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Natural killer (NK) cells are lymphocytes that can mediate lysis of certain tumor cells and virus-infected cells without previous activation. Zusätzlich bieten wir Ihnen KLRG1 Proteine (10) und und viele weitere Produktgruppen zu diesem Protein an.
Showing 10 out of 169 products:
Mouse (Murine) Monoclonal KLRG1 Primary Antibody für FACS - ABIN2689801
Beyersdorf, Ding, Karp, Hanke: Expression of inhibitory "killer cell lectin-like receptor G1" identifies unique subpopulations of effector and memory CD8 T cells. in European journal of immunology 2001
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Mouse (Murine) Monoclonal KLRG1 Primary Antibody für FACS - ABIN1176845
Corral, Hanke, Vance, Cado, Raulet: NK cell expression of the killer cell lectin-like receptor G1 (KLRG1), the mouse homolog of MAFA, is modulated by MHC class I molecules. in European journal of immunology 2000
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Mouse (Murine) Monoclonal KLRG1 Primary Antibody für FACS - ABIN135349
Nakamura, Niemi, Fisher, Shultz, Seaman, Ryan: Mouse Ly-49A interrupts early signaling events in natural killer cell cytotoxicity and functionally associates with the SHP-1 tyrosine phosphatase. in The Journal of experimental medicine 1997
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Human Polyclonal KLRG1 Primary Antibody für ELISA, WB - ABIN564422
Yoon, Lim, Park, Lee: Cell-to-cell contact with hepatitis C virus-infected cells reduces functional capacity of natural killer cells. in Journal of virology 2011
Mouse (Murine) Monoclonal KLRG1 Primary Antibody für FACS - ABIN2475242
Hanke, Corral, Vance, Raulet: 2F1 antigen, the mouse homolog of the rat "mast cell function-associated antigen", is a lectin-like type II transmembrane receptor expressed by natural killer cells. in European journal of immunology 1999
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Mouse (Murine) Monoclonal KLRG1 Primary Antibody für FACS - ABIN376381
Tan, Sande, Pufnock, Blattman, Greenberg: Retinoic acid as a vaccine adjuvant enhances CD8+ T cell response and mucosal protection from viral challenge. in Journal of virology 2011
Human Polyclonal KLRG1 Primary Antibody für ELISA - ABIN4228594
Marcolino, Przybylski, Koschella, Schmidt, Voehringer, Schlesier, Pircher: Frequent expression of the natural killer cell receptor KLRG1 in human cord blood T cells: correlation with replicative history. in European journal of immunology 2004
KLRG1 expression identifies short-lived Foxp3 (zeige FOXP3 Antikörper)(+) Treg effector cells with functional plasticity in islets of NOD mice.
Treg cell accumulation in intestinal tumours from APC (zeige APC Antikörper)(min/+) mice was exclusively due to the increase in KLRG1(+) GATA3 (zeige GATA3 Antikörper)(+) Treg cells.
Although absence of KLRG1 is not enough to increase intestinal Treg cells in KLRG1 knockout mice, KLRG1 ligation reduces T-cell receptor signals and the competitive fitness of individual Treg cells in the intestine.
Ly6C, 4-1BB (zeige TNFRSF9 Antikörper), and KLRG1 have roles in the activation of lamina propria lymphocytes in the small intestine in a mouse model of Crohn's disease
This finding provides a rationale for the reciprocal expression of KLRG1 and CD103 (zeige ITGAE Antikörper) in different CD8 (zeige CD8A Antikörper)(+) T-cell subsets.
Cytotoxic KLRG1-expressed CD8 (zeige CD8A Antikörper) effector cells and defective T regulatory cells cause murine autoimmune cholangitis.
KLRG1(+) iNKT cells coexpressing CD49d and granzyme A (zeige GZMA Antikörper) persisted for several months and displayed a potent secondary response to cognate antigen.
high levels of TfRs such as those found on activated lymphocytes were found to be associated with decreased KLRG1 inhibitory function, indicating that TfRs may sequester KLRG1 from interacting with cadherins.
data further demonstrate that the inhibitory activity of KLRG1 is decreased in T cells expressing high levels of TfR (zeige TFRC Antikörper), indicating that association of KLRG1 with TfR (zeige TFRC Antikörper) hinders KLRG1-mediated silencing.
Data indicate that CD103 (zeige ITGAE Antikörper)(+)CD8 (zeige CD8A Antikörper)(+) T(RM) cells developed in the skin from epithelium-infiltrating precursor cells that lacked expression of the effector-cell marker KLRG1.
KLRG1 might contribute to the impaired antitumor immunity of memory T cells in tumor microenvironment
Killer Cell Lectin-like Receptor G1 Inhibits NK Cell Function through Activation of Adenosine 5'-Monophosphate-Activated Protein Kinase
The results suggest that a disease-associated SNP located within the 3'UTR (zeige UTS2R Antikörper) of KLRG1 directly interferes with miR (zeige MLXIP Antikörper)-584-5p binding, allowing for KLRG1 mRNA differential accumulation, which in turn may contribute to pathogenesis of pemphigus.
Demonstrate the presence of increased KLRG1-expressing T-cells in tuberculosis-treated individuals, and present KLRG1 as a marker of decreased human T-cell proliferation following BCG (zeige SLC11A1 Antikörper)-vaccination.
More T cells were KLRG1+ in the synovial fluid of patients with spondylarthritis/rheumatoid arthritis than crystal arthritis or controls. Those T cells were more functionally active, and migrated towards the synovial fluid of SpA (zeige FASL Antikörper)/RA patients.
KLRG1 was overexpressed on CD4 (zeige CD4 Antikörper)(+) T cells.
Data indicate that increments of CD8 (zeige CD8A Antikörper) + effector memory T cells in human and mouse chronic lymphocytic leukemia (CLL)(Emu-TCL1 (zeige TCL1A Antikörper) model) were due to an expansion of the inhibitory killer cell lectin-like receptor (zeige Klra6 Antikörper) G1 (KLRG1) expressing cellular subset.
These results indicate that KLRG1 negatively regulates natural killer cell numbers and functions via the Akt (zeige AKT1 Antikörper) pathway, thus providing a novel marker and therapeutic target for hepatitis C virus infection.
Natural killer (NK) cells are lymphocytes that can mediate lysis of certain tumor cells and virus-infected cells without previous activation. They can also regulate specific humoral and cell-mediated immunity. The protein encoded by this gene belongs to the killer cell lectin-like receptor (KLR) family, which is a group of transmembrane proteins preferentially expressed in NK cells. Studies in mice suggested that the expression of this gene may be regulated by MHC class I molecules. Alternatively spliced transcript variants have been reported, but their full-length natures have not yet been determined.
killer cell lectin-like receptor subfamily G, member 1
, killer cell lectin-like receptor subfamily G member 1
, mast cell function-associated antigen 2F1
, C-type lectin domain family 15 member A
, C-type lectin domain family 15, member A
, ITIM-containing receptor MAFA-L
, MAFA-like receptor
, mast cell function-associated antigen (ITIM-containing)
, mast cell function-associated antigen