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The protein encoded by SMAD9 is a member of the SMAD family, which transduces signals from TGF-beta family members. Zusätzlich bieten wir Ihnen SMAD9 Antikörper (88) und SMAD9 Kits (10) und viele weitere Produktgruppen zu diesem Protein an.
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Differential expression of TGF-beta (zeige TGFB1 Proteine) superfamily members and role of Smad1 (zeige GARS Proteine)/5/9-signalling in chondral versus endochondral chondrocyte differentiation.
Mutations in SMAD9 gene encoding for transforming growth factor (TGF)-[beta] superfamily have been identified in Pulmonary Arterial Hypertension.
Phosphorylated Smad1/5/8/9 specifically bound to the BREs of Smad8/9 gene. The present study reveals that Smad8/9 is a unique R-Smad regulated through the BMP pathway at the mRNA level.
Case Report: hamartomatous olyposis and gastrointestinal ganglioneuromas with SMAD9 germline mutation that reduces PTEN expression.
a detailed computational model for TGF-beta (zeige TGFB1 Proteine) signalling that incorporates elements of previous models together with crosstalking between Smad1 (zeige GARS Proteine)/5/8 and Smad2 (zeige SMAD2 Proteine)/3 channels through a negative feedback loop dependent on Smad7 (zeige SMAD7 Proteine).
ATP production by NaF (zeige IL8 Proteine) promotes hypertrophy-like changes via activation of phospho-Smad8
TNF (zeige TNF Proteine) activated NF-kappaB (zeige NFKB1 Proteine) pathway and inhibited the phosphorylation of Smad 1 (zeige GARS Proteine)/5/8 and BMP-2 (zeige BMP2 Proteine)-induced osteoblastic differentiation in BMMSCs
CREBZF (zeige CREBZF Proteine), a novel Smad8-binding protein.
Data suggest that Smads 1, 5 and 8 as potential prognostic markers and therapeutic targets for mTOR (zeige FRAP1 Proteine) inhibition therapy of prostate cancer.
Endoglin (zeige ENG Proteine) promotes transforming growth factor beta-mediated Smad 1 (zeige GARS Proteine)/5/8 signaling and inhibits endothelial cell migration through its association with GIPC (zeige GIPC1 Proteine)
Sphingosine 1 phosphate also up-regulated runt-related transcription factor 2 (Runx2 (zeige RUNX2 Proteine)) expression through S1PR2 (zeige S1PR2 Proteine)/RhoA (zeige RHOA Proteine)/ROCK/Smad1 (zeige SMAD1 Proteine)/5/8 signaling.
BetA can enhance in vivo osteogenic potentials of BMP2 (zeige BMP2 Proteine), possibly via stimulating Smad 1 (zeige SMAD1 Proteine)/5/8 and p38 (zeige CRK Proteine) pathways, and combination of both agents can be considered as a therapeutic strategy for bone diseases.
findings suggest that Smad9 is a new type of transcriptional regulator in BMP signaling.
The findings demonstrate for the first time that KMUP-1 can promote osteoblast maturation and differentiation in vitro via BMP-2 (zeige BMP2 Proteine)/Smad1 (zeige SMAD1 Proteine)/5/8 and Wnt (zeige WNT2 Proteine)/beta-catenin (zeige CTNNB1 Proteine) pathways.
Data indicate that PDGF (zeige PDGFA Proteine)-AA promotes mesenchymal stem cel migration via bone morphogenetic protein 2 (BMP2 (zeige BMP2 Proteine))-smad (zeige SMAD1 Proteine) proteins smad1 (zeige SMAD1 Proteine)/5/8 activation requires lysosome-mediated degradation of PDGF (zeige PDGFA Proteine) alpha Receptor (PDGFRalpha).
our data suggest a pivotal role for canonical BMP signaling demonstrating distinguished nonoverlapping function of pSmad8 with pSmad1 and pSmad5 in hfSCs regulation and hair morphogenesis but a redundant role in adult hair progenitors differentiation.
Data show that HtrA1 (zeige HTRA1 Proteine) is produced during osteoclastogenesis, and negatively regulates osteoblast differentiation, osteoblast differentiation and BMP2 (zeige BMP2 Proteine)-induced Smad1 (zeige SMAD1 Proteine)/5/8, ERK1/2 and p38 (zeige CRK Proteine) phosphorylation in pre-osteoblasts.
FGF2 (zeige FGF2 Proteine) inhibited BMP9 (zeige GDF2 Proteine)-induced osteogenic differentiation by blocking BMP9 (zeige GDF2 Proteine)-induced Smads signaling and subsequently reducing Smads dependent up-regulation of ALK1 (zeige ACVRL1 Proteine) and ALK2 (zeige ACRV1 Proteine) in mesenchymal stem cells.
a detailed computational model for TGF-beta (zeige TGFB1 Proteine) signalling that incorporates elements of previous models together with crosstalking between Smad1 (zeige SMAD1 Proteine)/5/8 and Smad2 (zeige SMAD2 Proteine)/3 channels through a negative feedback loop dependent on Smad7 (zeige SMAD7 Proteine).
The protein encoded by this gene is a member of the SMAD family, which transduces signals from TGF-beta family members. The encoded protein is activated by bone morphogenetic proteins and interacts with SMAD4. Two transcript variants encoding different isoforms have been found for this gene.
MAD, mothers against decapentaplegic homolog 9
, Mothers against decapentaplegic-like protein 9
, mothers against decapentaplegic homolog 9
, SMAD family member 9
, mothers against decapentaplegic homolog 9-like
, MAD homolog 9
, Mothers against decapentaplegic, drosophila, homolog of, 9
, SMAD, mothers against DPP homolog 9
, mothers against DPP homolog 9
, SMAD 9
, SMAD, mothers against DPP 8
, TGF-beta signal transducer Smad8