Intercellular Adhesion Molecule 3 (ICAM3) ELISA Kits

Human Intercellular Adhesion Molecule 3 (ICAM3) Interaktionspartner

1. Lewis-antigen-containing ICAM-2/3 on Jurkat leukemia cells interact with DC-SIGN to regulate DC functions.

2. exploration of the underlying mechanism demonstrated that ICAM3 not only binds to LFA-1 with its extracellular domain and structure protein ERM but also to lamellipodia with its intracellular domain which causes a tension that pulls cells apart (metastasis).

3. Increased expression of PECAM-1, ICAM-3, and VCAM-1 in colonic biopsies from patients with inflammatory bowel disease (IBD) in clinical remission is associated with subsequent flares; this suggests that increases in the expression of these proteins may be early events that lead to flares in patients with IBD

4. with larger patient groups and preferably detailed histopathological and clinical evaluations, are needed to explain the severity of ICAM-1, ICAM-2, and ICAM-3 molecules in Barrett's esophagus

5. ICAM3 acts as recognition receptors in the phagocytosis portals of macrophages for engulfment of apoptotic neutrophils.

6. this data clearly indicate that ICAM-3 promotes drug resistance via inhibition of apoptosis.

7. Intercellular adhesion molecule (ICAM)-3 mRNA is upregulated in non-adherent endothelial forming cells.

8. Results indicate that the ICAM-3 gene promoter is negatively regulated by RUNX3.

9. analysis of activated apoptotic cells induce dendritic cell maturation via engagement of Toll-like receptor 4 (TLR4), dendritic cell-specific intercellular adhesion molecule 3 (ICAM-3)-grabbing nonintegrin (DC-SIGN), and beta2 integrins

10. ICAM-3 may be an important adhesion molecule involved in chemotaxis to apoptotic human leukocytes.

11. the molecular basis of allergen-induced Th2 cell polarization

12. the cross-talk between neutrophils and NK cells is mediated by ICAM-3 and CD11d/CD18, respectively.

13. Single nucleotide polymorphisms in ICAM3 gene is associated with lymphoma.

14. induction of morphological polarization in primary T lymphocytes and Jurkat cells enhances Kidins220/ARMS colocalization with ICAM-3

15. CCR1 antagonist, BX471, did not significantly alter ICAM-3 expression in relapsing-remitting multiple sclerosis patients.

16. ICAM-3 enhances the migratory and invasive potential of human non-small cell lung cancer cells by inducing MMP-2 and MMP-9 via Akt and CREB

17. no significant risk association was found for SARS infection for the ICAM-3 Asp143Gly SNP.

18. A novel serine-rich motif in the intercellular adhesion molecule 3 is critical for its ezrin/radixin/moesin-directed subcellular targeting

19. identification of DC-SIGN binding sites

20. interactions with DC-SIGN does not promote DC-SIGN mediated HIV-1 transmission