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Receptor for gastrin releasing peptide (GRP). Zusätzlich bieten wir Ihnen GRPR Antikörper (65) und GRPR Proteine (6) und viele weitere Produktgruppen zu diesem Protein an.
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These data suggest that GRPR expression in fibroblast-like synoviocytes (FLS)and that exogenous GRP are able to activate FLS invasion. This effect occurs at least in part through the AKT activation. Therefore, understanding of the GRP/GRPR pathway could be relevant in the development of FLS-targeted therapy for Rheumatoid arthritis .
ablating GRP (zeige UCMA ELISA Kits) receptor-expressing neurons eliminated basal and hypoxia-induced sighing, but left breathing otherwise intact initially
Together, we define the respective function of NMBR and GRPR in itch transmission, and reveal an unexpected relationship not only between the two receptors but also between the two populations of interneurons in itch signaling.
BNP-NPRA (zeige NPR1 ELISA Kits) signaling is involved in both itch and pain and does not function upstream of the GRP (zeige UCMA ELISA Kits)-GRPR dedicated neuronal pathway.
The present results suggest that BB2 receptor-expressing spinal neurons transmit herpes-associated itch by BB2 receptor-independent signaling.
spinal GRPr and NMBr independently drive itch neurotransmission in mice.
GRPR and stathmin (zeige STMN1 ELISA Kits) control in opposite directions both cued fear extinction and neural activities of the amygdala and prefrontal cortex
Gastrin-releasing peptide receptor mediates chemotaxis in neutrophils, and may be an alternative chemotactic receptor playing a role in the pathogenesis of inflammatory disorders.
The data suggest that opioid-induced itch is an active process concomitant with but independent of opioid analgesia, occurring via the unidirectional cross-activation of GRPR signaling by MOR1D heterodimerization
We propose that deletion of GRPR leads to the induction of depression-like behavior which is paralleled by dysregulation of amygdala gene expression, potentially resulting from deficient light-induced corticosterone release in GRPR-knockout mice
GRP/GRP-R signaling activation contributes to castration-resistant prostate cancer progression
Expression of Gastrin-Releasing Peptide Receptor in Breast Cancer and Its Association with Pathologic, Biologic, and Clinical Parameters
Our results suggest that, similar to what happens in neutrophils, gastrin-releasing peptide (zeige GRP ELISA Kits) is a migratory, rather than a proliferative, stimulus, for non-small cell lung carcinoma cells, indicating a putative role for gastrin-releasing peptide (zeige GRP ELISA Kits) and gastrin-releasing peptide receptor in metastasis
GRP-R regulates glucose metabolism in neuroblastoma by modulating HIF-1alpha, PDK4 and PDP2.
BBS caused a significant increase in Shh (zeige SHH ELISA Kits) gene transcription and protein secretion that was dependent on BBS-induced GPCR (zeige NMUR1 ELISA Kits)/Galphaq (zeige GNAQ ELISA Kits)-//Rho mediated activation of nuclear factor kappaB (NFkappaB (zeige NFKB1 ELISA Kits)), which can stimulate a NF-kappaB (zeige NFKB1 ELISA Kits) response element in the Shh (zeige SHH ELISA Kits) gene promoter
No association of 16 GRP and 7 GRPR variants were found with agoraphobia with/without panic disorder.
GRPR is highly expressed in epidermoid carcinoma of the anal canal, suggesting this receptor might have a role in anal carcinogenesis.
a key mechanism for GRPR-regulated colon cancer cell migration through the Galpha13-PRG (zeige PRB3 ELISA Kits)-RhoA (zeige RHOA ELISA Kits)-ROCK pathway.
GRPR expression was more pronounced in an advanced-stage lung cancer
integrin ss1 subunit critically regulates GRP-R-mediated neuroblastoma cell migration and invasion
Receptor for gastrin releasing peptide (GRP). This receptor mediates its action by association with G proteins that activate a phosphatidylinositol-calcium second messenger system.
gastrin-releasing peptide receptor
, gastrin-releasing peptide receptor-like
, GRP-preferring bombesin receptor
, bombesin receptor