ihnen TNXB Antikörper (62) und TNXB Proteine (4) und viele weitere Produktgruppen zu diesem Protein an.

list all ELISA KIts Gen GeneID UniProt
TNXB 7148 P22105
TNXB 81877  
TNXB 415089  
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Top TNXB ELISA Kits auf antikoerper-online.de

Showing 10 out of 29 products:

Katalog Nr. Reaktivität Sensitivität Bereich Bilder Menge Anbieter Lieferzeit Preis Details
Human 0.061 ng/mL 0.15 ng/mL - 10 ng/mL 96 Tests Anmelden zum Anzeigen 13 bis 16 Tage
Kaninchen 1.0 ng/mL 5.0-100 ng/mL   96 Tests Anmelden zum Anzeigen 15 bis 18 Tage
Maus 1.0 ng/mL 5.0-100 ng/mL   96 Tests Anmelden zum Anzeigen 15 bis 18 Tage
Meerschweinchen 1.0 ng/mL 5.0-100 ng/mL   96 Tests Anmelden zum Anzeigen 15 bis 18 Tage
Ratte 1.0 ng/mL 5.0-100 ng/mL   96 Tests Anmelden zum Anzeigen 15 bis 18 Tage
Schwein 0.938 ng/mL 1.563-100 ng/mL   96 Tests Anmelden zum Anzeigen 12 bis 14 Tage
Huhn 1.875 ng/mL 3.125-200 ng/mL   96 Tests Anmelden zum Anzeigen 12 bis 14 Tage
Affe 1.0 ng/mL 5.0-100 ng/mL   96 Tests Anmelden zum Anzeigen 15 bis 18 Tage
Hund 1.0 ng/mL 5.0-100 ng/mL   96 Tests Anmelden zum Anzeigen 15 bis 18 Tage
  96 Tests Anmelden zum Anzeigen 15 bis 18 Tage

Am meisten referenzierte TNXB ELISA Kits

  1. Human TNXB ELISA Kit für Sandwich ELISA - ABIN415102 : Kim, Woo, Suh, Ahn, Park, Hong, Lee, Ahn, Hwang, Kim, Park, Lee: Identification of vinculin as a potential plasma marker for age-related macular degeneration. in Investigative ophthalmology & visual science 2014 (PubMed)

Weitere ELISA Kits für TNXB Interaktionspartner

Human Tenascin XB (TNXB) Interaktionspartner

  1. Study describes a biallelic TNXB variants in patients with congenital adrenal hyperplasia due to CYP21A2 (zeige CYP21A2 ELISA Kits) deletions resulting in a classical Ehlers-Danlos syndrome phenotype with skin hyperextensibility, widened atrophic scars and joint hypermobility.

  2. patients with the TNX-deficient type EDS typically have generalized joint hypermobility, skin hyperextensibility and easy bruising. In contrast to the classical type, the inheritance pattern is autosomal recessive and atrophic scarring is absent. Molecular analysis of TNXB in a diagnostic setting is challenging.

  3. the identification of a rare missense variant in TNXB in combination with a positive family history of VUR and joint hypermobility may represent a non-invasive method to diagnose PVUR and warrants further evaluation in other cohorts

  4. We then quantified the tenascin-X level in serum of patients and identified tenascin-X as potent marker for ovarian cancer, showing that secretomic analysis is suitable for the identification of protein biomarkers when combined with protein immunoassay.

  5. these results suggest that mutations in TNXB can cause hereditary primary vesicoureteral reflux .

  6. Noticeable decreased expression of tenascin-X in calcific aortic valves.

  7. Tenascin-X haploinsufficiency was associated with Ehlers-Danlos syndrome in patients with congenital adrenal hyperplasia

  8. Genome-wide association study of age-related macular degeneration identifies TNXB, FKBPL (zeige FKBPL ELISA Kits) and NOTCH4 (zeige NOTCH4 ELISA Kits) as candidate susceptibility genes.

  9. Combined analysis of tenascin-C (zeige TNC ELISA Kits) expression and the nodule size improved the prediction of malignancy in this patient cohort.

  10. rs204887 itself or a nearby variant is unlikely to play a major role in the development of schizophrenia although a cumulative contribution of rare variants in the TNXB gene cannot be ruled out.

Mouse (Murine) Tenascin XB (TNXB) Interaktionspartner

  1. Results suggest that tenascin-X secreted from Schwann cells and/or fibroblasts is involved in blood vessel formation in peripheral nerves.

  2. Tenascin-x is an initiator of myocardial fibrosis and ACM development via upregulation of TGFbeta (zeige TGFB1 ELISA Kits)(1) and downregulation of PPARgamma (zeige PPARG ELISA Kits).

  3. Altered properties of the force transmission pathways of muscle due to TNX deficiency directly affect muscle function in TNX KO mice. Such effects are likely to contribute to muscle weakness experienced by patients with Ehlers-Danlos syndrome.

  4. Tenascin-X deficiency mimics Ehlers-Danlos syndrome in mice through alteration of collagen deposition

  5. TNX has either a redundant or a very subtle function in the macromolecular organization in the peripheral nerve

  6. Tnx plays a role in the regulation of cell-cell and cell-matrix interactions: Tnx-null fibroblasts exhibit weaker adhesive properties to fibronectin (zeige FN1 ELISA Kits) and B16 melanoma cells than do wild-type fibroblasts.

  7. Induction of MMP-2 by Tnx deficiency is mediated through the c-Jun N-terminal kinase and protein tyrosine kinase phosphorylation pathway.

  8. TNX is unlikely to be involved in matrix deposition in the early phase of wound healing, but it is required in the later phase when remodeling and maturation of the matrix establishes and improves its biomechanical properties.

  9. TNX knockout mice have mild pregnancy-related abnormalities.

  10. localizations of Tn-X in the leptomeningeal trabecula (TB) of adult mice and in the connective tissue of the choroid plexus (CP) in the brains of mice

Cow (Bovine) Tenascin XB (TNXB) Interaktionspartner

  1. Tenascin-X promotes activation of latent TGF-beta1 (zeige TGFB1 ELISA Kits) and subsequent epithelial to mesenchymal transition in mammary epithelial cells.

  2. mechanical analysis of collagen gels showed an increased compressive resistance of the gels containing tenascin-X, indicating that this protein might be directly involved in determining the mechanical properties of collagen-rich tissues in vivo.

  3. tenascin-X, via trimerization and multiple interactions with components of collagenous fibrils, plays a crucial role in the organisation of extracellular matrices.

  4. Tenascin-X is an elastic protein and the fibronectin (zeige FN1 ELISA Kits) type III (FnIII) domains can unfold under a stretching force and refold to regain their mechanical stability upon the removal of the stretching force.

TNXB Antigen-Profil

Beschreibung des Gens

This gene encodes a member of the tenascin family of extracellular matrix glycoproteins. The tenascins have anti-adhesive effects, as opposed to fibronectin which is adhesive. This protein is thought to function in matrix maturation during wound healing, and its deficiency has been associated with the connective tissue disorder Ehlers-Danlos syndrome. This gene localizes to the major histocompatibility complex (MHC) class III region on chromosome 6. It is one of four genes in this cluster which have been duplicated. The duplicated copy of this gene is incomplete and is a pseudogene which is transcribed but does not encode a protein. The structure of this gene is unusual in that it overlaps the CREBL1 and CYP21A2 genes at its 5' and 3' ends, respectively. Multiple transcript variants encoding different isoforms have been found for this gene.

Genbezeichner und Symbole assoziert mit Tenascin XB (TNXB) ELISA Kits

  • tenascin XB (TNXB) Antikörper
  • tenascin XB (Tnxb) Antikörper
  • avian tenascin X (TNX) Antikörper
  • tenascin-X (LOC100434533) Antikörper
  • EDS3 Antikörper
  • HXBL Antikörper
  • TENX Antikörper
  • Tn-mhc Antikörper
  • TN-X Antikörper
  • TN-Y Antikörper
  • Tnx Antikörper
  • TNXB Antikörper
  • TNXB1 Antikörper
  • TNXB2 Antikörper
  • TNXBS Antikörper
  • XB Antikörper
  • XBS Antikörper

Bezeichner auf Proteinebene für Tenascin XB (TNXB) ELISA Kits

growth-inhibiting protein 45 , hexabrachion-like protein , tenascin XB1 , tenascin XB2 , tenascin-X , tenascin X , tenascin XB , tenascin Y , tenascin X B , tenascin X pseudogene , tenascin-X-like

7148 Homo sapiens
81877 Mus musculus
282654 Bos taurus
396106 Gallus gallus
415089 Rattus norvegicus
462578 Pan troglodytes
100434533 Pongo abelii
100597679 Nomascus leucogenys
100682877 Canis lupus familiaris
445520 Sus scrofa
Ausgewählte Anbieter für TNXB (TNXB) ELISA Kits
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