Glucose-6-Phosphate Dehydrogenase X-Linked (G6PDX) ELISA Kits

Catalyzes the rate-limiting step of the oxidative pentose-phosphate pathway, which represents a route for the dissimilation of carbohydrates besides glycolysis. Zusätzlich bieten wir Ihnen und viele weitere Produktgruppen zu diesem Protein an.

list all ELISA KIts Gen GeneID UniProt
G6PDX 14381 Q00612;
G6PDX    
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Weitere ELISA Kits für Glucose-6-Phosphate Dehydrogenase X-Linked Interaktionspartner

Mouse (Murine) Glucose-6-Phosphate Dehydrogenase X-Linked (G6PDX) Interaktionspartner

  1. G6PD protects ischemic brain injury through increasing pentose phosphate pathway.

  2. Hepatic transcriptional profiling response to fava bean-induced oxidative stress in glucose-6-phosphate dehydrogenase-deficient mice has been reported.

  3. ATM/G6PD-driven redox metabolism promotes FLT3 inhibitor resistance in acute myeloid leukemia that can be successfully reversed.

  4. 20-HETE elicited mitochondrial superoxide production and promoted secretory phenotype of vascular smooth muscle cells by activating MAPK1-Elk-1, all of which are blocked by inhibition of G6PD.

  5. yeast hydrolysate supplementation suppressed body fat accumulation by attenuating fatty acid synthesis through the downregulation of hepatic G6PD and malic enzyme activities.

  6. Serine arginine splicing factor 3 is involved in enhanced splicing of glucose-6-phosphate dehydrogenase RNA in response to nutrients and hormones in liver

  7. F2 homozygous Gpdx mutant with C57L/J background exhibited the G6PD activity of 0.9+/-0.1 U/g Hb, level similar to those of G6PD deficiency in human.

  8. Control of hepatic nuclear superoxide production by glucose 6-phosphate dehydrogenase and NADPH oxidase-4

  9. as compared with control mice, G6PD-deficient mice had increased oxidative stress, as manifested by decreased NADPH levels and decreased GSH levels, and increased markers of lipid peroxidation.

  10. not indispensable for early embryo development; severe deficiency in the extraembryonic tissues impairs development of the placenta and causes death of the embryo; is indispensable for survival when the embryo is exposed to oxygen through its blood supply

  11. G6PD is required for limiting oxidative mutagenesis in the mouse spleen; Gpdx(a-m2Neu) is the first hypomorphic allele of a mouse housekeeping gene associated with elevated somatic mutagenesis in vivo

  12. G6PD is an essential myocardial antioxidant enzyme, required for maintaining cellular glutathione levels and protecting against oxidative stress-induced cardiac dysfunction during ischemia-reperfusion.

  13. G6pd deleted ES cells are very sensitive to oxidants, activating an apoptotic pathway at oxidant concentrations otherwise sublethal for wt ES cells.

  14. G6PD proves indispensable for definitive erythropoiesis.

  15. The study findings demonstrate a novel association between G6PD-status and inducibility of CYP2A5 and indicate that oxidative stress is not likely the key regulator of CYP2A5 expression.

Glucose-6-Phosphate Dehydrogenase X-Linked (G6PDX) Antigen-Profil

Beschreibung des Gens

Catalyzes the rate-limiting step of the oxidative pentose-phosphate pathway, which represents a route for the dissimilation of carbohydrates besides glycolysis. The main function of this enzyme is to provide reducing power (NADPH) and pentose phosphates for fatty acid and nucleic acid synthesis (By similarity). {ECO:0000250}.

Genbezeichner und Symbole assoziert mit G6PDX

  • glucose-6-phosphate dehydrogenase X-linked (G6pdx) Antikörper
  • G6pd Antikörper
  • G28A Antikörper
  • Gpdx Antikörper

Bezeichner auf Proteinebene für G6PDX

glucose-6-phosphate 1-dehydrogenase X

GENE ID SPEZIES
14381 Mus musculus
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