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IL17RC encodes a single-pass type I membrane protein that shares similarity with the interleukin-17 receptor (IL-17RA). Zusätzlich bieten wir Ihnen Interleukin 17 Receptor C Antikörper (83) und Interleukin 17 Receptor C Proteine (16) und viele weitere Produktgruppen zu diesem Protein an.
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Five SNPs in the IL17RC (and COL6A1) genes were found to be associated with susceptibility to ossification of the posterior longitudinal ligament in Han Chinese patients.
IL-17RC rs708567 polymorphism in A/A genotype, G/G genotype, and G/a genotype did not seem to influence RA susceptibility in Tunisian population.
methylation of IL17RC could play as a marker in CNV and degeneration of RPE (zeige RPE ELISA Kits) cells in vitro.
Interleukin 17A (IL17a (zeige IL17A ELISA Kits)) and interleukin-23 (IL-23 (zeige IL23A ELISA Kits)) - dependent interleukin-17 receptor C (IL-17RC) are expressed by sputum and neutrophils in deltaF508-CFTR (zeige CFTR ELISA Kits) protein (F508del) cystic fibrosis (zeige S100A8 ELISA Kits) patients.
human IL-17RC is essential for mucocutaneous immunity to C. albicans but is otherwise largely redundant.
results suggested a potential involvement of IL-17RC+CD8 (zeige CD8A ELISA Kits)+ T cells in pathogenesis of ocular sarcoidosis
IL-17RC predisposes to the development of adolescent idiopathic scoliosis in a Chinese Han population.
Data show that IL-17RA (zeige IL17RA ELISA Kits), IL-17RC, IL-22R1, ERK1/2 (zeige MAPK1/3 ELISA Kits) MAPK (zeige MAPK1 ELISA Kits) and NF-kappaB (zeige NFKB1 ELISA Kits) pathways are involved in Th17 cytokine-induced proliferation.
Overall, our study found a significant association of IL-17RC gene polymorphisms with AIS (zeige AR ELISA Kits) in a Chinese Han population, indicating IL-17RC gene may be as a susceptibility gene for AIS (zeige AR ELISA Kits).
IL-17 (zeige IL17A ELISA Kits) and its receptor IL-17RC are involved in rheumatoid arthritis synovial fluid-mediated chemotaxis in human lung microvascular endothelial cell culture.
Study reveals a critical role of IL-17RC in maintaining baseline A20 (zeige TNFAIP3 ELISA Kits) production and a novel role of the IL-17RC-A20 (zeige TNFAIP3 ELISA Kits) axis in controlling JNK (zeige MAPK8 ELISA Kits) isoform-dependent tumor-specific homeostatic proliferation.
IL-17A (zeige IL17A ELISA Kits) and IL-17F (zeige IL17F ELISA Kits) exert distinct biological effects during pulmonary infection; the IL-17F (zeige IL17F ELISA Kits)/IL-17RC signaling axis has the potential to significantly worsen pathogen-associated inflammation of the lower respiratory tract.
Our study shows for the first time that psoriatic inflammation leads to hepatic inflammation which results in dysregulated protein/lipid metabolism through IL-17RC/NFkappaB signaling.
Expression of these mediators was confirmed in end-stage COPD (zeige ARCN1 ELISA Kits). Thus, accumulation of mast cells in COPD (zeige ARCN1 ELISA Kits) may contribute to vascular remodeling.
These data show that IL-17R signaling in the lung epithelium plays a critical role in establishing chemokine (zeige CCL1 ELISA Kits) gradients that are essential for mucosal immunity against pulmonary bacterial pathogens.
Interleukin-17 receptor C deficient mice had higher rates of apoptosis in castration-resistant prostate cancer.
Data show that signaling in IL-17RC(-/-) fibroblasts required the SEF (zeige IL17RD ELISA Kits)/IL-17R signaling domain (SEFIR).
IL-17RC is essential for mediating the functions of IL-17A (zeige IL17A ELISA Kits), IL-17F (zeige IL17F ELISA Kits), and IL-17A (zeige IL17A ELISA Kits)/F both in vitro and in vivo and directly participates in the pathogenesis of central nervous system inflammation.
The biologic activity of IL-17 (zeige IL17A ELISA Kits) is dependent on a complex composed of receptors IL-17RA (zeige IL17RA ELISA Kits) and IL-17RC.
This gene encodes a single-pass type I membrane protein that shares similarity with the interleukin-17 receptor (IL-17RA). Unlike IL-17RA, which is predominantly expressed in hemopoietic cells, and binds with high affinity to only IL-17A, this protein is expressed in nonhemopoietic tissues, and binds both IL-17A and IL-17F with similar affinities. The proinflammatory cytokines, IL-17A and IL-17F, have been implicated in the progression of inflammatory and autoimmune diseases. Multiple alternatively spliced transcript variants encoding different isoforms have been detected for this gene, and it has been proposed that soluble, secreted proteins lacking transmembrane and intracellular domains may function as extracellular antagonists to cytokine signaling.
interleukin 17 receptor C
, interleukin-17 receptor C
, interleukin-17 receptor C-like
, IL-17 receptor C
, IL17F receptor
, interleukin-17 receptor homolog
, interleukin-17 receptor-like protein