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Retinoids exert biologic effects such as potent growth inhibitory and cell differentiation activities and are used in the treatment of hyperproliferative dermatological diseases. Zusätzlich bieten wir Ihnen Retinoic Acid Receptor Responder (Tazarotene Induced) 3 Proteine (4) und Retinoic Acid Receptor Responder (Tazarotene Induced) 3 Kits (1) und viele weitere Produktgruppen zu diesem Protein an.
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Results provide evidence that RIG-I (zeige DDX58 Antikörper) as an essential mediator for influenza A virus-induced COX-2 (zeige COX2 Antikörper) expression.
perifascicular pattern of RIG-I (zeige DDX58 Antikörper) expression supports the diagnosis of dermatomyositis
The current study establishes that hypoxia can profoundly influence the inducible RIG-I (zeige DDX58 Antikörper) protein expression in malignant cells of both human and murine origin, whereas this phenomenon was not identified in nonmalignant cell lines or primary cells.
our study demonstrates that the novel pathway lncRNA Ftx/miR (zeige MLXIP Antikörper)-545/RIG-I (zeige DDX58 Antikörper) promotes hepatocellular carcinoma development
this review describes antiviral activities of RIG-I (zeige DDX58 Antikörper) against influenza viruses (standing on three legs)
Study uncovered a novel aspect of Rig-I (zeige DDX58 Antikörper) in monitoring gut (zeige GUSB Antikörper) microbiota through regulating IgA (zeige IgA Antikörper) and IL6 (zeige IL6 Antikörper)-STAT3 (zeige STAT3 Antikörper)-dependent Reg3gamma pathway. Besides, Rig-I (zeige DDX58 Antikörper) loss could also promote colorectal cancer progression both in the presence and absence of intestinal bacteria.
this study indicated that increased expression of TIG3 in primary glioblastoma is a novel biomarker for predicting poor outcome of patients. We then hypothesize that TIG3 may function in a different pattern in glioblastoma
Foot-and-mouth disease virus Viroporin 2B antagonizes RIG-I (zeige DDX58 Antikörper)-mediated antiviral effects by inhibition of its protein expression.
Overexpression of TIG3 suppresses tumor growth in hepatocellular carcinoma
miR (zeige MLXIP Antikörper)-34a is an antioncogene in multiple tumors, in this study, RIG-I (zeige DDX58 Antikörper) and miR (zeige MLXIP Antikörper)-34a suppressed cell growth, proliferation, migration, and invasion in cervical cancer cells in vitro. miR (zeige MLXIP Antikörper)-34a was validated as a new regulator of RIG-I (zeige DDX58 Antikörper) by binding to its 3' untranslated region and upregulating its expression level.
Retinoids exert biologic effects such as potent growth inhibitory and cell differentiation activities and are used in the treatment of hyperproliferative dermatological diseases. These effects are mediated by specific nuclear receptor proteins that are members of the steroid and thyroid hormone receptor superfamily of transcriptional regulators. RARRES1, RARRES2, and RARRES3 are genes whose expression is upregulated by the synthetic retinoid tazarotene. RARRES3 is thought act as a tumor suppressor or growth regulator.
HRAS-like suppressor 4
, RAR-responsive protein TIG3
, retinoic acid receptor responder protein 3
, retinoic acid-inducible gene 1
, retinoid-inducible gene 1 protein
, tazarotene-induced gene 3 protein
, retinoic acid receptor responder (tazarotene induced) 3